30 research outputs found

    Effect van verdund vezelrijk voer op ontwikkeling van verenschade en dierprestaties van opfok- en leghennen onder pikkerij-bevorderende omstandigheden = Effect of diluted NSP-high diets under feather pecking prone conditions on the development of feather damage and performance of rearing and laying hens

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    In this study, the effect of diluted NSP-high diets under feather pecking prone conditions, on the development of feather damage and performance of rearing and laying hens was investigated. Compared to hens fed undiluted diets, feather damage at the end of the laying period was reduced by 20% by feeding both diluted rearing and laying diets

    Influence of Conversion and Anastomotic Leakage on Survival in Rectal Cancer Surgery; Retrospective Cross-sectional Study

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    Verenschade afgeremd

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    Verdund voer lijkt een onvermijdelijk onderdeel van een strategie tegen verenpikken. Het leidde in onderzoek tot de minste verenschade

    Effect van verdund vezelrijk voer op ontwikkeling van verenschade en dierprestaties van opfok- en leghennen onder pikkerij-bevorderende omstandigheden = Effect of diluted NSP-high diets under feather pecking prone conditions on the development of feather damage and performance of rearing and laying hens

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    In this study, the effect of diluted NSP-high diets under feather pecking prone conditions, on the development of feather damage and performance of rearing and laying hens was investigated. Compared to hens fed undiluted diets, feather damage at the end of the laying period was reduced by 20% by feeding both diluted rearing and laying diets

    Verenschade afgeremd

    Full text link
    Verdund voer lijkt een onvermijdelijk onderdeel van een strategie tegen verenpikken. Het leidde in onderzoek tot de minste verenschade

    The role of genetic factors in age at natural menopause

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    Background: Environmental factors explain only a small part of the age variance at which menopause commences. The variation in natural menopause is a trait predominantly determined by interaction of multiple genes, whose identity and causative genetic variation remains to be determined. Menopause is a retrospective marker for the reproductive capacity of preceding years, since subfertility and infertility precede menopause at distinct time-intervals. In the present study we have investigated the contribution of genetic factors to menopausal age. Methods: Data were collected from a random population sample of singleton and twin sisters participating in a prospective breast cancer screening project, who had subsequently experienced natural menopause. Heritability of menopausal age was estimated with analysis of variance, Mx modelling and Gibbs sampling. Results: All produced almost identical heritability estimates of 0.85-0.87 for singleton sisters, suggesting a strong genetic contribution to menopausal age. Twin data were used to distinguish additive genetic from common environmental effects; a heritability of 0.71-0.72 was determined, which does not deviate significantly from the estimate for singleton sisters. Conclusions: According to our findings, a woman with a family history of early menopause risks early menopause and consequently early reproductive failure herself
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