テイGlycemic Indexショクヒン ワ 2ガタトウニョウビョウ ノ ハッショウ オ ヨボウシ チリョウ コウカ オ ユウスル

The incidence of type 2 diabetes should be reduced either by decreasing insulin demand or by improving insulin sensitivity. Jenkins et al . found that a low-glycemic-index diet containing mainly intact whole grains significantly reduced C-peptide concentrations compared with a high-glycemic-index diet containing primarily refined grain product. In this review, we examine evidence relating dietary glycemic index and glycemic load to type 2 diabetes incidence and the role of the form of dietary carbohydrate in the management of diabetes. Both metabolic and epidemiologic evidence suggests that replacing high-glycemic-index forms of carbohydrate with low-glycemic-index carbohydrate will reduce the risk of type 2 diabetes. Among patients with diabetes, the weight of evidence suggests that replacing high-glycemic-index with low-glycemic-index forms of carbohydrate will improve glycemic control and reduce hypoglycemic episodes among those treated with insulin

Increasing early insulin secretion compensate adequately for hepatic insulin resistance in CCl4-induced cirrhosis rats

A number of recent publications have reported an increased frequency prevalence of glucose intolerance with hyperinsulinemia in liver cirrhosis. The aim of this work was to detect, in CCl4-induced liver cirrhosis rat, the presence and starting point of muscle and liver insulin resistance. Eighteen rats received intraperitoneal injection of 2 ml of soybean oil containing of CCl4 twice a week for 20 weeks. We executed standard oral glucose tolerance and clamp study to evaluate systemic insulin resistance. Hepatic glucose uptake was much lower in CCl4 group than that in control group, but peripheral glucose uptake was not decreased in this study. In contrast, early-phase insulin secretion was enhanced in CCl4 rat using oral glucose load during clamp methods. These data suggested that increased early insulin secretion compensate adequately for hepatic insulin resistance in rats. However there was a report that peripheral glucose uptake was decreased in the case of human liver cirrhosis, which was formed in the course of time. In a chronic condition, this may be associated with reduced insulin content and developed systemic insulin resistance in liver cirrhosis. Then a long term observation study will be required to examine the presence of muscle insulin resistance in liver cirrhosis

イメージスキャン画像分割アーキテクチャのLSI設計

画像認識処理技術,特にリアルタイムでの動物体検出には,膨大な視覚情報を処理する必要がある.画像分割処理は画像中に存在する物体を抽出する処理で,オブジェクトベースの画像認識などには欠かせない前処理である.これまでに我々は領域成長に基づくイメージスキャン画像分割アーキテクチャを提案した.同アーキテクチャは処理回路 (以下ISE) のサイズとメモリの構成を変えることで,アプリケーションに応じて処理速度と処理回路サイズの最適化が可能な柔軟性を有する.本研究では,同アーキテクチャのボトルネックとなるリーダセル検索を非同期化を行い,180 nm CMOS プロセスでASIC設計を行った

Increasing early insulin secretion compensate adequately for hepatic insulin resistance in CCl4-induced cirrhosis rats.

A number of recent publications have reported an increased frequency prevalence of glucose intolerance with hyperinsulinemia in liver cirrhosis. The aim of this work was to detect, in CCl(4)-induced liver cirrhosis rat, the presence and starting point of muscle and liver insulin resistance. Eighteen rats received intraperitoneal injection of 2 ml of soybean oil containing of CCl(4) twice a week for 20 weeks. We executed standard oral glucose tolerance and clamp study to evaluate systemic insulin resistance. Hepatic glucose uptake was much lower in CCl(4) group than that in control group, but peripheral glucose uptake was not decreased in this study. In contrast, early-phase insulin secretion was enhanced in CCl(4) rat using oral glucose load during clamp methods. These data suggested that increased early insulin secretion compensate adequately for hepatic insulin resistance in rats. However there was a report that peripheral glucose uptake was decreased in the case of human liver cirrhosis, which was formed in the course of time. In a chronic condition, this may be associated with reduced insulin content and developed systemic insulin resistance in liver cirrhosis. Then a long term observation study will be required to examine the presence of muscle insulin resistance in liver cirrhosis.Journal ArticleResearch Support, Non-U.S. Gov'tinfo:eu-repo/semantics/publishe