musrfit: A free platform-independent framework for muSR data analysis

A free data-analysis framework for muSR has been developed. musrfit is fully written in C++, is running under GNU/Linux, Mac OS X, as well as Microsoft Windows, and is distributed under the terms of the GNU GPL. It is based on the CERN ROOT framework and is utilizing the Minuit optimization routines for fitting. It consists of a set of programs allowing the user to analyze and visualize the data. The fitting process is controlled by an ascii-input file with an extended syntax. A dedicated text editor is helping the user to create and handle these files in an efficient way, execute the fitting, show the data, get online help, and so on. A versatile tool for the generation of new input files and the extraction of fit parameters is provided as well. musrfit facilitates a plugin mechanism allowing to invoke user-defined functions. Hence, the functionality of the framework can be extended with a minimal amount of overhead for the user. Currently, musrfit can read the following facility raw-data files: PSI-BIN, MDU (PSI), ROOT (LEM/PSI), WKM (outdated ascii format), MUD (TRIUMF), NeXus (ISIS).Comment: 4 pages, 4 figure

Abelian ideals in a Borel subalgebra of a complex simple Lie algebra

Let g be a complex simple Lie algebra and b a fixed Borel subalgebra of g. We shall describe the abelian ideals of b in a uniform way, that is, independent of the classification of complex simple Lie algebras.Comment: 43 pages, LaTeX2e, youngtab.st

Cloning and Characterization of the Cell Wall Acting Enzyme CD1034 from the Pathogen Clostridium difficile

The manifestation of multidrug resistance in bacteria over the past several decades has resulted in one of the foremost challenges in the management of infectious diseases. The question arises, “How do we address this growing problem?” One solution to stem the growing rise in antimicrobial resistance is to investigate new targets, while another approach is to re-examine classical antibacterial targets with a fresh perspective. The aim of this paper is to begin the process of antibacterial development for the pathogen Clostridium difficile by characterizing the cell wall acting glucosaminidase CD1034. It is inunderstanding how CD1034 functions biochemically that it can be targeted for antibacterial development. The gene CD1034 was successfully cloned and sequenced in this project. Results have implications in the further development of an antibiotic for C. difficile and other Gram-positive pathogens