12 research outputs found

    Formation Of Groups As Psychological And Social Support In Epilepsy [formação De Grupos Como Suporte Psicológico E Social Na Epilepsia]

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    Epilepsy is a neurological condition that affects patients and their family behavior and well-being often leading to social withdrawn. For this reason, it is important that people learn about epilepsy, and which are the social implications on the lives of people with epilepsy stop them of being victims of the stigma in the community. This article aims to show the importance of working with groups in epilepsy. We try to provide basic guidelines towards achieving effective results. The group dynamics improves the interpersonal relationships, focusing the treatment of their problems and difficulties, especially in chronic diseases. Our experience has shown that this intervention has a great potential to provide well-being, reducing the people's anxiety and ultimately providing improvement in the quality of life and reduction of the felt stigma.103171174Fernandes, P.T., Souza, E.A.P., Identification of family variables in parent's groups of children with epilepsy (2001) Arquivos de Neuropsiquiatria, 59 (4), pp. 854-858Baker, G.A., The psychosocial burden of epilepsy (2002) Epilepsia, 43 (6), pp. 26-30www.aspebrasil.org, ASPEPalmini, A., Costa, J.C., Introdução à epileptologia clínica e classificação das epilepsias e crises epilépticas (1998) Fundamentos neurobiológicos das epilepsias: Aspectos clínicos e cirúrgicos, pp. 149-161. , Costa JC, Palmini A, Yacubian EMT, Cavalheiro EA, editors:, São Paulo. Lemos editorial;Guerreiro, C.A.M., Guerreiro, M.M., (1999) Epilepsia: O paciente otimamente controlado, , São Paulo: Lemos editorial;, 83pGoldstein, J., Seidenberg, M., Peterson, R., Fear of seizures and behavioral functioning in adults with epilepsy (1990) Journal of Epilepsy, 3, pp. 101-106Scambler, G., Hopkins, A., Being epileptic: Coming to terms with stigma (1986) Sociology of health and illness, 8, pp. 26-43Fernandes, P.T., Souza, E.A.P., Procedimento educativo na epilepsia infantil (2001) Estudos de Psicologia, 6 (1), pp. 115-120Baker, G.A., Jacoby, A., Buck, D., Stalgis, C., Monnet, D., Quality of Life of people with epilepsy: An European study (1997) Epilepsia, 38 (3), pp. 353-362Gumnit, R.J., (1997) Living well with epilepsy, , New York: Demos Vermande;, 249pGuerreiro, M.M., Silva, E.A., Scotoni, A.E., Souza, E.A.P., Qualidade de vida em epilepsia na infância (1994) Jornal da Liga Brasileira de Epilepsia, 7 (1), pp. 21-26Fernandes, P.T., Souza, E.A.P., Grupos de pais na epilepsia infantil: Uma visão psicológica (2003) Journal of Epilepsy and Clinical Neurophysiology, 9 (4), pp. 225-229Guerreiro, C.A.M., Guerreiro, M.M., Cendes, F., Lopes-Cendes, I., (1999) Epilepsia, , São Paulo, Lemos Editorial;Guerreiro CAM, Guerreiro MM. Epilepsia - 50 FAQ (Frequently Asked Questions). São Paulo, EPM (Editora de Produtos Médicos)2004Revistas, A.S.P.E., Sem Crise, , www.aspebrasil. org, Disponível emRevista ComCiência (Neurociências: Epilepsia 200234). Disponível em: 〈www.comciencia.br/reportagens/epilepsia

