54,312 research outputs found

    Enhanced Macroscopic Quantum Tunneling in Bi2_2Sr2_2CaCu2_2O8+δ_{8+\delta} Intrinsic Josephson Junction Stacks

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    We have investigated macroscopic quantum tunneling in Bi2_2Sr2_2CaCu2_2O8+δ_{8+\delta} intrinsic Josephson junctions at millikelvin temperatures using microwave irradiation. Measurements show that the escape rate for uniformly switching stacks of N junctions is about N2N^2 times higher than that of a single junction having the same plasma frequency. We argue that this gigantic enhancement of macroscopic quantum tunneling rate in stacks is boosted by current fluctuations which occur in the series array of junctions loaded by the impedance of the environment.Comment: 4 pages and 5 figure

    Bulk Superconductivity at 14 K in Single Crystals of Fe1+yTexSe1-x

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    Resistivity, magnetic susceptibility and heat capacity measurements are reported for single crystals of Fe1+yTexSe1-x grown via a modified Bridgeman method with 0 < y < 0.15, and x= 1, 0.9, 0.75, 0. 67, 0.55 and 0.5. Although resistivity measurements show traces of superconductivity near 14 K for all x except x=1, only crystals grown with compositions near x=0.5 exhibit bulk superconductivity. The appearance of bulk superconductivity correlates with a reduction in the magnitude of the magnetic susceptibility at room temperature and smaller values of y, the concentration of Fe in the Fe(2) site.Comment: Submitted to Phys. Rev.

    Thermodynamics of lattice QCD with 2 sextet quarks on N_t=8 lattices

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    We continue our lattice simulations of QCD with 2 flavours of colour-sextet quarks as a model for conformal or walking technicolor. A 2-loop perturbative calculation of the β\beta-function which describes the evolution of this theory's running coupling constant predicts that it has a second zero at a finite coupling. This non-trivial zero would be an infrared stable fixed point, in which case the theory with massless quarks would be a conformal field theory. However, if the interaction between quarks and antiquarks becomes strong enough that a chiral condensate forms before this IR fixed point is reached, the theory is QCD-like with spontaneously broken chiral symmetry and confinement. However, the presence of the nearby IR fixed point means that there is a range of couplings for which the running coupling evolves very slowly, i.e. it 'walks'. We are simulating the lattice version of this theory with staggered quarks at finite temperature studying the changes in couplings at the deconfinement and chiral-symmetry restoring transitions as the temporal extent (NtN_t) of the lattice, measured in lattice units, is increased. Our earlier results on lattices with Nt=4,6N_t=4,6 show both transitions move to weaker couplings as NtN_t increases consistent with walking behaviour. In this paper we extend these calculations to Nt=8N_t=8. Although both transition again move to weaker couplings the change in the coupling at the chiral transition from Nt=6N_t=6 to Nt=8N_t=8 is appreciably smaller than that from Nt=4N_t=4 to Nt=6N_t=6. This indicates that at Nt=4,6N_t=4,6 we are seeing strong coupling effects and that we will need results from Nt>8N_t > 8 to determine if the chiral-transition coupling approaches zero as Nt→∞N_t \rightarrow \infty, as needed for the theory to walk.Comment: 21 pages Latex(Revtex4) source with 4 postscript figures. v2: added 1 reference. V3: version accepted for publication, section 3 restructured and interpretation clarified. Section 4 future plans for zero temperature simulations clarifie

    Lifetime Difference and Endpoint effect in the Inclusive Bottom Hadron Decays

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    The lifetime differences of bottom hadrons are known to be properly explained within the framework of heavy quark effective field theory(HQEFT) of QCD via the inverse expansion of the dressed heavy quark mass. In general, the spectrum around the endpoint region is not well behaved due to the invalidity of 1/mQ1/m_Q expansion near the endpoint. The curve fitting method is adopted to treat the endpoint behavior. It turns out that the endpoint effects are truly small and the explanation on the lifetime differences in the HQEFT of QCD is then well justified. The inclusion of the endpoint effects makes the prediction on the lifetime differences and the extraction on the CKM matrix element ∣Vcb∣|V_{cb}| more reliable.Comment: 11 pages, Revtex, 10 figures, 6 tables, published versio

    Phase structure of SU(3) gauge theory with two flavors of symmetric-representation fermions

