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    Improvement of shift quality of power shuttle transmission by controlling input speed

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    ν•™μœ„λ…Όλ¬Έ (석사)-- μ„œμšΈλŒ€ν•™κ΅ λŒ€ν•™μ› : λ°”μ΄μ˜€μ‹œμŠ€ν…œΒ·μ†Œμž¬ν•™λΆ€(λ°”μ΄μ˜€μ‹œμŠ€ν…œκ³΅ν•™), 2016. 8. κΉ€κ²½μš±.κ΅­λ¬Έ 초둝 전후진 νŒŒμ›Œμ‹œν”„νŠΈ λ³€μ†κΈ°λŠ” νŠΈλž™ν„° ꡬ동 μ‹œμ— 높은 μž‘μ—…λŠ₯λ₯ κ³Ό μ „λ™νš¨μœ¨μ„ 높일 수 μžˆμ–΄ 보급이 점차 λŠ˜μ–΄λ‚˜λŠ” 좔세이닀. κ·ΈλŸ¬λ‚˜ 이에 λΉ„ν•΄ 전후진 νŒŒμ›Œμ‹œν”„νŠΈ 변속기에 κ΄€ν•œ μ—°κ΅¬λŠ” λ―Έν‘ν•˜λ©°, 전후진 νŒŒμ›Œμ‹œν”„νŠΈ λ³€μ†κΈ°λ‘œ 인해 λ°œμƒν•˜λŠ” 변속좩격 완화에 κ΄€ν•œ 연ꡬ도 λ§Žμ§€ μ•Šλ‹€. λ”°λΌμ„œ λ³Έ μ—°κ΅¬λŠ” 전후진 νŒŒμ›Œμ‹œν”„νŠΈ 변속기에 μ˜ν•œ 변속좩격 및 λ³€μ†μ‹œκ°„ κ°œμ„ μ— κ΄€ν•œ 연ꡬλ₯Ό μˆ˜ν–‰ν•˜μ˜€λ‹€. λ³Έ μ—°κ΅¬μ˜ κ²°κ³Όλ₯Ό μš”μ•½ν•˜λ©΄ λ‹€μŒκ³Ό κ°™λ‹€. μ‹€μ°¨μ‹€ν—˜μ„ 톡해 νŠΈλž™ν„° 변속좩격을 μ˜ˆμΈ‘ν•  수 μžˆλŠ” 해석λͺ¨λΈμ„ κ°œλ°œν•˜μ˜€κ³ , 이λ₯Ό 톡해 μž…λ ₯속도λ₯Ό μ œμ–΄ν•˜μ—¬ 변속좩격 및 λ³€μ†μ‹œκ°„μ„ κ°μ†Œμ‹œμΌ°λ‹€. 해석λͺ¨λΈμ€ 엔진, 전후진 νŒŒμ›Œμ‹œν”„νŠΈ λ³€μ†κΈ°μ˜ 클러치 및 ν”ΌμŠ€ν†€, κΈ°μ–΄λ₯Ό ν¬ν•¨ν•œ μ „λ™λΌμΈμ˜ μš”μ†Œλ‘œ κ΅¬μ„±λ˜μ—ˆμœΌλ©° λŒ€μƒ νŠΈλž™ν„°μ˜ μ œμ›μ„ μ‚¬μš©ν•˜μ˜€λ‹€. 엔진 속도 및 였일 μ••λ ₯은 μ‹€μ°¨μ‹€ν—˜μ—μ„œ κ³„μΈ‘ν•œ 데이터λ₯Ό μž…λ ₯λ³€μˆ˜λ‘œ μ‚¬μš©ν•˜μ˜€λ‹€. μ™„μ„±λœ 해석λͺ¨λΈμ€ μ‹€μ°¨ μ‹€ν—˜ 결과와 λΉ„κ΅ν•˜μ—¬ μ°¨λŸ‰ μƒνƒœλ₯Ό λͺ¨λ°©ν•˜λŠ” 것을 κ²€μ¦ν•˜μ˜€λ‹€. 해석결과가 μ‹€μ°¨κ²°κ³Όλ₯Ό μ™„μ „νžˆ λͺ¨λ°©ν•˜μ§€ λͺ»ν•˜λŠ” μ΄μœ λŠ” 해석 ν”„λ‘œκ·Έλž¨μ΄ μ‹€μ œ ν™˜κ²½μ„ μ™„λ²½ν•˜κ²Œ κ΅¬ν˜„ν•˜μ§€ λͺ»ν•˜κΈ° λ•Œλ¬ΈμœΌλ‘œ νŒλ‹¨ν•˜μ˜€λ‹€. 변속성λŠ₯ μ§€ν‘œλ₯Ό κ°œλ°œν•˜μ—¬ μž…λ ₯속도 μ œμ–΄ μ „ν›„μ˜ 변속좩격과 λ³€μ†μ‹œκ°„μ„ λΉ„κ΅ν•˜μ˜€λ‹€. 변속좩격은 νŠΈλž™ν„° 진행방ν–₯의 가속도변화, λ³€μ†μ‹œκ°„μ€ μ˜€μΌμ••λ ₯ 인가 μ‹œμ λΆ€ν„° 엔진속도와 변속기 ν”Όλ™μΆ•μ˜ λ™κΈ°ν™”κΉŒμ§€μ˜ μ‹œμ μœΌλ‘œ μ •μ˜ν•˜μ˜€λ‹€. μž…λ ₯속도 μ œμ–΄λ₯Ό μœ„ν•΄ μŠ€λ‘œν‹€ μ—΄λ¦ΌλŸ‰ μ‹ ν˜Έλ₯Ό μž‘μ„±ν•˜μ˜€λ‹€. 였일 μ••λ ₯μ‹ ν˜Έκ°€ μΈκ°€λ˜μ—ˆμ„ λ•Œ μŠ€λ‘œν‹€ μ—΄λ¦ΌλŸ‰ μ‹ ν˜Έλ₯Ό 톡해 엔진속도λ₯Ό κ°μ†Œμ‹œμΌœ 변속 좩격 및 λ³€μ†μ‹œκ°„μ„ κ°œμ„ ν•˜μ˜€λ‹€. 