    Myoclonic Seizures In Symptomatic Temporal Lobe Epilepsy Of Childhood

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    Rationale and objective: Generalized epileptic manifestations may occur in symptomatic focal epilepsies in children, particularly in infants and pre-school children. Our aim here is to report two patients with myoclonic seizures associated with temporal lobe epilepsy. Methodology: Case report. Results: Two children with tumor in temporal lobe presented myoclonic seizures or spasms since infancy besides other types of seizures. Conclusion: Our findings support the idea that myoclonic seizures may be generalized manifestations of focal insults to the CNS, and this pattern may be an age-dependent specific response of a developing brain to a severe insult.1114547Faught, E., Clinical presentations and phenomenology of myoclonus (2003) Epilepsia, 44 (SUPPL. 11), pp. 7-12Hallet, M., Myoclonus and myoclonic syndromes (1997) Epilepsy: A comprensive textbook, pp. 2717-2723. , Engel J, Pedley, TA, editores, Philadelphia: Lippincott- Raven;Nordli, D.R., Kurada, M.M., Hirsch, L.J., The ontogeny of partial seizures in infants and young children (2001) Epilepsia, 42 (8), pp. 986-990Fogarasi, A., Jokeit, H., Feveret, E., Jansky, J., Tuxhorn, J., The effect of age on seizure semiology in childhood temporal lobe epilepsy (2002) Epilepsia, 43 (6), pp. 638-643Franzon, R.C., Montenegro, M.A., Guimarães, C.A., Guerreiro, C.A.M., Cendes, F., Guerreiro, M.M., Clinical, electroencephalographic and behavioral features of temporal lobe epilepsy in chidhood (2004) J Child Neurol, 19 (6), pp. 418-423Mimaki, T., Ono, J., Yabuuchi, H., Temporal lobe astrocytoma with infantile spasms (1983) Ann Neurol, 14, pp. 695-696Singer, W., Haller, J., Mills, C., Rabe, E., The value of neuroradiology in infantile spasms (1982) Pediatrics, 100, pp. 47-50Palm, D.G., Brandt, M., Korinthenberg, R., West syndrome and Lennox-Gastaut syndrome in children with porencephalic cysts: Long-term follow-up after neurosurgical treatment (1988) The Lennox-Gastaut syndrome, pp. 419-426. , Niedermeyer E, Degn R, editores, New York: Alan R. Liss;Chugani, H.T., Shields, D., Shewmon, D.A., Olson, D.M., Phelps, M.E., Peacock, W.J., Infantile Spasms: I. PET Identifies Focal Cortical Dysgenesis in Cryptogenic Cases for Surgical Treatment (1990) Ann Neurol, 27, pp. 406-413Chugani, H.T., Infantile Spasms (1995) Neurology, 8, pp. 139-144Miyazaki, M., Hashimoto, T., Fujii, E., Tayama, M., Kuroda, Y., Infantile spasms: Localized cerebral lesions on SPECT (1994) Epilepsia, 35, pp. 988-992Chugani, H.T., Silva, E., Chugani, D.C., Infantile Spasms: III. Prognostic Implications of Bitemporal Hypometabolism on Positron Emission Tomography (1996) Ann Neurol, 39, pp. 643-649Dalla Bernardina, B., Colamaria, V., Capoville, G., Epileptic syndromes and cerebral malformations in infancy: Multicentric study (1984) Boll Lega Ital Epil, 45-46, pp. 65-67Carrazana, E.J., Lombroso, C.T., Mikati, M., Helmers, S., Holmes, G.L., Facilitation of Infantile Spasms by partial seizures (1993) Epilepsia, 34, pp. 686-691Alvarez, L.A., Shinnar, S., Moshe, S.L., Infantile spasms due to unilateral cerebral infarcts (1987) Pediatrics, 79, pp. 1024-102

    Fighting Against The Stigma In Epilepsy By Awareness Through Out Of Media [combate Ao Estigma Na Epilepsia Pela Conscientização Através Da Mídia]

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    Epilepsy is still a stigmatizing condition nowadays. It is time to react and to bring down the prejudice. The goal of this paper is to provide an easy instrument for fighting against the stigma in epilepsy, through an appropriate expression and widespread information of this condition, so that the stigma can be minimized. The instrument used is part of the Kit anti-stigma CD, released in March, 2003, by ASPE (Assistência à Saúde de Pacientes com Epilepsia) and refers to a letter addressed to the media, seeking the promotion and awareness of epilepsy.103167170Jacoby, A., Stigma, epilepsy and quality of life (2002) Epilepsy & Behavior, 3, pp. S10-S20Baker, G.A., The psychosocial burden of epilepsy (2002) Epilepsia, 43 (6), pp. 26-30Guerreiro, C.A.M., Guereiro, M.M., (1999) Epilepsia - o paciente otimamente controlado, , São Paulo: Lemos Editorial;, 83pScambler, G., Hopkins, A., Generating a model of epileptic stigma: The role of quantitative analysis (1990) Soc Sci Med, 30 (11), pp. 1187-1194ASPE. CD Kit anti-estigma 2003Guerreiro, C.A.M., Guerreiro, M.M., Cendes, F., Lopes-Cendes, I., (1999) Epilepsia, , São Paulo: Lemos Editorial;Guerreiro CAM, Guerreiro MM. Epilepsia - 50 FAQ (Frequently Asked Questions). São Paulo: EPM (Editora de Produtos Médicos)2004Revista, A.S.P.E., Sem Crise, , www.aspebrasil. org, Disponível emRevista ComCiência (Neurociências: Epilepsia 200234). Disponível em: 〈www.comciencia.br/reportagens/epilepsia

