<i>amn</i> mutants show defects in egg-laying decisions.

Abstract

<p>(A) In sucrose/caffeine chamber, <i>amn</i>^<i>1</i>, <i>amn</i>^<i>28A</i>, <i>amn</i>^<i>c651</i>, and <i>amn</i>^<i>X8</i> flies showed significant difference in egg-laying decisions compared to wild-type flies. <i>amn</i>^<i>X8</i> showed significant difference compared to the other <i>amn</i> mutants. Each value represents mean ± SEM (n = 34–35, ***P < 0.001). (B) In sucrose/plain chamber, <i>amn</i>^<i>1</i>, <i>amn</i>^<i>28A</i>, <i>amn</i>^<i>c651</i>, and <i>amn</i>^<i>X8</i> flies showed significant difference in egg-laying decisions compared to wild-type flies. <i>amn</i>^<i>X8</i> showed significant difference compared to the other <i>amn</i> mutants. Each value represents mean ± SEM (n = 20, ***P < 0.001, **P < 0.01). (C) In caffeine/plain chamber, <i>amn</i>^<i>1</i>, <i>amn</i>^<i>28A</i>, <i>amn</i>^<i>c651</i>, and <i>amn</i>^<i>X8</i> flies showed significant difference in egg-laying decisions compared to wild-type flies. Each value represents mean ± SEM (n = 23–27, ***P < 0.001).</p