87 research outputs found
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Genetic variation in the HLA region is associated with susceptibility to herpes zoster.
- Author
- Armstrong G
- Baldwin E
- Borthwick KM
- Bottinger E
- Burt A
- Carlson CS
- Carrell DS
- Carroll RJ
- Comstock BA
- Crane PK
- Crawford DC
- Crosslin DR
- de Andrade M
- Denny JC
- Doheny KF
- Hanna DS
- Harley JB
- Hayes MG
- Jarvik GP
- Keating B
- Kho A
- Kim DS
- Kuivaniemi H
- Kullo IJ
- Larson EB
- Li R
- McCarty CA
- Mirel DB
- Mukherjee S
- Pacheco J
- Peissig PL
- Pugh E
- Ritchie MD
- Stallings S
- Tromp G
- Underwood JG
- Verma SS
- Publication venue
- eScholarship, University of California
- Publication date
- 01/01/2015
- Field of study
Herpes zoster, commonly referred to as shingles, is caused by the varicella zoster virus (VZV). VZV initially manifests as chicken pox, most commonly in childhood, can remain asymptomatically latent in nerve tissues for many years and often re-emerges as shingles. Although reactivation may be related to immune suppression, aging and female sex, most inter-individual variability in re-emergence risk has not been explained to date. We performed a genome-wide association analyses in 22,981 participants (2280 shingles cases) from the electronic Medical Records and Genomics Network. Using Cox survival and logistic regression, we identified a genomic region in the combined and European ancestry groups that has an age of onset effect reaching genome-wide significance (P>1.0 Ă 10(-8)). This region tags the non-coding gene HCP5 (HLA Complex P5) in the major histocompatibility complex. This gene is an endogenous retrovirus and likely influences viral activity through regulatory functions. Variants in this genetic region are known to be associated with delay in development of AIDS in people infected by HIV. Our study provides further suggestion that this region may have a critical role in viral suppression and could potentially harbor a clinically actionable variant for the shingles vaccine
Magnetic Modulation in Mechanical Alloyed Cr1.4fe0.6o3 Oxide
- Author
- A Aharoni
- AK Bhattarcharya
- C Frandsen
- D New
- D Tobia
- DE Cox
- DN Astrov
- E Sekhar Srinadhu
- GK Williamson
- H Schmid
- HJ Levistein
- IE Dzyaloshinskii
- IJ Chicinas
- J Manjanna
- JE Jaffe
- KF McCarty
- Li Yin-Yuan
- M Nrisimha Murty
- M Vasquez Mansilla
- NA Hill
- P Merchant
- R Abe
- R Hamzaoui
- RD Zysler
- RN Bhowmik
- RN Bhowmik
- RN Bhowmik
- S Di Matteo
- S Music
- SA Chambers
- ST Lin
- WP Osmond
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 02/10/2008
- Field of study
We have synthesized Cr1.4Fe0.6O3 compound through mechanical alloying of
Cr2O3 and Fe2O3 powders and subsequent thermal annealing. The XRD spectrum, SEM
picture and microanalysis of EDAX spectrum have been used to understand the
structural evolution in the alloyed compound. The alloyed samples are matching
to rhombohedral structure with R3C space group. The observation of a modulated
magnetic order confirmed a systematic diffusion of Fe atoms into the Cr sites
of lattice structure. A field induced magnetic behaviour is seen in the field
dependence of magnetization data of the annealed samples. The behaviour is
significantly different from the mechanical alloyed samples. The experimental
results provided the indications of considering the present material as a
potential candidate for opto-electronic applications.Comment: 8 figure
A Combination of Nutriments Improves Mitochondrial Biogenesis and Function in Skeletal Muscle of Type 2 Diabetic GotoâKakizaki Rats
- Author
- A Giancaterini
- AJ Gilde
- BB Lowell
- BJ Regouw
- BN Ames
- BN Ames
- BN Ames
- C Bonnard
- C Luo
- Carl W. Cotma
- CC Liu
- Cheng Luo
- Chuan Tian
- CM Palmeira
- DC Wallace
- DE Estrada
- DE Kelley
- Don Husereau
- G Mingrone
- GM Reaven
- HB Anstall
- I Bogacka
- I Bogacka
- J Liu
- J Zempleni
- Jiankang Liu
- Jiejie Hao
- Jinmin Ren
- JS Pankow
- JX Rong
- K Satoh
- K Shima
- K Tsuji
- KF Petersen
- KF Petersen
- KF Petersen
- KM Humphries
- L Packer
- L Packer
- L Packer
- L Sun
- Lu Yang
- M Kirsch
- M Mensink
- M Yamanaka
- MA Birch-Machin
- MF McCarty
- MM Velykyi
- N Dachicourt
- N Haramaki
- N Produit-Zengaffinen
- P Rosen
- PI Moreira
- R Walczak
- RE McCaman
- RH Unger
- RM Reznick
- S Ghosh
- S Maru
