Supplementary Figure 3 from Tumor-Derived CCL5 Recruits Cancer-Associated Fibroblasts and Promotes Tumor Cell Proliferation in Esophageal Squamous Cell Carcinoma

ERK1/2 signaling pathway in CCL5 knockout cells.</p

Supplementary Figure 1 from Tumor-Derived CCL5 Recruits Cancer-Associated Fibroblasts and Promotes Tumor Cell Proliferation in Esophageal Squamous Cell Carcinoma

Confirmation of CCL5 knockout clones.</p

Supplementary Figure 4 from Tumor-Derived CCL5 Recruits Cancer-Associated Fibroblasts and Promotes Tumor Cell Proliferation in Esophageal Squamous Cell Carcinoma

Targeted CCR5 inhibition replicates CCL5 loss with reduced tumor cell proliferation in vitro and in vivo.</p

Supplementary Figure Legends from Tumor-Derived CCL5 Recruits Cancer-Associated Fibroblasts and Promotes Tumor Cell Proliferation in Esophageal Squamous Cell Carcinoma

Supplementary Figure Legends 1-4</p

Supplementary Figure 2 from Tumor-Derived CCL5 Recruits Cancer-Associated Fibroblasts and Promotes Tumor Cell Proliferation in Esophageal Squamous Cell Carcinoma

Loss of tumor-derived CCL5 reduces tumor cell proliferation in TE-1 cells.</p

Supplementary Figure S6 from p53 Gain-of-Function Mutation Induces Metastasis via BRD4-Dependent CSF-1 Expression

Supplementary Figure S6. Graphical abstract depicting the pro-metastatic role and mechanism of Trp53R172H-dependent Csf-1/Csf-1r signaling.</p

Supplementary Table S2 from p53 Gain-of-Function Mutation Induces Metastasis via BRD4-Dependent CSF-1 Expression

Supplementary Table S2. List of antibodies and dilutions used in the study.</p

Supplementary Table S1 from p53 Gain-of-Function Mutation Induces Metastasis via BRD4-Dependent CSF-1 Expression

Supplementary Table S1. List of oligonucleotides used in the study.</p

Supplementary Figure S4 from p53 Gain-of-Function Mutation Induces Metastasis via BRD4-Dependent CSF-1 Expression

Supplementary Figure S4. Depletion of Brd4 reduces Csf1 secretion and ESCC tumor cell invasion.</p

Supplementary Figure S2 from p53 Gain-of-Function Mutation Induces Metastasis via BRD4-Dependent CSF-1 Expression

Supplementary Figure S2. Trp53R172H-dependent Csf-1/Csf-1r signaling promotes metastasis only in the presence of p53-R172H, but not p53 null or wild-type p53.</p