638 research outputs found

    Supercurrent tunneling between conventional and unconventional superconductors: A Ginzburg-Landau approach

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    We investigate the Josephson tunneling between a conventional and an unconventional superconductor via a Ginzburg-Landau theory. This approach allows us to write down the general form of the Josephson coupling between the two superconductors, and to see which terms are forbidden or allowed by spatial symmetries. The time-reversal symmetry is also considered. We discuss the current-phase relationships, magnetic, and ac effects if we just include this direct coupling to the unconventional superconductor. In addition we consider the Josephson coupling between two short-coherence-length superconductors, extending the work of Deutscher and Müller (DM) to a finite-current calculation. We find that the critical current is suppressed below the DM value due to the fact that the coupling between the two superconductors across the junction depends on the phase difference and hence the current itself. Finally we investigate the possibility of the proximity effect, in particular the possibility that the conventional-type pairing is induced and hence coexists with the unconventional pairing near the junction. This would give the dominant contribution to the tunneling current if the direct tunneling to the unconventional pairs are suppressed for some reason. We point out that there is no possibility of dissipationless tunneling above the transition temperature of the unconventional superconductor. Even in the case in which the unconventional superconductor is below its transition temperature, we find that, for the possibility of a dissipationless current, it is crucial to have a coupling between the induced s wave and the unconventional superconductor that depends on their phase difference, which allows the conversion of the supercurrent from one type to the other. The behavior of this current, in particular as a function of temperature, is discussed. We also discuss the magnetic and time-dependent effects of the junction in the presence of this proximity effect. We see that, while some of these remain unaffected, some, in particular the time-dependent processes, are affected in a rather nontrivial manner

    Master Operators Govern Multifractality in Percolation

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    Using renormalization group methods we study multifractality in percolation at the instance of noisy random resistor networks. We introduce the concept of master operators. The multifractal moments of the current distribution (which are proportional to the noise cumulants CR(l)(x,x′)C_R^{(l)} (x, x^\prime) of the resistance between two sites x and x′x^\prime located on the same cluster) are related to such master operators. The scaling behavior of the multifractal moments is governed exclusively by the master operators, even though a myriad of servant operators is involved in the renormalization procedure. We calculate the family of multifractal exponents ψl{\psi_l} for the scaling behavior of the noise cumulants, CR(l)(x,x′)∼∣x−x′∣ψl/νC_R^{(l)} (x, x^\prime) \sim | x - x^\prime |^{\psi_l /\nu}, where ν\nu is the correlation length exponent for percolation, to two-loop order.Comment: 6 page

    Logarithmic Corrections in Dynamic Isotropic Percolation

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    Based on the field theoretic formulation of the general epidemic process we study logarithmic corrections to scaling in dynamic isotropic percolation at the upper critical dimension d=6. Employing renormalization group methods we determine these corrections for some of the most interesting time dependent observables in dynamic percolation at the critical point up to and including the next to leading correction. For clusters emanating from a local seed at the origin we calculate the number of active sites, the survival probability as well as the radius of gyration.Comment: 9 pages, 3 figures, version to appear in Phys. Rev.

    Human cachexia induces changes in mitochondria, autophagy and apoptosis in the skeletal muscle

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    Cachexia is a wasting syndrome characterized by the continuous loss of skeletal muscle mass due to imbalance between protein synthesis and degradation, which is related with poor prognosis and compromised quality of life. Dysfunctional mitochondria are associated with lower muscle strength and muscle atrophy in cancer patients, yet poorly described in human cachexia. We herein investigated mitochondrial morphology, autophagy and apoptosis in the skeletal muscle of patients with gastrointestinal cancer-associated cachexia (CC), as compared with a weight-stable cancer group (WSC). CC showed prominent weight loss and increased circulating levels of serum C-reactive protein, lower body mass index and decreased circulating hemoglobin, when compared to WSC. Electron microscopy analysis revealed an increase in intermyofibrillar mitochondrial area in CC, as compared to WSC. Relative gene expression of Fission 1, a protein related to mitochondrial fission, was increased in CC, as compared to WSC. LC3 II, autophagy-related (ATG) 5 and 7 essential proteins for autophagosome formation, presented higher content in the cachectic group. Protein levels of phosphorylated p53 (Ser46), activated caspase 8 (Asp384) and 9 (Asp315) were also increased in the skeletal muscle of CC. Overall, our results demonstrate that human cancer-associated cachexia leads to exacerbated muscle-stress response that may culminate in muscle loss, which is in part due to disruption of mitochondrial morphology, dysfunctional autophagy and increased apoptosis. To the best of our knowledge, this is the first report showing quantitative morphological alterations in skeletal muscle mitochondria in cachectic patients

