5 research outputs found
Match-Fixing en tenis : análisis sobre el amaño de partidos en el mundo del tenis
El presente artículo expone, a partir de un hipotético caso práctico, la situación de los amaños deportivos o match-fixing en el ámbito del tenis profesional y cuáles son, a nivel jurídico, la normativa y sanciones que se establecen para los supuestos casos de amaños deportivos
Additional file 4: of C-Myc-dependent repression of two oncogenic miRNA clusters contributes to triptolide-induced cell death in hepatocellular carcinoma cells
Figure S2. Triptolide did not cause obvious liver or kidney damages in nude mice. a,b The concentrations of serum alanine aminotransferase (ALT) and aspartate aminotransferase (AST), two common indicators of liver function, were measured by colorimetric analysis. c,d The concentrations of blood urea nitrogen (BUN) and creatinine (Creat), two common indicators of kidney function, were measured by colorimetric analysis. (TIFF 755Â kb
Additional file 7: of C-Myc-dependent repression of two oncogenic miRNA clusters contributes to triptolide-induced cell death in hepatocellular carcinoma cells
Table S3. Clinicopathologic characteristics of HCC subtypes defined by miR-17 expression. (DOC 46Â kb
Additional file 5: of C-Myc-dependent repression of two oncogenic miRNA clusters contributes to triptolide-induced cell death in hepatocellular carcinoma cells
Figure S3. Triptolide reduced pri-miR-17-92 and pri-miR-106b-25 expression in vivo. Xenografted tumors were obtained from nude mice treated with DMSO and triptolide, respectively (nâ=â3), and total RNA was extracted using Trizol reagent. The expression of pri-miR-17-92 and pri-miR-106b-25 was quantified by qRT-PCR. (TIFF 736Â kb
Additional file 2: of C-Myc-dependent repression of two oncogenic miRNA clusters contributes to triptolide-induced cell death in hepatocellular carcinoma cells
Table S1. Summary of clinicopathologic features. (DOC 49Â kb