21 research outputs found
Spin-Orbit-Induced Magnetic Anisotropy for Impurities in Metallic Samples I. Surface Anisotropy
Motivated by the recent measurements of Kondo resistivity in thin films and
wires, where the Kondo amplitude is suppressed for thinner samples, the surface
anisotropy for magnetic impurities is studied. That anisotropy is developed in
those cases where in addition to the exchange interaction with the impurity
there is strong spin-orbit interaction for conduction electrons around the
impurity in the ballistic region. The asymmetry in the neighborhood of the
magnetic impurity exhibits the anisotropy axis which, in the case of a
plane surface, is perpendicular to the surface. The anisotropy energy is
for spin , and the anisotropy constant is
inversionally proportional to distance measured from the surface and
. Thus at low temperature the spin is frozen in a singlet or doublet of
lowest energy. The influence of that anisotropy on the electrical resistivity
is the subject of the following paper (part II).Comment: 28 pages, RevTeX (using epsfig), 8 eps figures included, submitted to
PR
The Screening Cloud in the k-Channel Kondo Model: Perturbative and Large-k Results
We demonstrate the existence of a large Kondo screening cloud in the
k-channel Kondo model using both renormalization group improved perturbation
theory and the large-k limit. We study position (r) dependent spin Green's
functions in both static and equal time cases. The equal-time Green's function
provides a natural definition of the screening cloud profile, in which the
large Kondo scale appears. At large distances it consists of both a slowly
varying piece and a piece which oscillates at twice the Fermi wave-vector. This
function is calculated at all r in the large-k limit. Static Green's functions
(Knight shift or susceptibility) consist only of a term oscillating at 2kF, and
appear to factorize into a function of r times a function of T for rT << vF, in
agreement with NMR experiments. Most of the integrated susceptibility comes
from the impurity-impurity part with conduction electron contributions
suppressed by powers of the bare Kondo coupling. The single-channel and
overscreened multi-channel cases are rather similar although anomalous
power-laws occur in the latter case at large r and low T due to irrelevant
operator corrections.Comment: 22 Revtex pages, 12 figure
Differential Cerebral Cortex Transcriptomes of Baboon Neonates Consuming Moderate and High Docosahexaenoic Acid Formulas
BACKGROUND: Docosahexaenoic acid (DHA, 22:6n-3) and arachidonic acid (ARA, 20:4n-6) are the major long chain polyunsaturated fatty acids (LCPUFA) of the central nervous system (CNS). These nutrients are present in most infant formulas at modest levels, intended to support visual and neural development. There are no investigations in primates of the biological consequences of dietary DHA at levels above those present in formulas but within normal breastmilk levels. METHODS AND FINDINGS: Twelve baboons were divided into three formula groups: Control, with no DHA-ARA; “L”, LCPUFA, with 0.33%DHA-0.67%ARA; “L3”, LCPUFA, with 1.00%DHA-0.67%ARA. All the samples are from the precentral gyrus of cerebral cortex brain regions. At 12 weeks of age, changes in gene expression were detected in 1,108 of 54,000 probe sets (2.05%), with most showing <2-fold change. Gene ontology analysis assigns them to diverse biological functions, notably lipid metabolism and transport, G-protein and signal transduction, development, visual perception, cytoskeleton, peptidases, stress response, transcription regulation, and 400 transcripts having no defined function. PLA2G6, a phospholipase recently associated with infantile neuroaxonal dystrophy, was downregulated in both LCPUFA groups. ELOVL5, a PUFA elongase, was the only LCPUFA biosynthetic enzyme that was differentially expressed. Mitochondrial fatty acid carrier, CPT2, was among several genes associated with mitochondrial fatty acid oxidation to be downregulated by high DHA, while the mitochondrial proton carrier, UCP2, was upregulated. TIMM8A, also known as deafness/dystonia peptide 1, was among several differentially expressed neural development genes. LUM and TIMP3, associated with corneal structure and age-related macular degeneration, respectively, were among visual perception genes influenced by LCPUFA. TIA1, a silencer of COX2 gene translation, is upregulated by high DHA. Ingenuity pathway analysis identified a highly significant nervous system network, with epidermal growth factor receptor (EGFR) as the outstanding interaction partner. CONCLUSIONS: These data indicate that LCPUFA concentrations within the normal range of human breastmilk induce global changes in gene expression across a wide array of processes, in addition to changes in visual and neural function normally associated with formula LCPUFA