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    We have performed numerical simulations of SU(3) gauge theory coupled to Nf=2 flavors of symmetric representation fermions. The fermions are discretized with the tadpole-improved clover action. Our simulations are done on lattices of length L=6, 8, and 12. In all simulation volumes we observe a crossover from a strongly coupled confined phase to a weak coupling deconfined phase. Degeneracies in screening masses, plus the behavior of the pseudoscalar decay constant, indicate that the deconfined phase is also a phase in which chiral symmetry is restored. The movement of the confinement transition as the volume is changed is consistent with avoidance of the basin of attraction of an infrared fixed point of the massless theory.Comment: 12 pages, 11 figure

    Semi-Inclusive B\to K(K^*) X Decays with Initial Bound State Effects

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    The effects of initial bb quark bound state for the semi-inclusive decays B→K(K∗)XB\to K(K^*) X are studied using light cone expansion and heavy quark effective theory methods. We find that the initial bound state effects on the branching ratios and CP asymmetries are small. In the light cone expansion approach, the CP-averaged branching ratios are increased by about 2% with respect to the free bb-quark decay. For Bˉ0→K−(K∗−)X\bar B^0 \to K^- (K^{*-}) X, the CP-averaged branching ratios are sensitive to the phase γ\gamma and the CP asymmetry can be as large as 7% (14%), whereas for B−→Kˉ0(Kˉ∗0)XB^-\to \bar K^0 (\bar K^{*0})X the CP-averaged branching ratios are not sensitive to γ\gamma and the CP asymmetries are small (<1< 1%). The CP-averaged branching ratios are predicted to be in the ranges (0.53∼1.5)×10−4(0.53 \sim 1.5)\times 10^{-4} [(0.25∼2.0)×10−4(0.25 \sim 2.0)\times 10^{-4}] for Bˉ0→K−(K∗−)X\bar B^0 \to K^- (K^{*-})X and (0.77∼0.84)×10−4(0.77 \sim 0.84)\times 10^{-4} [(0.67∼0.74)×10−4(0.67 \sim 0.74)\times 10^{-4}] for B−→Kˉ0(Kˉ∗0)XB^-\to \bar K^0 (\bar K^{*0}) X, depending on the value of the CP violating phase γ\gamma. In the heavy quark effective theory approach, we find that the branching ratios are decreased by about 10% and the CP asymmetries are not affected. These predictions can be tested in the near future.Comment: 29 pages, 12 ps figure

    Attenuation of ischemic liver injury by prostaglandin E<inf>1</inf> analogue, misoprostol, and prostaglandin I<inf>2</inf> analogue, OP-41483

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    Background: Prostaglandin has been reported to have protective effects against liver injury. Use of this agent in clinical settings, however, is limited because of drugrelated side effects. This study investigated whether misoprostol, prostaglandin E1 analogue, and OP-41483, prostaglandin I2 analogue, which have fewer adverse effects with a longer half-life, attenuate ischemic liver damage. Study Design: Thirty beagle dogs underwent 2 hours of hepatic vascular exclusion using venovenous bypass. Misoprostol was administered intravenously for 30 minutes before ischemia and for 3 hours after reperfusion. OP-41483 was administered intraportally for 30 minutes before ischemia (2 μg/kg/min) and for 3 hours after reperfusion (0.5 μg/kg/min). Animals were divided into five groups: untreated control group (n = 10); high-dose misoprostol (total 100 μg/kg) group (MP-H, n = 5); middle-dose misoprostol (50 μg/kg) group (MP-M, n = 5); low-dose misoprostol (25 μg/kg) group (MP-L, n = 5); and OP-41483 group (OP, n = 5). Animal survival, hepatic tissue blood flow (HTBF), liver function, and histology were analyzed. Results: Two-week animal survival rates were 30% in control, 60% in MP-H, 100% in MP-M, 80% in MP-L, and 100% in OP. The treatments with prostaglandin analogues improved HTBF, and attenuated liver enzyme release, adenine nucleotrides degradation, and histologic abnormalities. In contrast to the MP-H animals that exhibited unstable cardiovascular systems, the MP- M, MP-L, and OP animals experienced only transient hypotension. Conclusions: These results indicate that misoprostol and OP-41483 prevent ischemic liver damage, although careful dose adjustment of misoprostol is required to obtain the best protection with minimal side effects
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