18 research outputs found
Successes and Failures in International Human Trafficking Law
Professor Carr yesterday remarked that human trafficking is too often discussed only in theoretical or academic ways. I\u27ve spent most of my career in the field, where interactions with victims, traffickers, and defense attorneys are anything but theoretical. But as keynote speaker for an academic symposium this morning, I\u27m going to try to lay out a bit of the conceptual state of play from my current vantage point. The title of this symposium, Successes and Failures in International Human Trafficking Law, is a bit binary. Perhaps, in the best diplomatic tradition, we can temper that to Limitations and Opportunities in International Human Trafficking Law for my purposes today. And I think I\u27m suited to offer some personal reflections here at the Law School today, having worked on this issue, whether as one of Professor MacKinnon\u27s students, helping with the groundbreaking symposium that brought cutting-edge work to the fore; as a young trial lawyer in the Civil Rights Division; as a staff member on the House Judiciary Committee; and now as Ambassador-at- Large to Monitor and Combat Trafficking in Persons
Successes and Failures in International Human Trafficking Law
Professor Carr yesterday remarked that human trafficking is too often discussed only in theoretical or academic ways. I\u27ve spent most of my career in the field, where interactions with victims, traffickers, and defense attorneys are anything but theoretical. But as keynote speaker for an academic symposium this morning, I\u27m going to try to lay out a bit of the conceptual state of play from my current vantage point. The title of this symposium, Successes and Failures in International Human Trafficking Law, is a bit binary. Perhaps, in the best diplomatic tradition, we can temper that to Limitations and Opportunities in International Human Trafficking Law for my purposes today. And I think I\u27m suited to offer some personal reflections here at the Law School today, having worked on this issue, whether as one of Professor MacKinnon\u27s students, helping with the groundbreaking symposium that brought cutting-edge work to the fore; as a young trial lawyer in the Civil Rights Division; as a staff member on the House Judiciary Committee; and now as Ambassador-at- Large to Monitor and Combat Trafficking in Persons
Ozymandias
https://scholarworks.boisestate.edu/bfa_exhibits_2018_spring/1000/thumbnail.jp
High Desert
https://scholarworks.boisestate.edu/bfa_exhibits_2018_spring/1003/thumbnail.jp
Longing
https://scholarworks.boisestate.edu/bfa_exhibits_2018_spring/1004/thumbnail.jp
Ozymandias (Detail)
https://scholarworks.boisestate.edu/bfa_exhibits_2018_spring/1001/thumbnail.jp
Pedagog
https://scholarworks.boisestate.edu/bfa_exhibits_2018_spring/1002/thumbnail.jp
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Reconfigurable mobile manipulation for accident response
The need for a telerobotic vehicle with hazard sensing and integral manipulation capabilities has been identified for use in transportation accidents where nuclear weapons are involved. The Accident Response Mobile Manipulation System (ARMMS) platform has been developed to provide remote dexterous manipulation and hazard sensing for the Accident Response Group (ARG) at Sandia National Laboratories. The ARMMS' mobility platform is a military HMMWV [High Mobility Multipurpose Wheeled Vehicle] that is teleoperated over RF or Fiber Optic communication channels. ARMMS is equipped with two high strength Schilling Titan II manipulators and a suite of hazardous gas and radiation sensors. Recently, a modular telerobotic control architecture call SMART (Sandia Modular Architecture for Robotic and Teleoperation) has been applied to ARMMS. SMART enables input devices and many system behaviors to be rapidly configured in the field for specific mission needs. This paper summarizes current SMART developments applied to ARMMS
and Epigenetic Dysregulation in Diabetes-prone Bicongenic B6.NODC11bxC1tb Mice
In Type 1 diabetic (T1D) human monocytes, STAT5 aberrantly binds to epigenetic regulatory sites of two proinflammatory genes, CSF2 (encoding granulocyte–macrophage colony-stimulating factor) and PTGS2 (encoding prostaglandin synthase 2/cyclooxygenase 2). Bicongenic B6.NOD C11bxC1tb mice re-create this phenotype of T1D monocytes with only two nonobese diabetic (NOD) Idd subloci (130.8 Mb–149.7 Mb, of Idd5 on Chr 1 and 32.08–53.85 Mb of Idd4.3 on Chr11 ) on C57BL/6 genetic background. These two Idd loci interact through STAT5 binding at upstream regulatory regions affecting Csf2 ( Chr 11 ) and Ptgs2 ( Chr 1 ) expression. B6.NODC11bxC1tb mice exhibited hyperglycemia and immune destruction of pancreatic islets between 8 and 30 weeks of age, with 12%–22% penetrance. Thus, B6.NODC11bxC1tb mice embody NOD epigenetic dysregulation of gene expression in myeloid cells, and this defect appears to be sufficient to impart genetic susceptibility to diabetes in an otherwise genetically nonautoimmune mouse