3 research outputs found
Pancreatic Ī²-cell signaling: toward better understanding of diabetes and its treatment
- Author
- Brown J.C., Mutt, V. and Pederson,
- Cras-Mé
- Desai B.M., Oliver-Krasinski, J.,
- Eliasson L., Abdulkader, F., Braun
- Eliasson L., Ma, X., Renstrom, E.,
- Elrick H., Stimmler, L., Hlad, C.J
- Fujimoto W., Miki, T., Ogura, T.,
- Holst J.J. and Gromada, J.
- McIntyre N., Holdsworth, C.D. and
- Miki T., Liss, B., Minami, K., Shi
- Miki T., Minami, K., Shinozaki, H.
- Okuno M., Minami, K., Okumachi, A.
- Prentki M. and Matschinsky, F.M.
- RAJAGOPAL J
- Seino S., Takahashi, H., Fujimoto,
- Srinivas S., Watanabe, T., Lin, C.
- Suzuki H., Fukushima, M., Okamoto,
- Ueno H., Shibasaki, T., Iwanaga, T
- Vilsboll T., Krarup, T., Madsbad,
- Wollheim C.B. and Sharp, G.W.
- Publication venue
- The Japan Academy
- Publication date
- 01/01/2010
- Field of study
Pancreatic Ī²-cells play a central role in the maintenance glucose homeostasis by secreting insulin, a key hormone that regulates blood glucose levels. Dysfunction of the Ī²-cells and/or a decrease in the Ī²-cell mass are associated closely with the pathogenesis and pathophysiology of diabetes mellitus, a major metabolic disease that is rapidly increasing worldwide. Clarification of the mechanisms of insulin secretion and Ī²-cell fate provides a basis for the understanding of diabetes and its better treatment. In this review, we discuss cell signaling critical for the insulin secretory function based on our recent studies
Comparison of indomethacin and phenylbutazone in acute gout.
- Author
- Publication venue
- 'BMJ'
- Publication date
- Field of study
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