(2002). Activation of astrocyte intracellular signaling pathways by interleukin-1 in rat primary striatal cultures. Glia
(1998). Ananko EA, Podkolodnaya OA, Kolpakov FA, et al.: Databases on transcriptional regulation: TRANSFAC, TRRD and COMPEL. Nucleic Acids Res
(2005). AS: Positive and negative regulation of EAAT2 by NF-kappaB: a role for N-myc in TNFalpha-controlled repression. Embo J
(2004). Ashkenazi A: Tumor necrosis factor: an apoptosis JuNKie? Cell
(1997). Breedveld FC: Analysis of the synovial cell infiltrate in early rheumatoid synovial tissue in relation to local disease activity. Arthritis Rheum
(2001). Camandola S: NF-kappaB in neuronal plasticity and neurodegenerative disorders.
(2002). Catechol-O-Methyltransferase (COMT)-mediated methylation metabolism of endogenous bioactive catechols and modulation by endobiotics and xenobiotics: importance in pathophysiology and pathogenesis. Curr Drug Metab
(2006). Catechol-O-methyltransferase gene polymorphisms are associated with multiple pain-evoking stimuli. Pain
(2007). Catechol-O-methyltransferase inhibition increases pain sensitivity through activation of both beta2- and beta3-adrenergic receptors. Pain
(2004). Cell type-specific interleukin-1beta signaling in the CNS.
(1999). Characterization and implications of estrogenic down-regulation of human catechol-O-methyltransferase gene transcription. Mol Pharmacol
(2004). Cho HJ: Spinal NF-kB activation induces COX-2 upregulation and contributes to inflammatory pain hypersensitivity.
(2004). Chung LW: Identification of a negative regulatory cis-element in the enhancer core region of the prostate-specific antigen promoter: implications for intersection of androgen receptor and nuclear factor-kappaB signalling in prostate cancer cells.
(1979). Creveling CR: Immunohistochemical demonstration of catechol-o-methyltransferase in mammalian brain. Brain Res
(2003). Dahiya R: Multiple promoters of catechol-O-methyltransferase gene are selectively inactivated by CpG hypermethylation in endometrial cancer. Cancer Res
(2003). DR: Catechol O-methyltransferase mRNA expression in human and rat brain: evidence for a role in cortical neuronal function. Neuroscience
(2006). DR: Catechol-o-methyltransferase, cognition, and psychosis: Val158Met and beyond. Biol Psychiatry
(1976). Erythrocyte catechol-O-methyltransferase activity in facial pain patients. J Dent Res
(2006). et al.: Hypomethylation of MB-COMT promoter is a major risk factor for schizophrenia and bipolar disorder. Hum Mol Genet