An Augmented Interface to Display Industrial Robot Faults

Technology advancement is changing the way industrial factories have to face an increasingly complex and competitive market. The fourth industrial revolution (known as industry 4.0) is also changing how human workers have to carry out tasks and actions. In fact, it is no longer impossible to think of a scenario in which human operators and industrial robots work side-by-side, sharing the same environment and tools. To realize a safe work environment, workers should trust robots as well as they trust human operators. Such goal is indeed complex to achieve, especially when workers are under stress conditions, such as when a fault occurs and the human operators are no longer able to understand what is happening in the industrial manipulator. Indeed, Augmented Reality (AR) can help workers to visualize in real-time robots’ faults. This paper proposes an augmented system that assists human workers to recognize and visualize errors, improving their awareness of the system. The system has been tested using both an AR see-through device and a smartphone

Accelerated functional brain aging in pre-clinical familial Alzheimer’s disease

Resting state functional connectivity (rs-fMRI) is impaired early in persons who subsequently develop Alzheimer’s disease (AD) dementia. This impairment may be leveraged to aid investigation of the pre-clinical phase of AD. We developed a model that predicts brain age from resting state (rs)-fMRI data, and assessed whether genetic determinants of AD, as well as beta-amyloid (Aβ) pathology, can accelerate brain aging. Using data from 1340 cognitively unimpaired participants between 18–94 years of age from multiple sites, we showed that topological properties of graphs constructed from rs-fMRI can predict chronological age across the lifespan. Application of our predictive model to the context of pre-clinical AD revealed that the pre-symptomatic phase of autosomal dominant AD includes acceleration of functional brain aging. This association was stronger in individuals having significant Aβ pathology