The loss of PPARγ in T cells and <i>Clostridium difficile</i> infection enhances Th17 responses in spleen and lamina propria of mice.

Abstract

<p>Splenocytes and lamina propria lymphocytes from wild type and T cell PPARγnull mice infected with <i>C. difficile</i> (n = 6) were immunophenotyped to identify immune cell subsets by flow cytometry. Data are represented as mean ± standard error. Points with an asterisk are significantly different when compared to the control group (<i>P</i><0.05).</p