215 research outputs found

    Revolution Unfinished: Comparing Collective Memory in the Kyiv Post and RT

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    From November 2013 – February 2014, Ukraine’s Independence Square (or Maidan) became the site of revolution. The Maidan Revolution culminated in the deaths of over 100 protesters and law enforcement, and the removal of former President Viktor Yanukovych. Subsequently, several studies have observed how Maidan is being remembered (see Kozachenko, 2020; Nuzov, 2016; Shevel, 2016). I rely on the perspectives of Maurice Halbwachs (2011/1925) on collective memory, Robin Wagner-Pacifici (1996; 2010; 2017) on events, and various perspectives on media framing in journalism. This paper builds upon existing literature by exploring the formation of collective memory in 52 newspaper articles from the Kyiv Post and RT. From my findings I argue both news outlets accept Maidan as part of their taken for granted memory. Both outlets primarily frame Maidan using national memory narratives. Like Kozachenko (2020), I observed the presence of Ukrainophile and Sovietophile historical frames. Though both news outlets frame Maidan as a failed revolution, I argue Maidan is characterized differently by the Kyiv Post and RT. Whereas, the Kyiv Post frames Maidan as a tragic unfinished revolution, RT constructs a framing of Maidan as a coup which allows them to compare it to current events. Presented in absentia on April 27, 2020 at "Student Research Day" at MacEwan University in Edmonton, Alberta. (Conference cancelled) Faculty Mentor: Jeff Stepnisky  Department: Sociology&nbsp

    Studyof the effects in the dog of ixodes Holocyclus

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    Lesão na camada glomerular do bulbo olfatório interfere na olfação e nos comportamentos tipo-depressivos em modelo animal de parkinsonismo

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    Orientador: Prof. Dr. Marcelo de Meira Santos LimaDissertação (mestrado) - Universidade Federal do Paraná, Setor de Ciências Biológicas, Programa de Pós-Graduação em Fisiologia. Defesa : Curitiba, 26/05/2017Inclui referênciasResumo: Os distúrbios olfatórios e a depressão são sintomas não-motores comumente relatados por indivíduos com a Doença de Parkinson (DP), sendo manifestados anos antes dos sintomas motores. Os mecanismos subjacentes a essas manifestações clínicas não estão completamente elucidados, no entanto, o desequilíbrio na neurotransmissão dopaminérgica parece ter um papel importante nesse contexto. Em indivíduos e em modelos animais da DP, é observado um aumento dos interneurônios dopaminérgicos da camada glomerular de bulbo olfatório (BO-gl), o que poderia contribuir para o prejuízo olfatório. Além disso, o desequilíbrio neuronal no BO está relacionado a sintomas depressivos, como demonstrado pela bulbectomia olfatória química. Dessa forma, nós hipotetizamos que a redução no número e/ou densidade dos neurônios dopaminérgicos presentes no BO poderia promover uma melhora olfatória e, em contrapartida, acentuaria o comportamento tipo-depressivo no modelo de Parkinsonismo induzido por 6-hidroxidopamina (6- OHDA). Assim, foi feita a administração intranigral de 6-OHDA e, posteriormente, a injeção da mesma droga na BO-gl em ratos, sendo esses avaliados em dois diferentes tempos (7 e 14 dias após a infusão intranigral de 6-OHDA). Observamos que, após 7 dias, o grupo lesionado apenas na SNpc apresentou comprometimento olfatório, assim como o grupo com a lesão apenas no BO. No entanto, a combinação das lesões na SNpc e BO foi capaz de reverter esse padrão, fazendo que os animais discriminassem ambos os compartimentos. Em relação ao comportamento tipodepressivo, observamos que a lesão na SNpc promoveu comportamento tipodepressivo, sendo esse acentuado após uma lesão dupla. A influência da SNpc e do BO no comportamento tipo-depressivo é corroborada por correlações entre parâmetros depressivos e neurônios TH-ir da SNpc em ambos tempos avaliados e com neurônios TH-ir do BO na avaliação de 14 dias. Esses resultados indicam um papel crucial do sistema dopaminérgico no BO e na SNpc tanto na modulação da olfação quanto no comportamento tipo-depressivo em um modelo de Parkinsonismo induzido por 6-OHDA. Palavras-chave: Doença de Parkinson, depressão, distúrbios olfatórios, sistema dopaminérgico.Abstract: Olfactory impairments and depressive behavior are commonly reported by individuals with Parkinson's disease (PD) being manifested before motor symptoms. The mechanisms underlying these clinical manifestations are not fully elucidated, however, the imbalance in dopaminergic neurotransmission seems to play an important role in the context. In individuals and animal models of PD, an increase in the dopaminergic interneurons of the glomerular layer in olfactory bulb (OB-gl) is observed, which could contribute to the olfactory impairment. In addition, neuronal imbalance in OB is related to depressive symptoms, as demonstrated by chemical olfactory bulbectomy. In view of that, we hypothesized that the reduction in the number and / or density of dopaminergic neurons present in OB could promote an olfactory improvement and, in contrast, would accentuate the depressive-like behavior in the 6-hydroxydopamine (6-OHDA) induced Parkinsonism model. Thereby, intranigral administration of 6-OHDA and subsequent injection of the same drug into OB-gl in rats were performed, being evaluated at two different time-points (7 and 14 days after intranigral infusion of 6-OHDA). We observed that, after 7 days, the group only injured in the SNpc presented olfactory impairment, as the group with the lesion only in the BO. However, the combination of the lesions in SNpc and BO was able to reverse this pattern, causing the animals to discriminate both compartments. In relation to depressive-like behaviors, we observed that the SNpc injury promoted depressive-like behavior, being accentuated after a double injury. The SNpc and OB influence on depressive-like behavior is corroborated by correlations between depressive parameters and SNpc TH-ir neurons at both evaluated times and with OB-gl TH-ir neurons in later time-point. These results indicate a crucial role of dopaminergic system in OB and SNpc in olfaction and humor modulation in a model of Parkinsonism induced by 6-OHDA. Keywords: Parkinson's disease, depression, olfactory disorders, dopaminergic system

