575 research outputs found

    Transition to Superfluid Turbulence

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    Turbulence in superfluids depends crucially on the dissipative damping in vortex motion. This is observed in the B phase of superfluid 3He where the dynamics of quantized vortices changes radically in character as a function of temperature. An abrupt transition to turbulence is the most peculiar consequence. As distinct from viscous hydrodynamics, this transition to turbulence is not governed by the velocity-dependent Reynolds number, but by a velocity-independent dimensionless parameter 1/q which depends only on the temperature-dependent mutual friction -- the dissipation which sets in when vortices move with respect to the normal excitations of the liquid. At large friction and small values of 1/q < 1 the dynamics is vortex number conserving, while at low friction and large 1/q > 1 vortices are easily destabilized and proliferate in number. A new measuring technique was employed to identify this hydrodynamic transition: the injection of a tight bundle of many small vortex loops in applied vortex-free flow at relatively high velocities. These vortices are ejected from a vortex sheet covering the AB interface when a two-phase sample of 3He-A and 3He-B is set in rotation and the interface becomes unstable at a critical rotation velocity, triggered by the superfluid Kelvin-Helmholtz instability.Comment: Short review; to be published in Journal of Low Temperature Physics (2006

    The dynamics of vortex generation in superfluid 3He-B

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    A profound change occurs in the stability of quantized vortices in externally applied flow of superfluid 3He-B at temperatures ~ 0.6 Tc, owing to the rapidly decreasing damping in vortex motion with decreasing temperature. At low damping an evolving vortex may become unstable and generate a new independent vortex loop. This single-vortex instability is the generic precursor to turbulence. We investigate the instability with non-invasive NMR measurements on a rotating cylindrical sample in the intermediate temperature regime (0.3 - 0.6) Tc. From comparisons with numerical calculations we interpret that the instability occurs at the container wall, when the vortex end moves along the wall in applied flow.Comment: revised & extended version. Journal of Low Temperature Physics, accepted (2008

    Computational and experimental characterization of 3D-printed PCL structures toward the design of soft biological tissue scaffolds

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    Degradable porous polymeric structures are attractive candidates for biological tissue scaffolds, and adequate mechanical, transport, chemical and biological properties determine their functionality. Aside from the properties of polymer-based materials, the scaffold's meso-structure controls its elasticity at the organ length-scale. This study investigated the effect of the meso-structure on scaffolds' mechanical and transport properties using finite element analysis (FEA) and computational fluid dynamics (CFD). A number of poly (ε-caprolactone) (PCL) - based scaffolds were 3D printed, analyzed by microcomputed tomography (micro-CT) and mechanically tested. We found that the gradient (G) and gradient and staggered (GS) meso-structure designs led to a higher scaffold permeability, a more homogeneous flow inside the scaffold, and a lower wall shear stress (WSS) in comparison with the basic (B) meso-structure design. The GS design resulted in scaffold stiffness as low as 1.07/0.97 MPa under compression/tension, figures that are comparative with several soft tissues. Image processing of micro-CT data demonstrated that the imposed meso-structures could have been adequately realized through 3D printing, and experimental testing validated FEA analysis. Our results suggest that the properties of 3D-printed PCL-based scaffolds can be tuned via meso-structures toward soft tissue engineering applications. The biological function of designed scaffolds should be further explored in-situ studies.publishedVersio

    Classical and quantum regimes of the superfluid turbulence

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    We argue that turbulence in superfluids is governed by two dimensionless parameters. One of them is the intrinsic parameter q which characterizes the friction forces acting on a vortex moving with respect to the heat bath, with 1/q playing the same role as the Reynolds number Re=UR/\nu in classical hydrodynamics. It marks the transition between the "laminar" and turbulent regimes of vortex dynamics. The developed turbulence described by Kolmogorov cascade occurs when Re >> 1 in classical hydrodynamics, and q << 1 in the superfluid hydrodynamics. Another parameter of the superfluid turbulence is the superfluid Reynolds number Re_s=UR/\kappa, which contains the circulation quantum \kappa characterizing quantized vorticity in superfluids. This parameter may regulate the crossover or transition between two classes of superfluid turbulence: (i) the classical regime of Kolmogorov cascade where vortices are locally polarized and the quantization of vorticity is not important; and (ii) the quantum Vinen turbulence whose properties are determined by the quantization of vorticity. The phase diagram of the dynamical vortex states is suggested.Comment: 12 pages, 1 figure, version accepted in JETP Letter

