Le SPECT à l'I-123 IMP et au Tc-99m HMPAO en neurologie pédiatrique: aspects méthodologiques et applications cliniques

Doctorat en sciences médicalesinfo:eu-repo/semantics/nonPublishe

Ne vous faites pas de bile, ça se soigne

SCOPUS: no.jinfo:eu-repo/semantics/publishe

Bilateral cerebral mediofrontal hypoactivity in Tc-99m HMPAO SPECT imaging

SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Detection of bilateral and symmetrical anomalies in technetium-99m-HMPAO brain SPECT studies

SCOPUS: ar.jinfo:eu-repo/semantics/publishe

La tomographie d'émission monophotonique au (99M)TC HMPAO en neurologie pédiatrique

SCOPUS: re.jinfo:eu-repo/semantics/publishe

Regional cerebral blood flow in chronic alcoholics: A Tc-99m HMPAO SPECT study

SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Regional brain blood flow in congenital dysphasia: Studies with technetium-99m HM-PAO SPECT

SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Functional imaging of brain maturation in humans using iodine-123 iodoamphetamine and SPECT

SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Metabolic changes in the rat brain after acute and chronic ethanol intoxication: A 31P NMR spectroscopy study

In this work, 31P phosphorus NMR (31P NMR) studies of the brain have been conducted in rats acutely and chronically intoxicated with ethanol. In both groups, changes in levels of high-energy phosphates were observed: increase of phosphocreatinine (PCr)/β AaTP and PCr/inorganic phosphate (Pi) in acute and long-term ethanol exposure, and decrease of Piβ ATP after acute ethanol administration. These changes in high-energy phosphates, indicative of a reduction of adenosine triphosphate (ATP) and PCr consumption (PCr+ ADP + H+ ATP + Cr; ATP ADP + Pi), suggest a reduction of cerebral metabolism both in acute and chronic ethanol exposure. In addition, in the group of rats chronically intoxicated with ethanol, there were variations in phosphodiester peak intensities (decrease of phosphomonoester (PME)/phosphodiester (PDE), increase of PDE/β ATP), suggesting increased breakdown of membrane phospholipids. These changes could provide a metabolic explanation for the development of cerebral atrophy in chronic alcoholism.SCOPUS: ar.jFLWNAinfo:eu-repo/semantics/publishe

Electroencephalographic mapping and 99mTc HMPAO single-photon emission computed tomography in carbon monoxide poisoning

SCOPUS: ar.jinfo:eu-repo/semantics/publishe