Ginzburg-Landau-type theory of non-polarized spin superconductivity

Since the concept of spin superconductor was proposed, all the related studies concentrate on spin-polarized case. Here, we generalize the study to spin-non-polarized case. The free energy of non-polarized spin superconductor is obtained, and the Ginzburg-Landau-type equations are derived by using the variational method. These Ginzburg-Landau-type equations can be reduced to the spin-polarized case when the spin direction is fixed. Moreover, the expressions of super linear and angular spin currents inside the superconductor are derived. We demonstrate that the electric field induced by super spin current is equal to the one induced by equivalent charge obtained from the second Ginzburg-Landau-type equation, which shows self-consistency of our theory. By applying these Ginzburg-Landau-type equations, the effect of electric field on the superconductor is also studied. These results will help us get a better understanding of the spin superconductor and the related topics such as Bose-Einstein condensate of magnons and spin superfluidity.Comment: 9 pages, 5 figure

The Strong Decay Patterns of the 1−+1^{-+} Exotic Hybrid Mesons

We calculate the coupling constants of the decay modes $1^{-+}\rightarrow\rho\pi, f_1\pi, b_1\pi, \eta\pi, \eta'\pi, a_1\pi, f_1\eta$ within the framework of the light-cone QCD sum rule. Then we calculate the partial width of these decay channels, which differ greatly from the existing calculations using phenomenological models. For the isovector $1^{-+}$ state, the dominant decay modes are $\rho\pi, f_1\pi$. For its isoscalar partner, its dominant decay mode is $a_1\pi$. We also discuss the possible search of the $1^{-+}$ state at BESIII, for example through the decay chains $J/\psi (\psi')\to \pi_1 +\gamma$ or $J/\psi (\psi')\to \pi_1 +\rho$ where $\pi_1$ can be reconstructed through the decay modes $\pi_1\to \rho\pi\to \pi^+\pi^-\pi^0$ or $\pi_1\to f_1(1285)\pi^0$. Hopefully the present work will be helpful to the experimental establishment of the $1^{-+}$ hybrid meson.Comment: 14 pages, 10 figure

General theory of decoy-state quantum cryptography with source errors

The existing theory of decoy-state quantum cryptography assumes the exact control of each states from Alice's source. Such exact control is impossible in practice. We develop the theory of decoy-state method so that it is unconditionally secure even there are state errors of sources, if the range of a few parameters in the states are known. This theory simplifies the practical implementation of the decoy-state quantum key distribution because the unconditional security can be achieved with a slightly shortened final key, even though the small errors of pulses are not corrected.Comment: Our results can be used securely for any source of diagonal states, including the Plug-&-Play protocol with whatever error pattern, if we know the ranges of errors of a few parameter

Tumor necrosis factor–Α contributes to below-level neuropathic pain after spinal cord injury

Objective Our objective was to elucidate the mechanisms responsible for below-level pain after partial spinal cord injury (SCI). Methods We used lateral hemisection to model central neuropathic pain and herpes simplex viral (HSV) vector–mediated transfer of the cleaved soluble receptor for tumor necrosis factor–Α (TNF-Α) to evaluate the role of TNF-Α in the pathogenesis of below-level pain. Results We found activation of microglia and increased expression of TNF-Α below the level of the lesion in the lumbar spinal cord after T13 lateral hemisection that correlated with emergence of mechanical allodynia in the hind limbs of rats. Lumbar TNF-Α had an apparent molecular weight of 27kDa, consistent with the full-length transmembrane form of the protein (mTNF-Α). Expression of the p55 TNF soluble receptor (sTNFRs) by HSV-mediated gene transfer resulted in reduced pain behavior and a decreased number of ED1-positive cells, as well as decreased phosphorylation of the p38 MAP kinase (p-p38) and diminished expression of mTNF-Α in the dorsal horn. Interpretation These results suggest that expression of mTNF-Α after injury is related to development of pain, and that reverse signaling through mTNF-Α by sTNFR at that level reduces cellular markers of inflammatory response and pain-related behavior. Ann Neurol 2006;59:843–851Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/50655/1/20855_ftp.pd

Strong quantum fluctuation of vortices in the new superconductor MgB2MgB_2

By using transport and magnetic measurement, the upper critical field $H_{c2}(T)$ and the irreversibility line $H_{irr}(T)$ has been determined. A big separation between $H_{c2}(0)$ and $H_{irr}(0)$ has been found showing the existence of a quantum vortex liquid state induced by quantum fluctuation of vortices in the new superconductor $MgB_2$. Further investigation on the magnetic relaxation shows that both the quantum tunneling and the thermally activated flux creep weakly depends on temperature. But when the melting field $H_{irr}$ is approached, a drastic rising of the relaxation rate is observed. This may imply that the melting of the vortex matter at a finite temperature is also induced by the quantum fluctuation of vortices.Comment: 4 pages, 4 figure

Stability of Excited Dressed States with Spin-Orbit Coupling

We study the decay behaviors of ultracold atoms in metastable states with spin-orbit coupling (SOC), and demonstrate that there are two SOC-induced decay mechanisms. One arises from the trapping potential and the other is due to interatomic collision. We present general schemes for calculating decay rates from these two mechanisms, and illustrate how the decay rates can be controlled by experimental parameters.We experimentally measure the decay rates over a broad parameter region, and the results agree well with theoretical calculations. This work provides an insight for both quantum simulation involving metastable dressed states and studies on few-body problems with SO coupling.Comment: 4.5 pages, 4 figures, the latest versio

Transcriptome landscape of the human placenta

<p>Abstract</p> <p>Background</p> <p>The placenta is a key component in understanding the physiological processes involved in pregnancy. Characterizing genes critical for placental function can serve as a basis for identifying mechanisms underlying both normal and pathologic pregnancies. Detailing the placental tissue transcriptome could provide a valuable resource for genomic studies related to placental disease.</p> <p>Results</p> <p>We have conducted a deep RNA sequencing (RNA-Seq) study on three tissue components (amnion, chorion, and decidua) of 5 human placentas from normal term pregnancies. We compared the placental RNA-Seq data to that of 16 other human tissues and observed a wide spectrum of transcriptome differences both between placenta and other human tissues and between distinct compartments of the placenta. Exon-level analysis of the RNA-Seq data revealed a large number of exons with differential splicing activities between placenta and other tissues, and 79% (27 out of 34) of the events selected for RT-PCR test were validated. The master splicing regulator <it>ESRP1 </it>is expressed at a proportionately higher level in amnion compared to all other analyzed human tissues, and there is a significant enrichment of ESRP1-regulated exons with tissue-specific splicing activities in amnion. This suggests an important role of alternative splicing in regulating gene function and activity in specific placental compartments. Importantly, genes with differential expression or splicing in the placenta are significantly enriched for genes implicated in placental abnormalities and preterm birth. In addition, we identified 604-1007 novel transcripts and 494-585 novel exons expressed in each of the three placental compartments.</p> <p>Conclusions</p> <p>Our data demonstrate unique aspects of gene expression and splicing in placental tissues that provide a basis for disease investigation related to disruption of these mechanisms. These data are publicly available providing the community with a rich resource for placental physiology and disease-related studies.</p