    Asymptotic localization of symbol correspondences for spin systems and sequential quantizations of S2S^2

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    Quantum or classical mechanical systems symmetric under SU(2)SU(2) are called spin systems. A SU(2)SU(2)-equivariant map from (n+1)(n+1)-square matrices to functions on the 22-sphere, satisfying some basic properties, is called a spin-jj symbol correspondence (n=2j∈Nn=2j\in\mathbb N). Given a spin-jj symbol correspondence, the matrix algebra induces a twisted jj-algebra of symbols. In this paper, we establish a new, more intuitive criterion for when the Poisson algebra of smooth functions on the 22-sphere emerges asymptotically (n→∞n\to\infty) from the sequence of twisted jj-algebras. This more geometric criterion, which in many cases is equivalent to the numerical criterion obtained in [Rios&Straume], is given in terms of a classical (asymptotic) localization of symbols of all projectors (quantum pure states) in a certain family. For some important kinds of symbol correspondence sequences, such a classical localization condition is equivalent to asymptotic emergence of the Poisson algebra. But in general, this classical localization condition is stronger than Poisson emergence. We thus also consider some weaker notions of asymptotic localization of projector-symbols. Finally, we obtain some relations between asymptotic localization of a symbol correspondence sequence and its sequential quantizations of the classical spin system, after carefully developing a theory of sequential quantizations of smooth functions on S2S^2 and their asymptotic actions on a ground Hilbert space.Comment: slight edition of expanded version, 56 page

    Doses de nitrogenio no crescimento e producao inicial do mamoeiro, sob gotejamento, no litoral piauiense.

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    Com objetivo de avaliar o efeito de doses de nitrogenio no crescimento e producao inicial do mamoeiro irrigado, instalou-se um experimento na estacao experimental da Embrapa Meio-Norte, em Parnaiba, PI, em solo pertencente a Unidade de Mapeamento Areias Quartzosas alicas e distroficas A fraco e moderado fase caatinga litoranea, relevo plano.bitstream/item/83331/1/CT970001.pd

    On the relevance of percolation theory to the vulcanization transition

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    The relationship between vulcanization and percolation is explored from the perspective of renormalized local field theory. We show rigorously that the vulcanization and percolation correlation functions are governed by the same Gell--Mann-Low renormalization group equation. Hence, all scaling aspects of the vulcanization transition are reigned by the critical exponents of the percolation universality class.Comment: 9 pages, 2 figure

    Percolation Threshold, Fisher Exponent, and Shortest Path Exponent for 4 and 5 Dimensions

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    We develop a method of constructing percolation clusters that allows us to build very large clusters using very little computer memory by limiting the maximum number of sites for which we maintain state information to a number of the order of the number of sites in the largest chemical shell of the cluster being created. The memory required to grow a cluster of mass s is of the order of sθs^\theta bytes where θ\theta ranges from 0.4 for 2-dimensional lattices to 0.5 for 6- (or higher)-dimensional lattices. We use this method to estimate dmind_{\scriptsize min}, the exponent relating the minimum path ℓ\ell to the Euclidean distance r, for 4D and 5D hypercubic lattices. Analyzing both site and bond percolation, we find dmin=1.607±0.005d_{\scriptsize min}=1.607\pm 0.005 (4D) and dmin=1.812±0.006d_{\scriptsize min}=1.812\pm 0.006 (5D). In order to determine dmind_{\scriptsize min} to high precision, and without bias, it was necessary to first find precise values for the percolation threshold, pcp_c: pc=0.196889±0.000003p_c=0.196889\pm 0.000003 (4D) and pc=0.14081±0.00001p_c=0.14081\pm 0.00001 (5D) for site and pc=0.160130±0.000003p_c=0.160130\pm 0.000003 (4D) and pc=0.118174±0.000004p_c=0.118174\pm 0.000004 (5D) for bond percolation. We also calculate the Fisher exponent, τ\tau, determined in the course of calculating the values of pcp_c: τ=2.313±0.003\tau=2.313\pm 0.003 (4D) and τ=2.412±0.004\tau=2.412\pm 0.004 (5D)

    Relações entre a contribuição da fixação biológica de nitrogênio e a duração do ciclo de diferentes genótipos de cultivos de leguminosas de grãos.

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    Contribuição da FBN por culturas de diferentes ciclos; A senescência dos nódulos como fator limitante da FBN em genótipos de ciclo curto; Mecanismos da senescência precoce dos nódulos.bitstream/CPAMN-2010/24174/1/doc197.pd
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