1,892 research outputs found
Polarization probes of vorticity in heavy ion collisions
We discuss the information that can be deduced from a measurement of particle
(hyperon or vector meson) polarization in ultrarelativistic nuclear collisions.
We describe the sensitivity of polarization to initial conditions, hydrodynamic
evolution and mean free path, and find that the polarization observable is
sensitive to all details and stages of the system's evolution. We suggest that
an experimental investigation covering production plane and reaction plane
polarizations, as well as the polarization of jet-associated particles in the
plane defined by the jet and particle direction, can help in disentangling the
factors contributing to this observable. Scans of polarization in energy and
rapidity might also point to a change in the system's properties.Comment: In press, Phys.Rev.C. One new figure, text streamlined and edited,
physics conclusions and reasoning not change
Sensitivity of Azimuthal Jet Tomography to Early Time Energy-Loss at RHIC and LHC
We compute the jet path-length dependence of energy-loss for higher azimuthal
harmonics of jet-fragments in a generalized model of energy-loss that can
interpolate between pQCD and AdS/CFT limits and compare results with Glauber
and CGC/KLN initial conditions. We find, however, that even the high-pT second
moment is most sensitive to the poorly known early-time evolution during the
first fm/c. Moreover, we demonstrate that quite generally the energy and
density-dependence leads to an overquenching jet fragments relative to the
first LHC -data, once the parameters of the energy-loss model are fixed
from -data at RHIC.Comment: 4 pages, 2 figures, version accepted for publication in J. Phys. G:
Nucl. Part. Phys. as conference proceedings for Quark Matter 2011, May 23 -
May 28, Annecy, Franc
Sodium Transport in Capillaries Isolated from Rat Brain
Brain capillary endothelial cells form a bloodbrain barrier (BBB) that appears to play a role in fluid and ion homeostasis in brain. One important transport system that may be involved in this regulatory function is the Na + ,K + -ATPase that was previously demonstrated to be present in isolated brain capillaries. The goal of the present study was to identify additional Na + transport systems in brain capillaries that might contribute to BBB function. Microvessels were isolated from rat brains and 22 Na + uptake by and efflux from the cells were studied. Total 22 Na + uptake was increased and the rate of 22 Na + efflux was decreased by ouabain, confirming the presence of Na + ,K + -ATPase in capillary cells. After inhibition of Na + ,K + -ATPase activity, another saturable Na + transport mechanism became apparent. Capillary uptake of 22 Na + was stimulated by an elevated concentration of Na + or H + inside the cells and inhibited by extracellular Na + , H + , Li + , and NH 4 + . Amiloride inhibited 22 Na + uptake with a K i between 10 −5 and 10 −6 M but there was no effect of 1 mM furosemide on 22 Na + uptake by the isolated microvessels. These results indicate the presence in brain capillaries of a transport system capable of mediating Na + / Na + and Na + /H + exchange. As a similar transport system does not appear to be present on the luminal membrane of the brain capillary endothelial cell, it is proposed that Na + /H + exchange occurs primarily across the antiluminal membrane.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/66187/1/j.1471-4159.1983.tb09065.x.pd
Derivation of fluid dynamics from kinetic theory with the 14--moment approximation
We review the traditional derivation of the fluid-dynamical equations from
kinetic theory according to Israel and Stewart. We show that their procedure to
close the fluid-dynamical equations of motion is not unique. Their approach
contains two approximations, the first being the so-called 14-moment
approximation to truncate the single-particle distribution function. The second
consists in the choice of equations of motion for the dissipative currents.
