Orbital anastomoses of the anterior deep temporal artery

The anterior deep temporal artery may provide a major collateral pathway to the intracranial circulation through anastomoses with branches of the ophthalmic artery. Review of carotid angiograms in 26 patients with internal carotid artery occlusive disease revealed anterior deep temporal to ophthalmic artery anastomoses in 16 cases. This route of collateral blood flow was associated in most instances with total occlusion of the cervical portion of the internal carotid artery. Three cases demonstrating the angiographic anatomy of the anterior deep temporal artery and its potential anastomoses with branches of the ophthalmic artery are presented. L'artère temporale profonde antérieure peut être à l'origine de circulation colatérale grâce à ses anastomoses avec l'artère ophtalmique. Une telle anastomose a été constatée 16 fois sur 26 cas de thrombose de l'artère carotide interne. Über die A. temporalis anterior ist über Anastomosen zu den Ästen der A. ophthalmica ein Kollateral-Kreislauf zu den intracraniellen Gefäßabschnitten möglich. Bei 26 Patienten mit einem A. carotis interna-Verschluß zeigte sich dieser Kreislauf in 16 Fällen. Es wird über 3 Fälle ausführlich berichtet, bei denen die angiographische Anatomie der A. temporalis anterior und die möglichen Anastomosen mit Ästen der A. ophthalmica besprochen wird.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/46672/1/234_2004_Article_BF00335020.pd

Localised neuronal migration disorder and intractable epilepsy: A prenatal vascular aetiology

Localised neuronal heterotopias are an increasingly recognised cause of intractable focal epilepsies. The aetiology of these circumscribed disorders of neuronal migration is often unknown although in some instances proximity to areas of prenatal infarction suggests that severe ischaemia was responsible. A patient is described with intractable complex partial seizures associated with heterotopic grey matter and cerebral hypoplasia confined to the territory of the left posterior cerebral artery; the left hippocampus was spared. Angiography showed a normal left anterior choroidal artery but a hypoplastic left posterior cerebral artery, implicating prenatal ischaemia without frank infarction as the aetiology of the malformation