Rethinking the adversary and operational characteristics of deniable storage

Aim: With the widespread adoption of disk encryption technologies, it has become common for adversaries to employ coercive tactics to force users to surrender encryption keys. For some users, this creates a need for hidden volumes that provide plausible deniability, the ability to deny the existence of sensitive information. Previous deniable storage solutions only offer pieces of an implementable solution that do not take into account more advanced adversaries, such as intelligence agencies, and operational concerns. Specifically, they do not address an adversary that is familiar with the design characteristics of any deniable system.Methods: We evaluated existing threat models and deniable storage system designs to produce a new, stronger threat model and identified design characteristics necessary in a plausibly deniable storage system. To better explore the implications of this stronger adversary, we developed Artifice, the first tunable, operationally secure, self repairing, and fully deniable storage system.Results: With Artifice, hidden data blocks are split with an information dispersal algorithm such as Shamir Secret Sharing to produce a set of obfuscated carrier blocks that are indistinguishable from other pseudorandom blocks on the disk. The blocks are then stored in unallocated space of an existing file system. The erasure correcting capabilities of an information dispersal algorithm allow Artifice to self repair damage caused by writes to the public file system. Unlike preceding systems, Artifice addresses problems regarding flash storage devices and multiple snapshot attacks through simple block allocation schemes and operational security measures. To hide the user’s ability to run a deniable system and prevent information leakage, a user accesses Artifice through a separate OS stored on an external Linux live disk.Conclusion: In this paper, we present a stronger adversary model and show that our proposed design addresses the primary weaknesses of existing approaches to deniable storage under this stronger assumed adversary

Role of p38 Mitogen-Activated Protein Kinase in Middle Ear Mucosa Hyperplasia during Bacterial Otitis Media

Hyperplasia of the middle ear mucosa contributes to the sequelae of acute otitis media. Understanding the signal transduction pathways that mediate hyperplasia could lead to the development of new therapeutic interventions for this disease and its sequelae. Endotoxin derived from bacteria involved in middle ear infection can contribute to the hyperplastic response. The p38 mitogen-activated protein kinase (MAPK) is known to be activated by endotoxin as well as cytokines and other inflammatory mediators that have been documented in otitis media. We assessed the activation of p38 in the middle ear mucosa of an in vivo rat bacterial otitis media model. Strong activity of p38 was observed 1 to 6 h after bacterial inoculation. Activity continued at a lower level for at least 7 days. The effects of p38 activation were assessed using an in vitro model of rat middle ear mucosal hyperplasia in which mucosal growth is stimulated by nontypeable Haemophilus influenzae during acute otitis media. Hyperplastic mucosal explants treated with the p38α and p38β inhibitor SB203580 demonstrated significant inhibition of otitis media-stimulated mucosal growth. The results of this study suggest that intracellular signaling via p38 MAPK influences the hyperplastic response of the middle ear mucosa during bacterial otitis media