87 research outputs found
Longitudinal-Transverse Separations of Structure Functions at Low for Hydrogen and Deuterium
We report on a study of the longitudinal to transverse cross section ratio,
, at low values of and , as determined from
inclusive inelastic electron-hydrogen and electron-deuterium scattering data
from Jefferson Lab Hall C spanning the four-momentum transfer range 0.06 GeV. Even at the lowest values of , remains
nearly constant and does not disappear with decreasing , as expected. We
find a nearly identical behaviour for hydrogen and deuterium.Comment: 4 pages, 2 gigure
Proton Spin Structure in the Resonance Region
We have examined the spin structure of the proton in the region of the
nucleon resonances (1.085 GeV < W < 1.910 GeV) at an average four momentum
transfer of Q^2 = 1.3 GeV^2. Using the Jefferson Lab polarized electron beam, a
spectrometer, and a polarized solid target, we measured the asymmetries
A_parallel and A_perp to high precision, and extracted the asymmetries A_1 and
A_2, and the spin structure functions g_1 and g_2. We found a notably non-zero
A_perp, significant contributions from higher-twist effects, and only weak
support for polarized quark--hadron duality.Comment: 6 pages, 4 figures, REVTeX4, similar to PRL submission, plots
colorized and appenix added, v3: minor edit, matches PR
Probing Quark-Gluon Interactions with Transverse Polarized Scattering
We have extracted QCD matrix elements from our data on double polarized
inelastic scattering of electrons on nuclei. We find the higher twist matrix
element \tilde{d_2}, which arises strictly from quark- gluon interactions, to
be unambiguously non zero. The data also reveal an isospin dependence of higher
twist effects if we assume that the Burkhardt-Cottingham Sum rule is valid. The
fundamental Bjorken sum rule obtained from the a0 matrix element is satisfied
at our low momentum transfer.Comment: formerly "Nachtmann Moments of the Proton and Deuteron Spin Structure
Functions
Proton G_E/G_M from beam-target asymmetry
The ratio of the proton's electric to magnetic form factor, G_E/G_M, can be
extracted in elastic electron-proton scattering by measuring either cross
sections, beam-target asymmetry or recoil polarization. Separate determinations
of G_E/G_M by cross sections and recoil polarization observables disagree for
Q^2 > 1 (GeV/c)^2. Measurement by a third technique might uncover an unknown
systematic error in either of the previous measurements. The beam-target
asymmetry has been measured for elastic electron-proton scattering at Q^2 =
1.51 (GeV/c)^2 for target spin orientation aligned perpendicular to the beam
momentum direction. This is the largest Q^2 at which G_E/G_M has been
determined by a beam-target asymmetry experiment. The result, \muG_E/G_M =
0.884 +/- 0.027 +/- 0.029, is compared to previous world data.Comment: 8 pages, 6 figures, Updated to be version published in Physical
Review
Evaluation of a service intervention to improve awareness and uptake of bowel cancer screening in ethnically-diverse areas
The Policy
Research Unit in Cancer Awareness, Screening and Early Diagnosis
receives funding for a research programme from the UK
Department of Health Policy Research Programme (grant no.
106/0001). It is a collaboration between researchers from seven
institutions (the Queen Mary University of London, the UCL, the
King’s College London, the London School of Hygiene and
Tropical Medicine, the Hull York Medical School, the Durham
University and the Peninsula Medical School)
p73 Regulates Neurodegeneration and Phospho-Tau Accumulation during Aging and Alzheimer's Disease
SummaryThe genetic mechanisms that regulate neurodegeneration are only poorly understood. We show that the loss of one allele of the p53 family member, p73, makes mice susceptible to neurodegeneration as a consequence of aging or Alzheimer's disease (AD). Behavioral analyses demonstrated that old, but not young, p73+/− mice displayed reduced motor and cognitive function, CNS atrophy, and neuronal degeneration. Unexpectedly, brains of aged p73+/− mice demonstrated dramatic accumulations of phospho-tau (P-tau)-positive filaments. Moreover, when crossed to a mouse model of AD expressing a mutant amyloid precursor protein, brains of these mice showed neuronal degeneration and early and robust formation of tangle-like structures containing P-tau. The increase in P-tau was likely mediated by JNK; in p73+/− neurons, the activity of the p73 target JNK was enhanced, and JNK regulated P-tau levels. Thus, p73 is essential for preventing neurodegeneration, and haploinsufficiency for p73 may be a susceptibility factor for AD and other neurodegenerative disorders
Measurements of electron-proton elastic cross sections for
We report on precision measurements of the elastic cross section for
electron-proton scattering performed in Hall C at Jefferson Lab. The
measurements were made at 28 unique kinematic settings covering a range in
momentum transfer of 0.4 5.5 . These measurements
represent a significant contribution to the world's cross section data set in
the range where a large discrepancy currently exists between the ratio of
electric to magnetic proton form factors extracted from previous cross section
measurements and that recently measured via polarization transfer in Hall A at
Jefferson Lab.Comment: 17 pages, 18 figures; text added, some figures replace
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