- S Soyal
- U Janssen
- W Shen
- WA Harrison
- Weili Shen
- WJ Lee
- Xuesen Li
- Y Sugiyama
- Publication venue
- Public Library of Science
- Publication date
- 04/06/2008
- Field of study
BACKGROUND: Recent evidence indicates that insulin resistance in skeletal muscle may be related to reduce mitochondrial number and oxidation capacity. However, it is not known whether increasing mitochondrial number and function improves insulin resistance. In the present study, we investigated the effects of a combination of nutrients on insulin resistance and mitochondrial biogenesis/function in skeletal muscle of type 2 diabetic Goto-Kakizaki rats. METHODOLOGY/PRINCIPAL FINDINGS: We demonstrated that defect of glucose and lipid metabolism is associated with low mitochondrial content and reduced mitochondrial enzyme activity in skeletal muscle of the diabetic Goto-Kakizaki rats. The treatment of combination of R-alpha-lipoic acid, acetyl-L-carnitine, nicotinamide, and biotin effectively improved glucose tolerance, decreased the basal insulin secretion and the level of circulating free fatty acid (FFA), and prevented the reduction of mitochondrial biogenesis in skeletal muscle. The nutrients treatment also significantly increased mRNA levels of genes involved in lipid metabolism, including peroxisome proliferator-activated receptor-alpha (Ppar alpha), peroxisome proliferator-activated receptor-delta (Ppar delta), and carnitine palmitoyl transferase-1 (Mcpt-1) and activity of mitochondrial complex I and II in skeletal muscle. All of these effects of mitochondrial nutrients are comparable to that of the antidiabetic drug, pioglitazone. In addition, the treatment with nutrients, unlike pioglitazone, did not cause body weight gain. CONCLUSIONS/SIGNIFICANCE: These data suggest that a combination of mitochondrial targeting nutrients may improve skeletal mitochondrial dysfunction and exert hypoglycemic effects, without causing weight gain
Magnetic and structural properties of pure and Cr-doped haematite: α-Fe2âxCr x O3 (0 †x †1)
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Search for the neutral Higgs bosons of the minimal supersymmetric standard model in pp collisions at root s=7 TeV with the ATLAS detector
- Author
- Aad G
- Abajyan T
- Abbott B
- Abdallah J
- Abdelalim AA
- Abdinov O
- Abi B
- Abolins M
- AbouZeid OS
- Abramowicz H
- Abreu H
- Acharya BS
- Adam ER
- Adamczyk L
- Adams DL
- Addy TN
- Adelman J
- Adomeit S
- Adragna P
- Adye T
- Aesky S
- Agar-Savedra JA
- Agustoni M
- Ahlen SP
- Ahles F
- Ahmad A
- Ahsan M
- Aielli G
- Akesson TPA
- Akimoto G
- Akimov AV
- Alam MA
- Albert J
- Albrand S
- Aleksa M
- Aleksandrov IN
- Alessandria F
- Alexa C
- Alexander G
- Alexandre G
- Alexopoulos T
- Alhroob M
- Aliev M
- Alimonti G
- Alison J
- Allbrooke BMM
- Allison LJ
- Allport PP
- Allwood-Spiers SE
- Almenar CC
- Almond J
- Aloisio A
- Alon R
- Alonso A
- Alonso F
- Altheimer A
- Alviggi MG
- Amako K
- Amelung C
- Ammosov VV
- Amor Dos Santos SP
- Amorim A
- Amoroso S
- Amram N
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders G
- Anderson KJ
- Andreazza A
- Andrei V
- Andrieux M-L
- Anduaga XS
- Angelidakis S
- Anger P
- Angerami A
- Anghinolfi F
- Anh TV
- Anisenkov A
- Anjos N
- Annovi A
- Antonaki A
- Antonelli M
- Antonov A
- Antos J
- Anulli F
- Aoki M
- Aoun S
- Apolle R
- Arabidze G
- Aracena I
- Arai Y
- Aranda CP
- Arce ATH
- Arfaoui S
- Arguin J-F
- Argyropoulos S
- Arik E
- Arik M
- Armbruster AJ
- Arnaez O
- Arnal V
- Artamonov A
- Artoni G
- Arutinov D
- Asai S
- Ask S
- Asman B
- Asquith L
- Assamagan K
- Astbury A
- Atkinson M
- Aubert B
- Auge E
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- Aurousseau M
- Avolio G
- Axen A
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- Azuma Y
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- Bacci C
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- Bachas K
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- Badescu E
- Bagnaia P
- Bai Y
- Bailey DC
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- Baker OK
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- Banerjee P
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- Banfi D
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- Bansal V
- Bansil HS
- Barak L
