A Study on the Effects of Personal Characteristics on Investment Behavior

[[abstract]]Finance behavior from psychological investors to carry out the decision-making process to explore, that investment management is not entirely of, in the process of investment, may be psychological bias on the judge or decision-making biases. And different types of investors, conditions because of their different personal, clinical decision-making when faced Consideriation different factors, Resulting in different investment decisions. Since the 80's, on the field of behavioral finance research a great deal of attention, input caused by many scholars because the criteria do not have a fully predict or measure of human behavior, for the financial market into a number of variables. This study attempted to drawn from the experience of daily life, observation of investors will affect the life style, to explain the investment behavior of non-rational decision-making, traditional financial theory and the assumption that investors are rational, investors not to consider the psychological factors(Lead to acts of irrational decision-making)questioned, and achieving the following objectives: First, empirical research by investors made the primary investment decision-making information. Second, identify the personal qualities of investors in their investment decision-making the impact assessment process. Third, investigate the investor's investment decision-making process to assess the impact on investment behavior. The results found that, risk attitude of decision-making process of investment analysis, commercial message of risk and risk preferences tend to accept the tendency of positive correlation, risk aversion and the tendency to negative correlation. Familiar with the degree of commodity risk and risk preferences tend to accept the tendency of positive correlation. The confidence of investors against the risk of preference was positively related to the tendency. Intent of the risk preferences of investors and risk tend to accept the tendency of positive correlation

AgSbS2 semiconductor-sensitized solar cells

We present a ternary semiconductor nanoparticle sensitizer – AgSbS2 – for solar cells. AgSbS2 nanoparticles were grown using a two-stage successive ionic layer adsorption and reaction process. First, Ag2S nanoparticles were grown on the surface of a nanoporous TiO2 electrode. Secondly, a Sb–S film was coated on top of the Ag2S. The double-layered structure was transformed into AgSbS2 nanoparticles ~ 40 nm in diameter, after post-deposition heating at 350 °C. The AgSbS2-sensitized TiO2 electrodes were fabricated into liquid-junction solar cells. The best cell yielded a power conversion efficiency of 0.34% at 1 sun and 0.42% at 0.1 sun. The external quantum efficiency (EQE) spectrum covered the range of 380–680 nm with a maximal EQE of 10.5% at λ = 470 nm. The method can be applied to grow other systems of ternary semiconductor nanoparticles for solar absorbers

Solitary plexiform neurofibroma of the forehead: A rare and unusual presentation

Plexiform neurofibromas are benign tumors of the peripheral nerves and are usually considered pathognomonic of neurofibromatosis type 1. We discuss the case of a child who presented with a forehead mass. Pathologic examination revealed a plexiform neurofibroma of the supraorbital nerve. No other signs or symptoms of neurofibromatosis were identified. Although rare, plexiform neurofibromas may rarely occur as solitary lesions not associated with the neurofibromatosis spectrum. It can be seen in unusual anatomic location such as the supraorbital or forehead region and should be considered in the diagnosis of soft tissue facial tumors

Band gap engineered ternary semiconductor PbxCd1-xS: Nanoparticle-sensitized solar cells with an efficiency of 8.5% under 1% sun-A combined theoretical and experimental study

We report the synthesis and photovoltaic properties of a ternary metal sulfide alloyed semiconductor PbxCd1−xS prepared by the two-stage sequential ionic layer adsorption reaction. The synthesized PbxCd1−xS nanoparticles (NPs) retain the hexagonal structure of the CdS host with Pb substituting a fraction of the Cd atom (x = 0-0.17). Band structures of PbxCd1−xS with various Pb contents x were calculated using the complementary density functional theory (DFT) method. Optical, quantum efficiency, cyclic voltammetry measurements, and band structure calculation revealed that the band gap of PbxCd1−xS decreased with increasing x, resulting in an increased optical absorption band from 500 to 720 nm (1.73-2.44 eV) for x = 0 to 0.17. Solid-state PbxCd1−xS semiconductor nanoparticle-sensitized solar cells (NSSCs) were fabricated from the synthesized NPs using spiro-OMeTAD as the hole-transporting material. The best Pb0.05Cd0.95S cell yielded a power conversion efficiency (PCE) of 3.67%, a Voc of 0.70 V, and a fill factor (FF) of 62.8% under 1 sun. The PCE increased to 5.93% under a reduced light intensity of 0.1 sun and further increased to 8.48% under 0.01 sun. The external quantum efficiency (EQE) spectrum covers the spectral range of 300 to 730 nm with a maximal EQE of 82% at λ = 580 nm. The PCE over 8% can be categorized into a high-efficiency NSSCs. In addition, the Voc of 0.70 V is a relatively high Voc among all NSSCs. The high PCE and Voc suggest that PbxCd1−xS has potential to be an efficient solar absorber

EGCG Inhibits Transforming Growth Factor-β-Mediated Epithelial-to-Mesenchymal Transition via the Inhibition of Smad2 and Erk1/2 Signaling Pathways in Nonsmall Cell Lung Cancer Cells

Transforming growth factor-β (TGF-β)-mediated epithelial mesenchymal transition (EMT) of human lung cancer cells may contribute to lung cancer metastasis. It has been reported that EGCG can inhibit tumorigenesis and cancer cell growth in lung cancer; however, the effect of EGCG on EMT in nonsmall cell lung cancer (NSCLC) cells has not been investigated. In this study, we found that NSCLC cells A549 and H1299 were converted to the fibroblastic phenotype in response to TGF-β. Epithelial marker E-cadherin was down-regulated, and mesenchymal marker vimentin was up-regulated simultaneously. Our results illustrated that TGF-β was able to induce EMT in NSCLC cells, and EGCG would reverse TGF-β-induced morphological changes, up-regulate the expression of E-cadherin, and down-regulate the expression of vimentin. Immunofluorescent staining also demonstrated that E-cadherin was up-regulated and that vimentin was down-regulated by EGCG pretreatment. Moreover, wound-healing and the in vitro invasion assay showed that EGCG could inhibit TGF-β-induced migration and invasion of NSCLC cells. By using the dual-luciferase reporter assay, we demonstrated that EGCG inhibited TGF-β-induced EMT at the transcriptional level. EGCG decreased the phosphorylation of Smad2 and Erk1/2, inhibited the nuclear translocation of Smad2, and repressed the expression of transcription factors ZEB1, Snail, Slug, and Twist, and up-regulated the expression of E-cadherin. In summary, our results suggest that EGCG can inhibit TGF-β-induced EMT via down-regulation of phosphorylated Smad2 and Erk1/2 in NSCLC cells