The Pulsation of Chi Cygni Imaged by Optical Interferometry; a Novel Technique to Derive Distance and Mass of Mira Stars

We present infrared interferometric imaging of the S-type Mira star Chi Cygni. The object was observed at four different epochs in 2005-2006 with the IOTA optical interferometer (H band). Images show up to 40% variation in the stellar diameter, as well as significant changes in the limb darkening and stellar inhomogeneities. Model fitting gave precise time-dependent values of the stellar diameter, and reveals presence and displacement of a warm molecular layer. The star radius, corrected for limb darkening, has a mean value of 12.1 mas and shows a 5.1mas amplitude pulsation. Minimum diameter was observed at phase 0.94+/-0.01. Maximum temperature was observed several days later at phase 1.02+/-0.02. We also show that combining the angular acceleration of the molecular layer with CO (Delta v = 3) radial velocity measurements yields a 5.9+/-1.5 mas parallax. The constant acceleration of the CO molecules -- during 80% of the pulsation cycle -- lead us to argument for a free-falling layer. The acceleration is compatible with a gravitational field produced by a 2.1(+1.5/-0.7) solar mass star. This last value is in agreement with fundamental mode pulsator models. We foresee increased development of techniques consisting in combining radial velocity with interferometric angular measurements, ultimately allowing total mapping of the speed, density, and position of the diverse species in pulsation driven atmospheres.Comment: 36 pages, accepted in Ap

Near-infrared IOTA interferometry of the symbiotic star CH Cyg

We present observations of the symbiotic star CH Cyg with a new JHK-band beam combiner mounted to the IOTA interferometer. The new beam combiner consists of an anamorphic cylindrical lens system and a grism, and allows the simultaneous recording of spectrally dispersed J-, H- and K-band Michelson interferograms. The observations of CH Cyg were conducted on 5, 6, 8 and 11 June 2001 using baselines of 17m to 25m. From the interferograms of CH Cyg, J-, H-, and K-band visibility functions can be determined. Uniform-disk fits to the visibilities give, e.g., stellar diameters of (7.8 ± 0.6) mas and (8.7 ± 0.8) mas in H and K, respectively. Angular stellar filter radii and Rosseland radii are derived from the measured visibilities by fitting theoretical center-to-limb intensity variations (CLVs) of Mira star models. The available HIPPARCOS parallax of CH Cyg allows us to determine linear radii. For example, on the basis of the K-band visibility, Rosseland radii in the range of 214 to 243 solar radii can be derived utilizing CLVs of different fundamental mode Mira models as fit functions. These radii agree well within the error bars with the corresponding theoretical model Rosseland radii of 230 to 282 solar radii. Models of first overtone pulsators are not in good agreement with the observations. The wavelength dependence of the stellar diameter can be well studied by using visibility ratios V(λ1)/V(λ2) since ratios of visibilities of different spectral channels can be measured with higher precision than absolute visibilities. We found that the 2.03 μm uniform disk diameter of CH Cyg is approximately 1.1 times larger than the 2.15 μm and 2.26 μm uniform-disk diameter

Tema 14: Aglomerados de estrelas

We present observations of the symbiotic star CH Cyg with a new JHK-band beam combiner mounted to the IOTA interferometer. The new beam combiner consists of an anamorphic cylindrical lens system and a grism, and allows the simultaneous recording of spectrally dispersed J-, H- and K-band Michelson interferograms. The observations of CH Cyg were conducted on 5, 6, 8 and 11 June 2001 using baselines of 17m to 25m. From the interferograms of CH Cyg, J-, H-, and K-band visibility functions can be determined. Uniform-disk fits to the visibilities give, e.g., stellar diameters of (7.8 ± 0.6) mas and (8.7 ± 0.8) mas in H and K, respectively. Angular stellar filter radii and Rosseland radii are derived from the measured visibilities by fitting theoretical center-to-limb intensity variations (CLVs) of Mira star models. The available HIPPARCOS parallax of CH Cyg allows us to determine linear radii. For example, on the basis of the K-band visibility, Rosseland radii in the range of 214 to 243 solar radii can be derived utilizing CLVs of different fundamental mode Mira models as fit functions. These radii agree well within the error bars with the corresponding theoretical model Rosseland radii of 230 to 282 solar radii. Models of first overtone pulsators are not in good agreement with the observations. The wavelength dependence of the stellar diameter can be well studied by using visibility ratios V(λ1)/V(λ2) since ratios of visibilities of different spectral channels can be measured with higher precision than absolute visibilities. We found that the 2.03 μm uniform disk diameter of CH Cyg is approximately 1.1 times larger than the 2.15 μm and 2.26 μm uniform-disk diameter

Potentiation of tumor necrosis factor -induced secreted phospholipase A2 (sPLA2)-IIA expression in mesangial cells by an autocrine loop involving sPLA2 and peroxisome Proliferator-activated Receptor Activation

In rat mesangial cells, exogenously added secreted phospholipases A2 (sPLA2s) potentiate the expression of pro-inflammatory sPLA2-IIA first induced by cytokines like tumor necrosis factor-? (TNF?) and interleukin-1?. The transcriptional pathway mediating this effect is, however, unknown. Because products of PLA2 activity are endogenous activators of peroxisome proliferator-activated receptor ? (PPAR?, we postulated that sPLA2s mediate their effects on sPLA2-IIA expression via sPLA2 activity and subsequent PPAR? activation. This study shows that various sPLA2s, including venom enzymes, human sPLA2-IIA, and wild-type and catalytically inactive H48Q mutant of porcine pancreatic sPLA2-IB, enhance the TNF?-induced sPLA2-IIA expression at the mRNA and protein levels. In cells transfected with luciferase sPLA2-IIA promoter constructs, sPLA2s are active only when the promoter contains a functional PPRE-1 site. The effect of exogenous sPLA2s is also blocked by the PPAR? inhibitor MK886. Interestingly, the expression of sPLA2-IIA induced by TNF? alone is also attenuated by MK886, by the sPLA2-IIA inhibitor LY311727, by heparinase, which prevents the binding of sPLA2-IIA to heparan sulfate proteoglycans, and by the specific cPLA2-? inhibitor pyrrolidine-1. Together, these data indicate that sPLA2-IIA released from mesangial cells by TNF? stimulates its own expression via an autocrine loop involving cPLA2 and PPAR?. This signaling pathway is also used by exogenously added sPLA2s including pancreatic sPLA2-IB and is distinct from that used by TNF?

Hypoxia and lipid signaling

Sufficient oxygen supply is crucial for the development and physiology of mammalian cells and tissues. When simple diffusion of oxygen becomes inadequate to provide the necessary flow of substrate, evolution has provided cells with tools to detect and respond to hypoxia by upregulating the expression of specific genes, which allows an adaptation to hypoxia-induced stress conditions. The modulation of cell signaling by hypoxia is an emerging area of research that provides insight into the orchestration of cell adaptation to a changing environment. Cell signaling and adaptation processes are often accompanied by rapid and/or chronic remodeling of membrane lipids by activated lipases. This review highlights the bi-directional relation between hypoxia and lipid signaling mechanisms