17 research outputs found
Electrocardiographic and vectorcardiographic findings in asymptomatic young men with primary T-wave abberations in the electrocardiogram. Relationship to sympatho-adrenal activity at rest and during different stress tests
Electrocardiographic abnormalities during exercise 3 weeks to 18 months after anterior myocardial infarction.
Sympathoadrenal and cardiovascular responses to mental stress, isometric handgrip, and cold pressor test in asymptomatic young men with primary T wave abnormalities in the electrocardiogram.
Sudden cardiac death in the young in Sweden: electrocardiogram in relation to forensic diagnosis
The Role of Stress and the Sympathetic Nervous System in Hypertension and Ischemic Heart Disease: Advantages of Therapy with β-Receptor Blockers
Effect of intravenous adrenaline on electrocardiogram, blood pressure, and serum potassium.
Increased catecholamines after myocardial infarction may contribute to the development of arrhythmias. We have infused adrenaline intravenously in nine normal volunteers to levels similar to those seen after myocardial infarction. Adrenaline caused an increase in systolic blood pressure, a decrease in diastolic blood pressure, and an increase in heart rate. Adrenaline also produced a decrease in T wave amplitude and an increase in the QTc interval. The serum potassium fell dramatically during the adrenaline infusion from a control value of 4.06 mmol/l to 3.22 mmol/l. Hypokalaemia after myocardial infarction is associated with an increased incidence of ventricular arrhythmias. Thus, circulating adrenaline may increase the frequency of arrhythmias both directly via changes in ventricular repolarisation and indirectly via adrenaline induced hypokalaemia