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Electrophysiological Mechanisms of Atrial Flutter

By Ching- Tai Tai and Shin-Ann Chen

Abstract

Atrial flutter (AFL) is a common arrhythmia in clinical practice. Several experimental models such as tricuspid regurgitation model, tricuspid ring model, sterile pericarditis model and atrial crush injury model have provided important information about reentrant circuit and can test the effect of antiarrhythmic drugs. Human atrial flutter has typical and atypical forms. Typical atrial flutter rotates around tricuspid annulus and uses the crista terminalis and sometimes sinus venosa as the boundary. The IVC-tricuspid isthmus is a slow conduction zone and the target of radiofrequency ablation. Atypical atrial flutter may arise from the right or left atrium. Right atrial flutter includes upper loop reentry, free wall reentry and figure of eight reentry. Left atrial flutter includes mitral annular atrial flutter, pulmonary vein-related atrial flutter and left septal atrial flutter. Radiofrequency ablation of the isthmus between the boundaries can eliminate these arrhythmias

Topics: Indian Pacing and Electrophysiology Journal
Publisher: Indian Pacing and Electrophysiology Group
Year: 2006
OAI identifier: oai:cogprints.org:4804
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    Citations

    1. A new model of atrial flutter.
    2. (1992). Activation patterns in experimental canine atrial flutter produced by right atrial crush-injury.
    3. Atrial reentry around an anatomic barrier with a partially refractory excitable gap: A canine model of atrial flutter.
    4. (1910). Auricular flutter and fibrillation.
    5. Characterization of low right atrial isthmus as the slow conduction zone and pharmacological target in typical atrial flutter. Circulation 1997;96:2601-
    6. (1997). Conduction velocity in the tricuspid valve-inferior vena cava isthmus is slower in patients with type I atrial flutter compared to those without a history of atrial flutter.
    7. (1990). Demonstration of an area of slow conduction in human atrial flutter.
    8. Iliesc TT. A demonstration of circus movement in clinical flutter of the auricles. Heart 1921;8:341.
    9. Influence of the passive anisotropic properties on directional differences in propagation following modification of the sodium conductance in human atrial muscle: A model of reentry based on anisotropic discontinuous propagation.
    10. Natural and evoked atrial flutter due to circus movement in dogs.
    11. (1986). Relating extracellular potentials and their derivatives to anisotropic propagation at a microscopic level in human cardiac muscle: Evidence for electrical uncoupling of side-to-side fiber connections with increasing age. Circ Res
    12. Studies on auricular tachycardia caused by aconitine administration.
    13. Studies on flutter and fibrillation. II:The influence of artificial obstacles on experimental auricular flutter.
    14. (1998). The architecture of the atrial musculature between the orifice of the inferior cava vein and the tricuspid valve: The anatomy of the isthmus.
    15. (1981). The effects on atrial electrophysiology and structure of surgically induced right atrial enlargement in dogs. Circ Res

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