변속좩격 및 λ³€μ†μ‹œκ°„μ€ λŒ€λΆ€λΆ„μ˜ κ²½μš°μ—μ„œ κ°œμ„ λ˜μ—ˆμœΌλ‚˜, 변속좩격이 κ°œμ„ λ˜λŠ” λΉ„μœ¨μ΄ λ³€μ†μ‹œκ°„λ³΄λ‹€ μ»Έλ‹€. λ˜ν•œ μ˜€μΌμ••λ ₯이 인가될 λ•Œ μž…λ ₯속도λ₯Ό μ΄ˆκΈ°μ†λ„μ˜ 50% μ΄ν•˜λ‘œ κΈ‰μž‘μŠ€λŸ½κ²Œ κ°μ†Œμ‹œν‚€λ©΄, 변속좩격 및 μ˜€λ²„μŠˆνŠΈκ°€ 생겨 μ œμ–΄ 전보닀 변속성λŠ₯이 λ–¨μ–΄μ‘Œλ‹€. μž…λ ₯μ†λ„μ œμ–΄κ°€ μ‹€μ œλ‘œ 변속성λŠ₯을 κ°œμ„ μ‹œν‚€λŠ”μ§€ ν™•μΈν•˜κΈ° μœ„ν•˜μ—¬ 전후진 νŒŒμ›Œμ‹œν”„νŠΈ 변속기가 μž₯착된 μ‹œν—˜ μž₯치λ₯Ό κ°œλ°œν•˜μ˜€λ‹€. μ‹œν—˜μž₯μΉ˜λŠ” 크게 μœ μ••λΆ€, μ‹œν—˜λΆ€, μ œμ–΄λΆ€λ‘œ κ΅¬μ„±ν•˜μ˜€λ‹€. μ‹œν—˜μž₯μΉ˜μ—μ„œ μ˜€μΌμ••λ ₯은 λΉ„λ‘€μ œμ–΄λ°ΈλΈŒλ‘œ, μž…λ ₯μ†λ„λŠ” μ„œλ³΄λͺ¨ν„°λ‘œ μ œμ–΄ν•˜μ˜€λ‹€. μ‹œν—˜μž₯치의 규λͺ¨μ— μ μ ˆν•œ μ˜€μΌμ••λ ₯을 μΈκ°€ν•˜κΈ° μœ„ν•΄ μ‹œν—˜ 쑰건에 따라 μ••λ ₯ν”„λ‘œνŒŒμΌμ„ μ‘°μ •ν•˜μ˜€μœΌλ©°, 적용된 μ••λ ₯ν”„λ‘œνŒŒμΌμ„ λ°”νƒ•μœΌλ‘œ μž…λ ₯속도λ₯Ό μ œμ–΄ν•˜μ˜€λ‹€. 100 rpm의 μΌμ •ν•œ κ°„κ²©μœΌλ‘œ μž…λ ₯속도 κ°μ†Œμ‹œμΌœ μž…λ ₯속도에 λ”°λ₯Έ 변속성λŠ₯의 κ²½ν–₯μ„± 및 졜적 속도 κ°μ†Œ(drop) μˆ˜μ€€μ„ 규λͺ…ν•˜μ˜€λ‹€. μ£Όμš”μ–΄ : 전후진 νŒŒμ›Œμ‹œν”„νŠΈ 변속기, 변속성λŠ₯, μž…λ ₯μ†λ„μ œμ–΄, 해석λͺ¨λΈ ν•™ 번 : 2014-20058제1μž₯ μ„œλ‘  1 제1절 μ—°κ΅¬μ˜ λ°°κ²½ 1 제2절 μ—°κ΅¬μ˜ λͺ©μ  3 제3절 연ꡬ사 4 제2μž₯ νŠΈλž™ν„° 해석 λͺ¨λΈ 개발 5 제1절 κ°œμš” 및 λŒ€μƒ νŠΈλž™ν„° 5 제2절 변속성λŠ₯ 해석λͺ¨λΈ 6 2.2.1 μŠ΅μ‹λ‹€νŒν΄λŸ¬μΉ˜ μ‹œμŠ€ν…œ 및 λͺ¨λΈ κ°œμš” 6 2.2.2 엔진 λͺ¨λΈ 8 2.2.3 클러치 및 ν”ΌμŠ€ν†€ λͺ¨λΈ 11 2.2.4 전동라인 및 λŒν•‘κ³Ό κ°•μ„± λͺ¨λΈ 15 2.2.5 데이터 블둝 λͺ¨λΈ 20 제3μž₯ 해석λͺ¨λΈ μž…λ ₯μ†λ„μ œμ–΄ 21 제1절 μ‹€μ°¨μ‹€ν—˜ 21 제2절 해석λͺ¨λΈ 검증 27 제3절 μž…λ ₯속도 μ œμ–΄ μ˜ˆλΉ„μ‹œν—˜ 33 제4절 μž…λ ₯속도 μ œμ–΄μ— λ”°λ₯Έ 변속성λŠ₯ 47 제4μž₯ μ‹œν—˜μž₯λΉ„ 개발 50 제1절 κ°œμš” 50 제2절 μ‹œν—˜μž₯λΉ„ κ³„μΈ‘μ‹œμŠ€ν…œ ꡬ성 50 제3절 μ‹œν—˜μž₯λΉ„ 개발 53 제5μž₯ μž…λ ₯속도 μ œμ–΄λ₯Ό ν†΅ν•œ 변속성λŠ₯ κ°œμ„  59 제1절 μœ μ••ν”„λ‘œνŒŒμΌ 59 제2절 μž…λ ₯속도 μ œμ–΄ 방법 62 제3절 κ²°κ³Ό 및 뢄석 64 제6μž₯ μš”μ•½ 및 κ²°λ‘  75 μ°Έκ³  λ¬Έν—Œ 77 뢀둝 A. νŠΈλž™ν„° μ‹€μ°¨ μ‹€ν—˜ 데이터 79 Abstract 103Maste

