Quotas to accelerate gender equity in sport leadership: do they work?

This chapter explores the use of gender quotas as a strategy to accelerate the growth of women in sport leadership, particularly in the governance of national sport organisations (NSOs) and international federations (IFs). First, I present an overview of the current global status of women’s participation in sport governance based on the Sydney Scoreboard, a global index for women in sport leadership. This provides compelling evidence that only limited progress has been made to date and gender equity in sport governance remains elusive. Second, I discuss several strategies for disrupting the status quo at an international level, including the Brighton Plus Helsinki Declaration and an important initiative by the United Nations (Adriaanse & Claringbould, 2016) as well as the introduction of gender quotas. The latter is controversial

David or Mia? The influence of gender on adolescent girls' choice of sport role models

This article explores the relationship between adolescent girls and their sport role models with a particular focus on gender. Social learning theory as well as gender theory are considered. Based on quantitative and qualitative data collected in Australia through two focus group interviews and questionnaires (n = 357) the researchers found that gender influences the choice of sport role models of adolescent girls. When asked to nominate their role model, girls overwhelmingly choose a female role model, however, when their role model came from the sporting domain the percentage of female role models decreased. Further, qualitative comments support the notion that gender and gendered heroism play a critical role in the adoption of sport role models by adolescent girls. The authors argue that sport role models, in traditional gender terms, are ideologically contradictory. A trend towards acceptance of a perspective of multiple femininities and masculinities may be a prospect for change. © 2008 Elsevier Ltd. All rights reserved

Gender equality in sport leadership: From the Brighton Declaration to the Sydney Scoreboard

This study investigated the development of the legacies of the five World Conferences on Women and Sport that have been convened by the International Working Group on Women and Sport from 1994 to 2010. In particular, it examined the ways in which gender is constructed in these legacies in relation to gender equality in sport leadership. The theoretical framework was drawn from Connell’s four-dimensional gender model, which suggests that gender relations can be characterized in terms of four interwoven dimensions of social life: production, power, emotion and symbolism. The method used was a comparative case study of five legacies. We conducted a content analysis of documents relevant to the five legacies. Findings show that, in all five legacies, gender in relation to sport leadership was mainly constructed on the dimension of production and power relations (more women in leadership positions) and symbolic relations (creating a sporting culture that values women’s participation at all levels). By contrast, the gendered dimension of emotional relations – collaboration between men and women – received limited attention. The implications of these findings for the acceleration of gender equality in sport leadership are discussed

Sarcopenic obesity and risk of new onset depressive symptoms in older adults: English Longitudinal Study of Ageing

We examined the role of sarcopenic obesity as a risk factor for new-onset depressive symptoms over 6-year follow-up in a large sample of older adults

Pituitary-hormone secretion by thyrotropinomas

Hormone secretion by somatotropinomas, corticotropinomas and prolactinomas exhibits increased pulse frequency, basal and pulsatile secretion, accompanied by greater disorderliness. Increased concentrations of growth hormone (GH) or prolactin (PRL) are observed in about 30% of thyrotropinomas leading to acromegaly or disturbed sexual functions beyond thyrotropin (TSH)-induced hyperthyroidism. Regulation of non-TSH pituitary hormones in this context is not well understood. We there therefore evaluated TSH, GH and PRL secretion in 6 patients with up-to-date analytical and mathematical tools by 24-h blood sampling at 10-min intervals in a clinical research laboratory. The profiles were analyzed with a new deconvolution method, approximate entropy, cross-approximate entropy, cross-correlation and cosinor regression. TSH burst frequency and basal and pulsatile secretion were increased in patients compared with controls. TSH secretion patterns in patients were more irregular, but the diurnal rhythm was preserved at a higher mean with a 2.5 h phase delay. Although only one patient had clinical acromegaly, GH secretion and IGF-I levels were increased in two other patients and all three had a significant cross-correlation between the GH and TSH. PRL secretion was increased in one patient, but all patients had a significant cross-correlation with TSH and showed decreased PRL regularity. Cross-ApEn synchrony between TSH and GH did not differ between patients and controls, but TSH and PRL synchrony was reduced in patients. We conclude that TSH secretion by thyrotropinomas shares many characteristics of other pituitary hormone-secreting adenomas. In addition, abnormalities in GH and PRL secretion exist ranging from decreased (joint) regularity to overt hypersecretion, although not always clinically obvious, suggesting tumoral transformation of thyrotrope lineage cells

Key mechanisms governing resolution of lung inflammation

Innate immunity normally provides excellent defence against invading microorganisms. Acute inflammation is a form of innate immune defence and represents one of the primary responses to injury, infection and irritation, largely mediated by granulocyte effector cells such as neutrophils and eosinophils. Failure to remove an inflammatory stimulus (often resulting in failed resolution of inflammation) can lead to chronic inflammation resulting in tissue injury caused by high numbers of infiltrating activated granulocytes. Successful resolution of inflammation is dependent upon the removal of these cells. Under normal physiological conditions, apoptosis (programmed cell death) precedes phagocytic recognition and clearance of these cells by, for example, macrophages, dendritic and epithelial cells (a process known as efferocytosis). Inflammation contributes to immune defence within the respiratory mucosa (responsible for gas exchange) because lung epithelia are continuously exposed to a multiplicity of airborne pathogens, allergens and foreign particles. Failure to resolve inflammation within the respiratory mucosa is a major contributor of numerous lung diseases. This review will summarise the major mechanisms regulating lung inflammation, including key cellular interplays such as apoptotic cell clearance by alveolar macrophages and macrophage/neutrophil/epithelial cell interactions. The different acute and chronic inflammatory disease states caused by dysregulated/impaired resolution of lung inflammation will be discussed. Furthermore, the resolution of lung inflammation during neutrophil/eosinophil-dominant lung injury or enhanced resolution driven via pharmacological manipulation will also be considered

Creating and Breaking Habit in Healthcare Professional Behaviours to Improve Healthcare and Health

Healthcare professionals (HCPs) prescribe, provide advice, conduct examinations, perform surgical procedures, and engage in a range of clinical behaviours. Their clinical actions are characteristically performed repeatedly—sometimes multiple times per day—in the same physical locations with the same colleagues and patients, under constant time pressure, and competing demands. This repetition under pressure in a stable setting provides ideal circumstances for creating contingencies between physical and social cues and clinical actions. HCP behaviour provides an ideal setting in which to advance theory, methods, and interventions to better understand habit formation and habit reversal. Contemporary theoretical and methodological development in the psychology of habit has begun to be applied to understand and promote the formation, breaking, and replacement of habitual behaviour in HCPs. This chapter highlights key theoretical approaches, methods, and intervention techniques that have been applied to conceptualize, measure, develop, and break habit and automaticity in HCPs. These insights have the potential to synergistically contribute novel perspectives to the wider habit literature