10 research outputs found
Exploring Social Environmental Identity of Students Participated in a Social Action-Oriented Climate Change SSI Club
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Όλ¬Έ (μμ¬) -- μμΈλνκ΅ λνμ : μ¬λ²λν κ³Όνκ΅μ‘κ³Ό(μ§κ΅¬κ³Όνμ 곡), 2021. 2. κΉμ°¬μ’
.As climate change threatens the survival of the Earth, many people have argued the need for climate change education. Compared to other countries, students in Korea have a high awareness of climate change. However, they do not take action for climate change mitigation and adaptation. Many researchers have begun to find that promoting environmentally sustainable behavior requires a strong community identity. Clayton (2003) claimed that the environmental identity can describe how abstract global issues become immediate and personal for an individual. And, the validity and utility of environmental identity have been proven by some evidence that it affects our thinking or behavior, and does so better than other determinants, such as attitudes.
Moreover, Kempton and Holland (2003) suggest social environmental identity that is strongly influenced by social factors. Social Environmental society is self-definition with respect to ones reference group, the environmental movement, the government, the marketplace, and lifestyle choice. And, they found three stages of social environmental identity development.
Because environmental identity inevitably contains a social component, I adapted social environmental identity as the main concept of this research. I explored the social environmental identity of students participating in a social action-oriented climate change SSI club program. For this purpose, a social action-oriented climate change education program was developed and implemented at a secondary school in the Seoul Metropolitan Area. Twelve high school students participated in individual pre-post interviews based on Kempton and Hollands (2003) social environmental identity theory.
Analysis of student responses to interview questions revealed 12 characteristics that make Kempton and Hollands theory concrete. Students that do not have social environmental identity show indifference to climate, no influence on me, the lack of knowledge related to climate change. And, the social environmental identity 'stage 1' also indicates three characteristics: interest in climate change, awareness of climate change seriousness, attention to my actions. Next, the social environmental identity 'stage 2 represents four characteristics: 'for me (the purpose of environmental action), redefinition of self, human, the cause of climate change, correspondence between identity and behaviors. The social environmental identity 'stage 3, lastly, shows 2 characteristics: Knowing how to do properly, 'Expanding the scope of practice'.
Secondly, students' social environmental identity has drawn environmental values, attitudes toward the environment for mitigating climate change. Moreover, social environmental identity can draw ones passion and a sense of agency in taking social action. Three students that show a high level of social environmental identity are confident of their social action ideas. They applied their ideas to group plans for social action, and developed ideas in detail. Lastly, students social environmental identities have changed during participation in the program. The main factor of the change in social environmental identity is social action. During the program, they found how to introduce the seriousness of climate change and make people take actions for mitigating climate change. Planning and implementing social action, they learned the impact of climate change, and gained a sense of agency. In other words. they realized the need for action and recognized themselves as actors.
From the findings of the research, we implicate the effort for developing and applying for future action-oriented climate change education programs. In climate change education, it is necessary for providing the opportunity that students plan and implicate social action by themselves. I hope that the movement toward social action can make students future citizens for solving climate change.κ³Όν κ΄λ ¨ μ¬νμ μμ (SSI) μ€ νλμΈ κΈ°νλ³νμ κ΄ν κ΄μ¬μ΄ μ¦κ°νλ©΄μ μ΄μ κ΄λ ¨ν νκ΅ λ΄μΈμ κ΅μ‘μ΄ νμ±νλκ³ μλ€. μ°λ¦¬λλΌ νμλ€μ κΈ°νλ³νμ λν΄ λμ μΈμμ 보μ¬μ£Όλ κ²κ³Ό λ°λλ‘, ν΄κ²°μ μν μ€μ μ μΈ μ€μ²μλ μκ·Ήμ μ΄κ³ μμ§κ° λ€μ λΆμ‘±νλ€. μ΄λ¬ν μ€μ²μ λΆμ‘±μ 극볡νκ³ , κΈ°νλ³ν κ΄λ ¨ νλμ μ΄λμ΄λ΄κΈ° μν μμΈμ νμν κΈ°μ‘΄μ μ°κ΅¬λ λ§λ€. μ΄λ¬ν μμΈμλ μμ§, νλ, κ°μ , κ°μΉκ΄ λ±μ΄ ν¬ν¨λλ€. νμ§λ§ νλκ³Ό μ€μ²μ 볡μ‘ν ꡬ쑰λ₯Ό μ΄ν΄νκΈ° μν΄μλ κΈ°μ‘΄μ κ°λ³μ μΌλ‘ μ°κ΅¬λμλ κΈ°μ‘΄μ μμΈλ€μ ν΅ν©ν μ μλ λΆμ λꡬλ₯Ό νμλ‘ νλ€.