    "School Strike 4 Climate": Social Media and the International Youth Protest on Climate Change

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    Beginning in 2018, youth across the globe participated in protest activities aimed at encouraging government action on climate change. This activism was initiated and led by Swedish teenager, Greta Thunberg. Like other contemporary movements, the School Strike 4 Climate used social media. For this article, we use Twitter trace data to examine the global dynamics of the student strike on March 15, 2019. We offer a nuanced analysis of 993 tweets, employing a combination of qualitative and quantitative analysis. Like other movements, the primary function of these tweets was to share information, but we highlight a unique type of information shared in these tweets - documentation of local events across the globe. We also examine opinions shared about youth, the tactic (protest/strike), and climate change, as well as the assignment of blame on government and other institutions for their inaction and compliance in the climate crisis. This global climate strike reflects a trend in international protest events, which are connected through social media and other digital media tools. More broadly, it allows us to rethink how social media platforms are transforming political engagement by offering actors - especially the younger generation - agency through the ability to voice their concerns to a global audience

    Evaluation of butorphanol, medetomidine and midazolam as a reversible narcotic combination in free-ranging African lions (Panthera leo)

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    Objective To evaluate the effects of the combination butorphanol, medetomidine and midazolam (BMM) and its reversibility in lions. Study design Prospective clinical trial. Animals Thirty free-ranging lions, 10 male and 20 female, weighing 81–210 kg. Methods Lions were immobilised with butorphanol mean 0.31 ± SD 0.034 mg kg-1, medetomidine 0.052 ± 0.006 mg kg-1, midazolam 0.21 ± 0.024 mg kg-1 and hyaluronidase 1250 IU administered intramuscularly with a dart gun. Upon recumbency, physiological parameters and anaesthetic depth were monitored 10–15 minutes after darting (T1) and repeated every 10 minutes for a further 30 minutes (T2, T3, T4). Arterial blood gas analyses were performed at T1 and T4. At the end of the procedure, 45–60 minutes after initial darting, immobilisation was reversed with naltrexone 0.68 ± 0.082 mg kg-1, atipamezole 0.26 ± 0.031 mg kg-1, and flumazenil 0.0032 ± 0.0007 mg kg-1 administered intravenously and subcutaneously. Results The BMM combination rapidly induced immobilisation and lateral recumbency was reached within 7.25 ± 2.3 minutes. Median induction score [scored 1 (excellent) to 4 (poor)] was 1.4 (range 1–2). Cardio-respiratory parameters were stable. Heart rate varied from 32 to 72 beats per minute, respiratory rate from 14 to 32 breaths minute-1 and rectal temperature from 36.6 to 40.3 C. No sudden arousals were observed. Arterial blood gas analyses revealed a mean pH of 7.33, PaCO2 of 33 mmHg and PaO2 of 87 mmHg. Mild to moderate hypoxemia was seen in four lions. Recovery was smooth and lions were walking within 4.4 ± 4.25 minutes. Median recovery score [scored 1 (excellent) to 4 (poor)] was 1.3 (range 1–2). Conclusion and clinical relevance The drug combination proved to be effective in immobilising freeranging healthy lions of both sexes with minimal cardio-respiratory changes
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