3,263 research outputs found

    Bipolaronic blockade effect in quantum dots with negative charging energy

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    We investigate single-electron transport through quantum dots with negative charging energy induced by a polaronic energy shift. For weak dot-lead tunnel couplings, we demonstrate a bipolaronic blockade effect at low biases which suppresses the oscillating linear conductance, while the conductance resonances under large biases are enhanced. Novel conductance plateau develops when the coupling asymmetry is introduced, with its height and width tuned by the coupling strength and external magnetic field. It is further shown that the amplitude ratio of magnetic-split conductance peaks changes from 3 to 1for increasing coupling asymmetry. Though we demonstrate all these transport phenomena in the low-order single-electron tunneling regime, they are already strikingly different from the usual Coulomb blockade physics and are easy to observe experimentally.Comment: 6 pages, 5 figure

    FAST polarization mapping of the SNR VRO 42.05.01

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    We have obtained the polarization data cube of the VRO 42.05.01 supernova remnant at 1240 MHz using the Five-hundred-meter Aperture Spherical radio Telescope (FAST). Three-dimensional Faraday Synthesis is applied to the FAST data to derive the Faraday depth spectrum. The peak Faraday depth map shows a large area of enhanced foreground RM of ~60 rad m-2 extending along the remnant's "wing" section, which coincides with a large-scale HI shell at -20 km/s. The two depolarization patches within the "wing" region with RM of 97 rad m-2 and 55 rad m-2 coincide with two HI structures in the HI shell. Faraday screen model fitting on the Canadian Galactic Plane Survey (CGPS) 1420 MHz full-scale polarization data reveals a distance of 0.7-0.8d_{SNR} in front of the SNR with enhanced regular magnetic field there. The highly piled-up magnetic field indicates that the HI shell at -20 km/s could originate from an old evolved SNR.Comment: 9 pages, 8 figures, accepted by Ap

    Effect of source tampering in the security of quantum cryptography

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    The security of source has become an increasingly important issue in quantum cryptography. Based on the framework of measurement-device-independent quantum-key-distribution (MDI-QKD), the source becomes the only region exploitable by a potential eavesdropper (Eve). Phase randomization is a cornerstone assumption in most discrete-variable (DV-) quantum communication protocols (e.g., QKD, quantum coin tossing, weak coherent state blind quantum computing, and so on), and the violation of such an assumption is thus fatal to the security of those protocols. In this paper, we show a simple quantum hacking strategy, with commercial and homemade pulsed lasers, by Eve that allows her to actively tamper with the source and violate such an assumption, without leaving a trace afterwards. Furthermore, our attack may also be valid for continuous-variable (CV-) QKD, which is another main class of QKD protocol, since, excepting the phase random assumption, other parameters (e.g., intensity) could also be changed, which directly determine the security of CV-QKD.Comment: 9 pages, 6 figure

    Changes of mitochondrial pathway in hypoxia/reoxygenation induced cardiomyocytes apoptosis.

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    The role of mitochondrial apoptotic pathway in cardiomyocytes subjected to hypoxia/reoxygenation(H/R) was studied. Cultured cardiomyocytes from neonatal Sprague-Dawley rats were exposed to hyoxia/reoxygenation, the apoptotic cardiomyocytes were stained with Annexin-V-FITC, Hoechst 33342 and TUNEL assay. Mitochondrial transmembrane potential of cardiomyocytes was assessed by JC-1 under fluorescence microscope, the expressions of bcl-2, bax, cytochrome c, apoptosis-induced factor (AIF), and caspase-3 were tested by western-blot. Our data showed apoptosis of cardiomyocytes was significantly increased during H/R, accompanied by translocation of bax to mitochondria, release of cytochrome c and AIF to cytosol. The results indicate that the mitochondrial-mediated apoptotic pathway is initiated as a result of H/R
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