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Abstract 169Maste
κ°μ‘±μ±μΌμΆμ 리체λ§λ§λ³μ¦μμ FZD4, LRP5, TSPAN12μ λΆμμ μ νμ μ°κ΅¬
νμλ
Όλ¬Έ (μμ¬)-- μμΈλνκ΅ λνμ : μνκ³Ό, 2014. 2. λ°μ±μ.Introduction: Familial exudative vitreoretinopathy (FEVR) is a rare hereditary disorder characterized by failure of peripheral retinal vascularization. Clinical manifestation of the disease can be variable, ranging from nonsymptomatic vascular anomalies in the peripheral retina to bilateral retinal detachments with blindness. FEVR is genetically heterogeneous, and it is found in various modes of inheritance. Autosomal dominant inheritance is the most common form in FEVR and FZD4, LRP5, and TSPAN12 are known to be associated with the disease. These genes are essential components of wingless (Wnt) pathway and pathogenic mutations affecting the function of these genes result in abnormal retinal vascular formation. In the Korean population, there had not been any report on the molecular identification of FEVR. We performed mutation screening for FZD4, LRP5 and TSPAN12 in patients with clinical diagnosis of FEVR. Identification of the molecular spectrum in Korean FEVR patients will be helpful in establishing an effective testing strategy, as well as providing informative genetic counseling for the family members of the proband.
Methods: Mutational studies were carried out in 42 unrelated patients with diagnosis of FEVR during 2008 to 2012 at Seoul National University Hospital. Diagnosis of FEVR was established by ophthalmic examinations. All patients had been previously screened for NDP mutation, which were found to be negative. Sequencing analyses for three causal genes (FZD4, LRP5, and TSPAN12) were performed. Gross deletions and duplications were screened via Multiplex Ligation-dependent Probe Amplification.
Results: Two previously reported mutations (c.313A>G, c.1282_1285delGACA), four novel pathogenic mutations (c.160C>T, c.539_540delAG, c.653_676dup24, c.1210_1211delTT) and a whole gene deletion of FZD4 were found in 11 patients. Three novel missense variants (c.456C>G, c.470T>C, c.676T>A) were also found. A previously reported mutation of c.3361A>G in LRP5 was detected in three patients, and two novel missense variants (c.731C>G, c.4098C>G) were detected in two patients. In case of TSAPN12, a previously reported mutation, c.212_218delGCTGTTT was found in one patient, and three more novel missense variants (c.56T>G, c.194C>T, c.484G>A) were found in another three patients.
Conclusions: In this study, we have identified four known mutations, four novel pathogenic mutations and a whole gene deletion in 15 patients. In cases of novel missense variants detected in nine patients, five of them were considered as pathogenic according to the predictions by in-silico analyses and allele frequencies in normal control group. Among 20 patients with pathogenic mutation detected, 13 were due to FZD4 mutations, showing the largest proportion of this gene in attribution to the autosomal dominant FEVR (13/42 patients, 31.0%). By applying this result, testing strategy of FEVR starting with screening for FZD4 mutations can be applied in the clinical fields. Genetic counseling of asymptomatic family members as well as proband will be helpful in further management and prevention of the disease progression.Introduction 1
Materials and Methods 5
Results 13
Discussion 23
References 26
Abstract in Korean 29Maste
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