<p>The 29 virulence-associated genes analysed were: <i>afa/draBC</i>, Dr-binding adhesins; <i>fimH</i>, mannose-specific adhesin of type 1 fimbriae; <i>papA</i>, P fimbriae structural subunit; <i>papE</i>, fimbriae tip pilins; <i>papC</i>, p fimbriae assembly; <i>papG</i>, p fimbriae adhesin (and alleles I, II, and III); <i>sfa/focDE</i>, S and F1C fimbriae; <i>sfaS</i>, S fimbriae; <i>focG</i>, <i>focA</i>, F1C fimbriae; <i>bmaE</i>, blood group M fimbriae; <i>hlyD</i>, <i>hlyA</i>, α-haemolysin; <i>cnf1</i>, cytotoxic necrotizing factor type 1; <i>kpsM</i> II, group 2 capsule in addition to specifically targeting K1 and K5 genes; <i>rfc</i>, O antigen polymerase; <i>fyuA</i>, ferric yersiniabactin receptor; <i>iutA</i>, aerobactin receptor; <i>iroN</i>, almochelin receptor; <i>ireA</i>, iron-responsive element gene; <i>ibeA</i>, invasion of brain endothelium; <i>traT</i>, serum-resistance associated; PAI, pathogenicity island; <i>cvaC</i>, Colicin-V.</p><p>The prevalence of virulence-associated genes among 74 uropathogenic <i>E</i>. <i>coli</i> isolates from cats.</p