Israel and Stewart used the second moment of the Boltzmann equation, but this
is not the only possible choice. In fact, there are infinitely many moments of
the Boltzmann equation which can serve as equations of motion for the
dissipative currents. All resulting equations of motion have the same form, but
the transport coefficients are different in each case.Comment: 15 pages, 3 figures, typos fixed and discussions added; EPJA: Topical
issue on "Relativistic Hydro- and Thermodynamics
Ontogeny of synaptophysin and synaptoporin in the central nervous system
The expression of the synaptic vesicle antigens synaptophysin (SY) and synaptoporin (SO) was studied in the rat striatum, which contains a nearly homogeneous population of GABAergic neurons. In situ hybridization revealed high levels of SY transcripts in the striatal anlage from embryonic day (E) 14 until birth. In contrast. SO hybridization signals were low, and no immunoreactive cell bodies were detected at these stages of development. At E 14, SY-immunoreactivity was restricted to perikarya. In later prenatal stages of development SY-immunoreactivity appeared in puncta (identified as terminals containing immunostained synaptic vesicles), fibers, thick fiber bundles and ‘patches’. In postnatal and adult animals, perikarya of striatal neurons exhibited immunoreaction for SO; ultrastructurally SO antigen was found in the Golgi apparatus and in multivesicular bodies. SO-positive boutons were rare in the striatum. In the neuropil, numerous presynaptic terminals positive for SY were observed. Our data indicate that the expression of synaptic vesicle proteins in GABAergic neurons of the striatum is developmentally regulated. Whereas SY is prevalent during embryonic development, SO is the major synaptic vesicle antigen expressed postnatally by striatal neurons which project to the globus pallidus and the substantia nigra. In contrast synapses of striatal afferents (predominantly from cortex, thalamus and substantia nigra) contain SY
Use of genetically modified muscle and fat grafts to repair defects in bone and cartilage
We report a novel technology for the rapid healing of large osseous and chondral defects, based upon the genetic modification of autologous skeletal muscle and fat grafts. These tissues were selected because they not only possess mesenchymal progenitor cells and scaffolding properties, but also can be biopsied, genetically modified and returned to the patient in a single operative session. First generation adenovirus vector carrying cDNA encoding human bone morphogenetic protein-2 (Ad.BMP-2) was used for gene transfer to biopsies of muscle and fat. To assess bone healing, the genetically modified ("gene activated") tissues were implanted into 5mm-long critical size, mid-diaphyseal, stabilized defects in the femora of Fischer rats. Unlike control defects, those receiving gene-activated muscle underwent rapid healing, with evidence of radiologic bridging as early as 10 days after implantation and restoration of full mechanical strength by 8 weeks. Histologic analysis suggests that the grafts rapidly differentiated into cartilage, followed by efficient endochondral ossification. Fluorescence in situ hybridization detection of Y-chromosomes following the transfer of male donor muscle into female rats demonstrated that at least some of the osteoblasts of the healed bone were derived from donor muscle. Gene activated fat also healed critical sized defects, but less quickly than muscle and with more variability. Anti-adenovirus antibodies were not detected. Pilot studies in a rabbit osteochondral defect model demonstrated the promise of this technology for healing cartilage defects. Further development of these methods should provide ways to heal bone and cartilage more expeditiously, and at lower cost, than is presently possible
Electro-Magnetic Nucleon Form Factors and their Spectral Functions in Soliton Models
It is demonstrated that in simple soliton models essential features of the
electro-magnetic nucleon form factors observed over three orders of magnitude
in momentum transfer are naturally reproduced. The analysis shows that
three basic ingredients are required: an extended object, partial coupling to
vector mesons, and relativistic recoil corrections. We use for the extended
object the standard skyrmion, one vector meson propagator for both isospin
channels, and the relativistic boost to the Breit frame. Continuation to
timelike leads to quite stable results for the spectral functions in the
regime from the 2- or 3-pion threshold to about two rho masses. Especially the
onset of the continuous part of the spectral functions at threshold can be
reliably determined and there are strong analogies to the results imposed on
dispersion theoretic approaches by the unitarity constraint.Comment: 24 pages, (RevTeX), 5 PS-figures; Data points in fig.2 and
corresponding references added. Final version, to be published in Z.Physik
A consistent first-order model for relativistic heat flow
This paper revisits the problem of heat conduction in relativistic fluids,
associated with issues concerning both stability and causality. It has long
been known that the problem requires information involving second order
deviations from thermal equilibrium. Basically, any consistent first-order
theory needs to remain cognizant of its higher-order origins. We demonstrate
this by carrying out the required first-order reduction of a recent variational
model. We provide an analysis of the dynamics of the system, obtaining the
conditions that must be satisfied in order to avoid instabilities and acausal
signal propagation. The results demonstrate, beyond any reasonable doubt, that
the model has all the features one would expect of a real physical system. In
particular, we highlight the presence of a second sound for heat in the
appropriate limit. We also make contact with previous work on the problem by
showing how the various constraints on our system agree with previously
established results.Comment: RevTeX, 1 eps Figur
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