Skip to main content
Article thumbnail
Location of Repository

Particles induce apical plasma membrane enlargement in epithelial lung cell line depending on particle surface area dose

By Christina Brandenberger, Barbara Rothen-Rutishauser, Fabian Blank, Peter Gehr and Christian Mühlfeld
Topics: Research
Publisher: BioMed Central
OAI identifier: oai:pubmedcentral.nih.gov:2661036
Provided by: PubMed Central
Download PDF:
Sorry, we are unable to provide the full text but you may find it at the following location(s):
  • http://www.pubmedcentral.nih.g... (external link)
  • Suggested articles

    Citations

    1. (2008). Interactions of nanoparticles with pulmonary structures and cellular responses.
    2. (2006). Ultrafine particlelung interactions: does size matter?
    3. (2006). Rothen-Rutishauser BM: Fate of inhaled particles after interaction with the lung surface. Paediatr Respir Rev
    4. (2005). Combustion-derived nanoparticles: a review of their toxicology following inhalation exposure. Part Fibre Toxicol
    5. (2004). III: Air pollution and health – good news and bad.
    6. (2005). A: Cardiovascular effects of fine and ultrafine particles.
    7. (2002). Pope CA III: Cardiopulmonary mortality and air pollution. Lancet
    8. (2003). Nel A: Ultrafine particulate pollutants induce oxidative stress and mitochondrial damage. Environ Health Perspect
    9. (2005). Gonzalez-Flecha B: PM-induced cardiac oxidative stress and dysfunction are mediated by autonomic stimulation. Biochim Biophys Acta
    10. (2008). AL: Combustion-derived hydrocarbons localize to lipid droplets in respiratory cells.
    11. (2005). Nanotoxicology: An emerging discipline evolving from studies of ultrafine particles. Environ Health Perspect
    12. (1990). F: Surfactant displaces particles toward the epithelium in airways and alveoli. Respir Physiol
    13. (1990). Particle retention in airways by surfactant.
    14. (2005). Hohlfeld JM: Surfactant protein D increases phagocytosis and aggregation of pollen-allergen starch granules. Am J Physiol Lung Cell Mol Physiol
    15. (2004). TA: Urban PM2.5 surface chemistry and interactions with bronchoalveolar lavage fluid. Inhal Toxicol
    16. (2008). Kreyling W: The role of macrophages in the clearance of inhaled ultrafine titanium dioxide particles.
    17. (2002). Morphological aspects of particle uptake by lung phagocytes. Microsc Res Tech
    18. (2008). Polystyrene nanoparticle trafficking across alveolar epithelium. Nanomedicine
    19. (2008). Translocation and cellular entering mechanisms of nanoparticles in the respiratory tract. Swiss Med Wkly
    20. (2007). Translocation of particles and inflammatory responses after exposure to fine particles and nanoparticles in an epithelial airway model. Part Fibre Toxicol
    21. (2007). Schins RPF: Cellular response to nanoparticles: Target structures and mechanisms. Nanotoxicology
    22. (2006). Cellular uptake of magnetic nanoparticle is mediated through energy-dependent endocytosis in A549 cells.
    23. (2001). Endocytosis of ultrafine particles by A549 cells.
    24. (2001). Hubmayr RD: Deformation-induced lipid trafficking in alveolar epithelial cells.
    25. (2002). Hubmayr RD: Role of deformation-induced lipid trafficking in the prevention of plasma membrane stress failure. Am J Respir Crit Care Med
    26. (2006). G: Exosomes: a common pathway for a specialized function.
    27. (2008). Exosome function: From tumor immunology to pathogen biology. Traffic
    28. (2002). Regulated exocytosis: a novel, widely expressed system. Nat Cell Biol
    29. (2006). An optimized in vitro model of the respiratory tract wall to study particle cell interactions.
    30. (2005). Ultrafine particles cross cellular membranes by nonphagocytic mechanisms in lungs and in cultured cells. Environ Health Perspect
    31. (2007). WCW: Elucidating the mechanism of cellular uptake and removal of protein-coated gold nanoparticles of different sizes and shapes. Nano Letters
    32. (1976). Continuous tumor-cell line from a human lung carcinoma with properties of type-II alveolar epithelial cells.
    33. (2007). Visualization and quantitative analysis of nanoparticles in the respiratory tract by transmission electron microscopy. Part Fibre Toxicol
    34. (1986). Cruz-Orive LM: Estimation of surface-area from vertical sections.
    35. (2008). Methylprednisolone fails to preserve pulmonary surfactant and blood-air barrier integrity in a porcine cardiopulmonary bypass model.
    36. (1984). The unbiased estimation of number and sizes of arbitrary particles using the dissector.
    37. (1993). HJG: The rotator.
    38. (2008). Taking tissue samples from the placenta: An illustration of principles and strategies. Placenta
    39. (2007). How much is there really? Why stereology is essential in lung morphometry.
    40. (2005). Nothturfft A: Transcriptional regulation of phagocytosis-induced membrane biogenesis by sterol regulatory element binding protein. PNAS
    41. (2008). P: In vitro models of the human epithelial airway barrier to study the toxic potential of particulate matter. Expert Opin Drug Metab Toxicol
    42. (2007). Cell selective glucocorticoid induction of caveolin-1 and caveolae in differentiating pulmonary alveolar epithelial cell cultures. Biochem Biophys Res Commun
    43. (2001). TS: Huntingtin interacting protein 1 is a clathrin coat binding protein required for differentiation of late spermatogenic progenitors. Mol Cell Biol
    44. (2005). Stark WJ: Oxide nanoparticle uptake in human lung fibroblasts: effects of particle size, agglomeration, and diffusion at low concentrations. Environ Sci Technol
    45. (2001). Homann U: Cell surface area regulation and membrane tension.
    46. (1998). Membrane capacitance changes associated with particle uptake during phagocytosis in macrophages.
    47. (2002). How to eat something bigger than your head. Cell
    48. (2007). Inflammatory response to TiO2 and Carbonaceous particles scales best with BET surface area. Environ Health Perspect
    49. (2006). H: Instillation of six different ultrafine carbon particles indicates a surface area threshold dose for acute lung inflammation in mice. Environ Health Perspect

    To submit an update or takedown request for this paper, please submit an Update/Correction/Removal Request.