<p>The interaction of <i>Lactobacillus acidophilus</i> and time on MLC<sub>20</sub> phosphorylation was not significant. (A and B) The levels of phospho-MLC<sub>20</sub> in intestinal smooth muscle were significantly decreased after TBI, *<i>P</i> < 0.05 compared with control. Treating with <i>Lactobacillus acidophilus</i> attenuated TBI-mediated inhibition of MLC<sub>20</sub> phosphorylation, #<i>P</i> < 0.05 compared with TBI. (C) The marked decreases in immunoreactivity for phospho-MLC<sub>20</sub> were observed in intestinal smooth muscle after TBI, and treatment with <i>Lactobacillus acidophilus</i> significantly attenuated TBI-mediated inhibition of MLC<sub>20</sub> phosphorylation.</p