    Structure of surface vortex sheet between two rotating 3He superfluids

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    We study a two-phase sample of superfluid 3He where vorticity exists in one phase (3He-A) but cannot penetrate across the interfacial boundary to a second coherent phase (3He-B). We calculate the bending of the vorticity into a surface vortex sheet on the interface and solve the internal structure of this new type of vortex sheet. The compression of the vorticity from three to two dimensions enforces a structure which is made up of half-quantum units, independently of the structure of the source vorticity in the bulk. These results are consistent with our NMR measurements.Comment: 4 pages, 4 figure

    Two Drinking Water Outbreaks Caused by Wastewater Intrusion Including Sapovirus in Finland

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    Drinking water outbreaks occur worldwide and may be caused by several factors, including raw water contamination, treatment deficiencies, and distribution network failure. This study describes two drinking water outbreaks in Finland in 2016 (outbreak I) and 2018 (outbreak II). Both outbreaks caused approximately 450 illness cases and were due to drinking water pipe breakage and subsequent wastewater intrusion into the distribution system. In both outbreaks, the sapovirus was found in patient samples as the main causative agent. In addition, adenoviruses and Dientamoeba fragilis (outbreak I), and noroviruses, astroviruses, enterotoxigenic and enterohemorragic Escherichia coli (ETEC and EHEC, respectively) and Plesiomonas shigelloides (outbreak II) were detected in patient samples. Water samples were analyzed for the selected pathogens largely based on the results of patient samples. In addition, traditional fecal indicator bacteria and host-specific microbial source tracking (MST) markers (GenBac3 and HF183) were analyzed from water. In drinking water, sapovirus and enteropathogenic E. coli (EPEC) were found in outbreak II. The MST markers proved useful in the detection of contamination and to ensure the success of contaminant removal from the water distribution system. As mitigation actions, boil water advisory, alternative drinking water sources and chlorination were organized to restrict the outbreaks and to clean the contaminated distribution network. This study highlights the emerging role of sapoviruses as a waterborne pathogen and warrants the need for testing of multiple viruses during outbreak investigation

    Two Drinking Water Outbreaks Caused by Wastewater Intrusion Including Sapovirus in Finland

    Get PDF
    Drinking water outbreaks occur worldwide and may be caused by several factors, including raw water contamination, treatment deficiencies, and distribution network failure. This study describes two drinking water outbreaks in Finland in 2016 (outbreak I) and 2018 (outbreak II). Both outbreaks caused approximately 450 illness cases and were due to drinking water pipe breakage and subsequent wastewater intrusion into the distribution system. In both outbreaks, the sapovirus was found in patient samples as the main causative agent. In addition, adenoviruses and Dientamoeba fragilis (outbreak I), and noroviruses, astroviruses, enterotoxigenic and enterohemorragic Escherichia coli (ETEC and EHEC, respectively) and Plesiomonas shigelloides (outbreak II) were detected in patient samples. Water samples were analyzed for the selected pathogens largely based on the results of patient samples. In addition, traditional fecal indicator bacteria and host-specific microbial source tracking (MST) markers (GenBac3 and HF183) were analyzed from water. In drinking water, sapovirus and enteropathogenic E. coli (EPEC) were found in outbreak II. The MST markers proved useful in the detection of contamination and to ensure the success of contaminant removal from the water distribution system. As mitigation actions, boil water advisory, alternative drinking water sources and chlorination were organized to restrict the outbreaks and to clean the contaminated distribution network. This study highlights the emerging role of sapoviruses as a waterborne pathogen and warrants the need for testing of multiple viruses during outbreak investigation

    System size scaling of topological defect creation in a second-order dynamical quantum phase transition

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    We investigate the system size scaling of the net defect number created by a rapid quench in a second-order quantum phase transition from an O(N) symmetric state to a phase of broken symmetry. Using a controlled mean-field expansion for large N, we find that the net defect number variance in convex volumina scales like the surface area of the sample for short-range correlations. This behaviour follows generally from spatial and internal symmetries. Conversely, if spatial isotropy is broken, e.g., by a lattice, and in addition long-range periodic correlations develop in the broken-symmetry phase, we get the rather counterintuitive result that the scaling strongly depends on the dimension being even or odd: For even dimensions, the net defect number variance scales like the surface area squared, with a prefactor oscillating with the system size, while for odd dimensions, it essentially vanishes.Comment: 20 pages of IOP style, 6 figures; as published in New Journal of Physic
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