    Idiopathic Generalized Epilepsies Misdiagnosed As Partial Epilepsies [epilepsias Generalizadas Idiopáticas Diagnosticadas Incorretamente Como Epilepsias Parciais]

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    Idiopathic generalized epilepsy (IGE) is often not recognized with serious consequences on the sufferers. We examined factors contributing to the missed diagnosis of IGE in 41 adults attending our epilepsy clinic with diagnosis of partial epilepsy who had semiology or EEG findings suggesting a possible differential diagnosis. After careful re-evaluation, the diagnosis of IGE was established in 25 patients: 22 (88%) with JME, one with juvenile absence, one with perioral myoclonia with absences, one with eyelid myoclonia with typical absences. Myoclonic jerks, the hallmark of the JME and other IGE, were not usually reported by patients or misdiagnosed as focal motor seizures. Brief and infrequent absence seizures and focal EEG abnormalities were other factors contributing to not recognizing JME. All 25 patients did not achieve seizure control before re-evaluation of diagnosis. After appropriate diagnosis of IGE and change of AED to valproate or valproic acid, 19 (76%) became seizure free and six (24%) had a significant improvement on seizure control. Association with lamotrigine provided further improvement in three of these patients. An appropriate questioning to identify myoclonic and absence seizures and a proper interpretation in the context of whole clinical constellation are essential for a correct seizure classification and diagnosis of IGE in adults.603 B788796Proposal for revised clinical and electroencephalographic classification of epileptic seizures (1981) Epilepsia, 22, pp. 489-501Proposal for revised classification of epilepsies and epileptic syndromes (1989) Epilepsia, 30, pp. 389-399Panayiotopoulos, C.P., Tahan, R., Obeid, T., Juvenile myoclonic epilepsy: Factors of error involved in the diagnosis and treatment (1991) Epilepsia, 32, pp. 672-676Panayiotopoulos, C.P., Obeid, T., Waheed, G., Differentiation of typical absence seizures in epileptic syndromes: A video EEG study of 224 seizures in 20 patients (1989) Brain, 112, pp. 1039-1056Aliberti, V., Grunewald, R.A., Panayiotopoulos, C.P., Chroni, E., Focal electroencephalographic abnormalities in juvenile myoclonic epilepsy (1994) Epilepsia, 35, pp. 297-301Panayiotopoulos, C.P., Koutroumanidis, M., Giannakodimos, S., Agathonikou, A., Idiopathic generalised epilepsy in adults manifested by phantom absences, generalised tonic-clonic seizures, and frequent absence status (1997) J Neurol Neurosurg Psychiatry, 63, pp. 622-627Berkovic, S.F., Andermann, F., Andermann, E., Gloor, P., Concepts of absence epilepsies: Discrete syndromes or biological continuum (1987) Neurology, 37, pp. 993-1000Guideline seven: A proposal for standard montages to be used in clinical EEG (1994) J Clin Neurophysiol, 11, pp. 30-36Panayiotopoulos, C.P., Perioral myoclonia with abscences: A new syndrome (1994) Epileptic Seizures and Syndromes, pp. 143-153. , Wolf P. (ed) London: John Libbey & CompanyAppleton, R.E., Panayiotopoulos, C.P., Acomb, B.A., Beirne, M., Eyelid myoclonia with typical absences: An epilepsy syndrome (1993) J Neurol Neurosurg Psychiatry, 56, pp. 1312-1316Williamson, P., Spencer, S.S., Clinical and EEG features of complex partial seizures of extratemporal origin (1986) Epilepsia, 27 (SUPPL.), pp. 46-63Quesney, L.F., Cendes, F., Olivier, A., Dubeau, F., Andermann, F., Intracranial electroencephalographic investigation in frontal lobe epilepsy (1995) Adv Neurol, 66, pp. 243-258Panayiotopoulos, C.P., Chroni, E., Daskalopoulos, C., Baker, A., Rowlinson, S., Walsh, P., Typical absence seizures in adults: Clinical, EEG, video-EEG findings and diagnostic/syndromic considerations (1992) J Neurol Neurosurg Psychiatry, 55, pp. 1002-1008Gelisse, P., Genton, P., Thomas, P., Rev, M., Samuelian, J.C., Dravet, C., Clinical factors of drug resistance in juvenile myoclonic epilepsy (2001) J Neurol Neurosurg Psychiatry, 70, pp. 240-243Lombroso, C.T., Consistent EEG focalities detected in subjects with primary generalized epilepsies monitored for two decades (1997) Epilepsia, 38, pp. 797-812Janz, D., Juvenile myoclonic epilepsy: Epilepsy with impulsive petit mal (1989) Cleve Clin J Med, 56 (SUPPL.), pp. S23-33Yacubian, E.M.T., Epilepsia mioclonica juvenil (2000) Epilepsia, pp. 215-222. , Guerreiro CAM, Guerreiro MM, Cendes F, Lopes-Cendes I (eds.) São Paulo: Lemos EditorialMontalenti, E., Imperiale, D., Rovera, A., Bergamasco, B., Benna, P., Clinical features, EEG findings and diagnostic pitfalls in juvenile myoclonic epilepsy: A series of 63 patients (2001) J Neurol Sci, 184, pp. 65-70Janz, D., The idiopathic generalized epilepsies of adolescence with childhood and juvenile age of onset (1997) Epilepsia, 38, pp. 4-11Panayiotopoulos, C.P., Obeid, T., Tahan, A.R., Juvenile myoclonic epilepsy: A 5-year prospective study (1994) Epilepsia, 35, pp. 285-296Delgado-Escueta, A.V., Enrile-Bacsal, F., Juvenile myoclonic epilepsy of Janz (1984) Neurology, 34, pp. 285-294Capovilla, G., Rubboli, G., Beccaria, F., Lorenzetti, M.E., Montagnini, A., Resi, C., A clinical spectrum of the myoclonic manifestations associated with typical absences in childhood absence epilepsy. A video-polygraphic study (2001) Epileptic Disord, 3, pp. 57-62King, M.A., Newton, M.R., Jackson, G.D., Epileptology of the first-seizure presentation: A clinical, electroencephalographic, and magnetic resonance imaging study of 300 consecutive patients (1998) Lancet, 352, pp. 1007-1011Meencke, H.J., Janz, D., Neuropathological findings in primary generalized epilepsy: A study of eight cases (1984) Epilepsia, 25, pp. 8-21Opeskin, K., Kalnins, R.M., Halliday, G., Cartwright, H., Berkovic, S.F., Idiopathic generalized epilepsy: Lack of significant microdysgenesis (2000) Neurology, 55, pp. 1101-1106Genton, P., Gelisse, P., Thomas, P., Dravet, C., Do carbamazepine and phenytoin aggravate juvenile myoclonic epilepsy? (2000) Neurology, 55, pp. 1106-1109Fernando-Dongas, M.C., Radtke, R.A., Vanlandingham, K.E., Husain, A.M., Characteristics of valproic acid resistant juvenile myoclonic epilepsy (2000) Seizure, 9, pp. 385-388Genton, P., When antiepileptic drugs aggravate epilepsy (2000) Brain Dev, 22, pp. 75-80Liporace, J.D., Sperling, M.R., Dichter, M.A., Absence seizures and carbamazepine in adults (1994) Epilepsia, 35, pp. 1026-102

