Abstract

In conclusion, the number and variety of ETV6 translocations not resulting in a fusion protein, together with loss of ETV6 by deletion and heterozygous or homozygous mutations, makes a mutually supportive and compelling case for loss and haploinsufficiency of ETV6 as a leukemogenic step in AML-M0

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Archivio istituzionale della ricerca - Università di Bari

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Last time updated on 12/11/2016

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