    Serum Ig E Level in Koreal Sound Controls and Bronchial Asthmatics

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    Total serum IgE level were determined in 83 Korean sound controls (male 38, female 45) and 250 bronchial asthmatics (male 136, female 114) who had not been treated with hyposensitization. The patients were divided into two small groups, i. e. group (a) with asthma only and group (b) with other coexistent atopic diseases. IgE in serum was measured by the radioimmunosorbent technique using the kits of Phadebas~ and following results were obtained. 1. In both control and asthma group, the frequency distributions of the serum IgE levels were continuous ut not normal. 2. In control group, the serum IgE levels ranged from 8 to 475 I.U./ml and the mean and the standard deviation was 151Β±1l4 I.U./ml (male 151Β±124, female 152Β±105). There were no differences between sexes or age groups 3. In patients group, the serum levels ranged from 33 to 7,200 I.U./ml and the mean and S.D. were 1401 Β±1412I.U./ml (male 1682Β±1420, female 1073Β±1217). The levels of male patients were higher than those of females. but the reasons were uncertain. 4. No significant differences were found between the serum IgE levels in group(a) and those in group (b). 5. The serum IgE level higher than 400I. U./ml was thought to be significantly raised and such levels were found in 4.8% of sound controls and in 74% of total asthma patients. In the laller, the allergic bases may play major roles in the pathogenesis of the disease G. Significantly raised serum IgE levels were found in 93% of the asthma patients under 10 years of age, indicating the high prevalence of allergic asthma in childhood

    The expression of matrix metalloproteinases (MMPs), tissue inhibitor of metalloproteinases (TIMPs) and angiogenesis in relation to the depth of tumor invasion and lymph node metastasis in submucosally invasive colorectal carcinoma

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    BACKGROUND/AIMS: Lymph node (LN) metastasis occurs in approximately 10% of patients with submucosally invasive colorectal carcinoma. This study was performed to determine the role of matrix metalloproteinases (MMPs), tissue inhibitors of metalloproteinases (TIMPs) production and microvessel formation on the LN metastasis in submucosally invasive colorectal carcinoma. METHODS: A total of forty-one subjects with surgically resected submucosally invasive colorectal carcinoma were included in this study. Immunohistochemical staining of MMP-2, MMP-9, TIMP-1, TIMP-2, and urokinase-type plasminogen activator were performed. Angiogenesis was evaluated by counting the number of microvessels in each pathologic specimen as identified by CD34 immunohistochemical staining. RESULTS: The depth of submucosal invasion was not significantly correlated with the expression of MMP-2, MMP-9, TIMP-1, TIMP-2, or urokinase-type plasminogen activator, but the microvessel count was significantly correlated with the absolute depth of invasion (r=0.312, p<0.05). Upregulation of TIMP-2 was positively correlated with adjacent lymphatic invasion (p<0.05) and increased TIMP-2 expression was correlated with LN metastasis in submucosally invasive colorectal carcinoma (p=0.088). CONCLUSIONS: These results suggest that the expression of TIMP-2 and the microvessel count may be useful parameters for considering additional surgery after endoscopic treatment of submucosally invasive colorectal carcinoma.
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