27 research outputs found
Pathogenesis of Henoch-Schönlein purpura nephritis
Get PDFThe severity of renal involvement is the major factor determining the long-term outcome of children with Henoch-Schönlein purpura (HSP) nephritis (HSPN). Approximately 40% children with HSP develop nephritis, usually within 4 to 6 weeks after the initial onset of the typical purpuric rashes. Although the pathogenetic mechanisms are still not fully delineated, several studies suggest that galactose-deficient IgA1 (Gd-IgA1) is recognized by anti-glycan antibodies, leading to the formation of the circulating immune complexes and their mesangial deposition that induce renal injury in HSPN
The difficulty in considering modifiable pathology risk factors in children with IgA nephropathy: crescents and timing of renal biopsy
No full textClinical manifestations of Henoch–Schönlein purpura nephritis and IgA nephropathy: comparative analysis of data from the Japan Renal Biopsy Registry (J-RBR)
No full textThe ISKDC classification and a new semiquantitative classification for predicting outcomes of Henoch–Schönlein purpura nephritis
No full textIncreased Intestinal Permeability to (51 Cr) EDTA Is Correlated with IgA Immune Complex-Plasma Levels in Children with IgA-Associated Nephropathies
No full text
Different histological classifications for Henoch-Schönlein purpura nephritis: which one should be used?
No full textSequential Measurements of the Reticulo-Endothelial System Function in Henoch-Schönlein Disease of Childhood: Correlations with Various Immunological Parameters
No full textValidation of the absolute renal risk of dialysis/death in adults with IgA nephropathy secondary to Henoch-Schönlein purpura: a monocentric cohort study
No full textUsing the Oxford classification of IgA nephropathy to predict long-term outcomes of Henoch–Schönlein purpura nephritis in adults
No full text