    Efeito de diferentes tamanhos de clareiras, sobre o crescimento e a mortalidade de espécies arbóreas, em Moju-PA Effect of diferente gap sizes on the growth and mortality of arboreal species, in Moju-PA

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    Crescimento e mortalidade de Sterculia pruriens, Vouacapoua americana, Jacaranda copaia, Protium paraensis, Newtonia suaveolens e Tabebuia serratifolia, considerando diferentes tamanhos de clareiras, foram avaliados em Moju PA(2º07'30" e 2º12'06" de latitude Sul e 48º46'57" e 48º48'30" de longitude a Oeste de Greenwitch). Selecionou-se nove clareiras da exploração florestal, que foram agrupadas em pequenas (200m²<Área<400m2­), médias(400m²<Área<600m²) e grandes (>600m²). Em seu torno instalou-se parcelas quadradas de cinco metros de lado, nas direções Norte, Sul, Leste e Oeste, onde foram plantados indivíduos da regeneração natural de espécies arbóreas. No centro de cada clareira foi instalada uma parcela de 5m X 5m como comparador. A média da mortalidade total foi de 46,9%, não havendo diferenças entre as clareiras pequenas(41,05%) e médias(43,86%), mas estas diferiram das grandes(54,96%). As clareiras pequenas são mais propícias para a maioria das espécies, exceto para J. copaia e N. suaveolens, cujas mortalidades foram menores nas clareiras médias. A mortalidade variou de 14,5%(S. pruriens) nas clareiras pequenas a 70,1%(V. americana) em clareiras grandes, sendo que S. pruriens mostrou menor mortalidade em todos os tamanhos de clareiras. As espécies morreram mais em clareiras grandes. A mortalidade está entre os valores encontrados na literatura, permitindo concluir que não se pode classificar com precisão as espécies em grupos ecológicos somente com base na mortalidade ou sobrevivência. Em termos de crescimento, os resultados indicam que os melhores sítios para desenvolvimento das espécies são as clareiras médias, seguidos pelas clareiras grandes e pequenas. Em termos gerais, a média de crescimento em altura foi de 11,34cm e de 0,11cm em diâmetro de base, com valores maiores para J. copaia. Somente V. americana e P. paraenses não apresentaram diferenças significativas no crescimento em altura em relação aos diferentes tamanhos de clareiras. Os valores de crescimento e mortalidade das espécies apresentaram variações em relação aos diferentes tamanhos de clareiras. J. copaia e N. suaveolens apresentaram melhor desempenho, tanto em termos de mortalidade como de crescimento em altura e diâmetro de base nas clareiras médias, todavia essa mortalidade foi elevada em comparação com S. pruriens.<br>Growth and mortality of Sterculia pruriens, Vouacapoua americana, Jacaranda copaia, Protium paraensis, Newtonia suaveolens and Tabebuia serratifolia regarding different gap sizes, had been evaluated in Moju Pará Brazil(2º07'30"S and 2º12'06"S and longitude 48º46'57"W and 48º48'30"W). Nine gaps from forest harvesting had been selected and grouped in small gaps(200m²<Área<400m²), medium-sized gaps(400m²<Área<600m²) and large gaps(Área>600m²). Around the gaps square plots 5m side had been installed, starting in the gap border to forest inside, according to North, South, East and West directions. In these plots seedlings from natural regeneration of tree species were planted. In the each gap center was installed a square plot of 5m side as testimony, where the plantation was repeated. The total mortality average was 46.9%, not showing significant differences between the small gaps (41,05%) and medium-sized gaps (43,86%) but these had differed from the large gaps (54,96%). The small gaps environment was most favorable for the establishment of most species, except for J.copaia and N. suaveolens whose mortalities had been lesser in the medium-sized gaps, even so has not had significant differences in mortality between the different gap sizes. Mortality varied from 14,5%(S. pruriens) in the small gaps to 70,1%(V. americana) in large gaps, but S. pruriens showed lesser mortality in all sizes of gap. All species had shown greater mortality in large gaps, with remark for V. americana (70,1%), J. copaia (69,1%) and N. suaveolens (58,7%). The mortality are between those finding in literature and for its variability they allow to conclude that if it cannot classify accurately the species in ecological groups only on mortality or survival basis. In terms of growth, however, the results are more conclusive, with evident indication of the medium-sized gaps as the best sites for development of the species, with average height growth of 15cm and base diameter growth 0,16cm, followed for the large gaps and for small ones. In general terms, the height growth average was 11,34cm and the base diameter ones was 0,11cm, with maximum values of 32,5cm (height) and 0,24cm (diameter of base) for J. copaia. P. paraensis and V. americana only had not presented significant differences in height growth regarding to different gap sizes, but the values had been a little bigger in the medium-sized gaps. The species growth and mortality, in two years of monitoring, had presented variations in relation to the different gap sizes. J. copaia and N. suaveolens had presented performance better, as much in terms of mortality as of growth in height and diameter of base in the medium-sized gaps, however this mortality was high in comparison with S. pruriens. It must, therefore, be care when taking silvicultural decisions like as thinning, because it was verified differences even though between species considered of the same ecological group
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