1,344,230 research outputs found

    Searching for demography's missing link: Momentum

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    This paper is both more theoretical and highly applied; both methodological and policy-oriented. Empirically until recently there has been little work on momentum effects. By way of case-studies on momentum effects, the paper reviews published policy-oriented work on New Zealand on national growth and age-structural transitions; on fertility patterns in New Zealand; and on survivorship and longevity, and the more local discussion of Maori-Pakeha differentials. The conclusion is that more attention must be paid to momentum. It calls for the demographic community to lead in moving policy analyses away from demography as a naive art form to one that is more multi-dimensional and sophisticated

    Magnetars and pulsars: a missing link

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    There is growing evidence that soft gamma-ray repeaters (SGRs) and anomalous X-ray pulsars (AXPs) are isolated neutron stars with superstrong magnetic fields, i.e., magnetars, marking them a distinguished species from the conventional species of spindown-powered isolated neutron stars, i.e., radio pulsars. The current arguments in favor of the magnetar interpretation of SGR/AXP phenomenology will be outlined, and the two energy sources in magnetars, i.e. a magnetic dissipation energy and a spindown energy, will be reviewed. I will then discuss a missing link between magnetars and pulsars, i.e., lack of the observational evidence of the spindown-powered behaviors in known magnetars. Some recent theoretical efforts in studying such behaviors will be reviewed along with some predictions testable in the near future.Comment: Invited talk at the Sixth Pacific Rim Conference on Stellar Astrophysics, a tribute to Helmut A. Abt, July 11-17, 2002, Xi'an. To appear in the proceedings (eds. K. S. Cheng, K. C. Leung & T. P. Li

    The Missing Link: Magnetism and Superconductivity

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    The effect of magnetic moments on superconductivity has long been a controversial subject in condensed matter physics. While Matthias and collaborators experimentally demonstrated the destruction of superconductivity in La by the addition of magnetic moments (Gd), it has since been suggested that magnetic fluctuations are in fact responsible for the development of superconducting order in other systems. Currently this debate is focused on several families of unconventional superconductors including high-Tc cuprates, borocarbides as well as heavy fermion systems where magnetism and superconductivity are known to coexist. Here we report a novel aspect of competition and coexistence of these two competing orders in an interesting class of heavy fermion compounds, namely the 1-1-5 series: CeTIn5 where T=Co, Ir, or Rh. Our optical experiments indicate the existence of regions in momentum space where local moments remain unscreened. The extent of these regions in momentum space appears to control both the normal and superconducting state properties in the 1-1-5 family of heavy fermion (HF) superconductors.Comment: 6 pages, 2 figure

    Ultraspinning instability: the missing link

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    We study linearized perturbations of Myers-Perry black holes in d=7, with two of the three angular momenta set to be equal, and show that instabilities always appear before extremality. Analogous results are expected for all higher odd d. We determine numerically the stationary perturbations that mark the onset of instability for the modes that preserve the isometries of the background. The onset is continuously connected between the previously studied sectors of solutions with a single angular momentum and solutions with all angular momenta equal. This shows that the near-extremality instabilities are of the same nature as the ultraspinning instability of d>5 singly-spinning solutions, for which the angular momentum is unbounded. Our results raise the question of whether there are any extremal Myers-Perry black holes which are stable in d>5.Comment: 19 pages. 1 figur

    Civic Capital as the Missing Link

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    This chapter reviews the recent debate about the role of social capital in economics. We argue that all the difficulties this concept has encountered in economics are due to a vague and excessively broad definition. For this reason, we restrict social capital to the set of values and beliefs that help cooperation—which for clarity we label civic capital. We argue that this definition differentiates social capital from human capital and satisfies the properties of the standard notion of capital. We then argue that civic capital can explain why differences in economic performance persist over centuries and discuss how the effect of civic capital can be distinguished empirically from other variables that affect economic performance and its persistence, including institutions and geography.

    Cognition: a missing link in mainstream CDA?

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    TRPV4-A Missing Link Between Mechanosensation and Immunity

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    Transient receptor potential vanilloid-type 4 (TRPV4) cation channel is widely expressed in all tissues as well as in immune cells and its function as mechanosensitive Ca2+ channel seems to be conserved throughout all mammalian species. Of late, emerging evidence has implicated TRPV4 in the activation and differentiation of innate immune cells, especially in neutrophils, monocytes, and macrophages. As such, TRPV4 has been shown to mediate neutrophil adhesion and chemotaxis, as well as production of reactive oxygen species in response to pro-inflammatory stimuli. In macrophages, TRPV4 mediates formation of both reactive oxygen and nitrogen species, and regulates phagocytosis, thus facilitating bacterial clearance and resolution of infection. Importantly, TRPV4 may present a missing link between mechanical forces and immune responses. This connection has been exemplary highlighted by the demonstrated role of TRPV4 in macrophage activation and subsequent induction of lung injury following mechanical overventilation. Mechanosensation via TRPV4 is also expected to activate innate immune cells and establish a pro-inflammatory loop in fibrotic diseases with increased deposition of extracellular matrix (ECM) and substrate stiffness. Likewise, TRPV4 may be activated by cell migration through the endothelium or the extracellular matrix, or even by circulating immune cells squeezing through the narrow passages of the pulmonary or systemic capillary bed, a process that has recently been linked to neutrophil priming and depriming. Here, we provide an overview over the emerging role of TRPV4 in innate immune responses and highlight two distinct modes for the activation of TRPV4 by either mechanical forces (“mechanoTRPV4”) or by pathogens (“immunoTRPV4”)
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