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- Barberio EL
- Barberis D
- Barbero N
- Bardin DY
- Barillari T
- Barisonzi M
- Barklow T
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- Barnett BM
- Barnett RM
- Barojas CAC
- Baroncelli A
- Barone G
- Barr AJ
- Barreiro F
- Barrera CO
- Bartoldus R
- Barton AE
- Bartsch V
- Basye A
- Bates RL
- Batkova L
- Batley JR
- Battaglia A
- Battistin M
- Bauer F
- Bawa HS
- Beale S
- Beau T
- Beauchemin PH
- Beccherle R
- Bechtle P
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- Becks KH
- Beddall A
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- Bednyakov VA
- Bee CP
- Beemster LJ
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- Bell WH
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- Bobbink GJ
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- Boddy CR
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- Boeriu OEV
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- Bogdanchikov A
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- Boldea V
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- Bomben M
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- Buat Q
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- Butler JM
- Buttar CM
- Butterworth JM
- Buttinger W
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- Castanheira MTD
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- Castillo IS
- Castillo LRF
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- Cataldi C
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- Catmore JR
- Cattai A
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- Cavalcanti TP
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- Chafaq A
- Chakraborty D
- Chalupkova I
- Chan K
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- Chapleau B
- Chapman JD
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- Charlton DG
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- Cheatham S
- Chekanov S
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- Chelkov GA
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- Collaboration ATLAS
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- Coniavitis E
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- Czirr H
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- D'Auria S
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- da Costa JBG
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- della Porta GZ
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- Delsart PA
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- Wijeratne PA
- Wildauer A
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- Winkelmann S
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- Yamada N
- Yamaguchi H
- Yamamoto A
- Yamamoto K
- Yamamoto S
- Yamamura T
- Yamanaka T
- Yamauchi K
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang H
- Yang H
- Yang UK
- Yang Y
- Yang Z
- Yanush S
- Yao L
- Yasu Y
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- Ye S
- Yen AL
- Yilmaz M
- Yoosoofmiya R
- Yorita K
- Yoshida R
- Yoshihara K
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- Young CJ
- Youssef S
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- Yuan L
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- Zaidan R
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- Zanello L
- Zanzi D
- Zaytsev A
- Zeitnitz C
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- Zemla A
- Zenin O
- Zenis T
- Zerwas D
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- Zhao Z
- Zhemchugov A
- Zhong J
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- Zhu CG
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- Zhu J
- Zhu Y
- Zhuang X
- Zhuravlov V
- Zibell A
- Zieminska D
- Zimin NI
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zitoun R
- Zivkovic L
- Zmouchko VV
- Zobernig G
- Zoccoli A
- zur Nedden M
- Zutshi V
- Zwalinski L
- Publication venue
- Publication date
- 01/02/2013
- Field of study
A search for neutral Higgs bosons of the Minimal Supersymmetric Standard Model (MSSM) is reported. The analysis is based on a sample of proton-proton collisions at a centre-of-mass energy of 7TeV recorded with the ATLAS detector at the Large Hadron Collider. The data were recorded in 2011 and correspond to an integrated luminosity of 4.7 fb-1 to 4.8 fb-1. Higgs boson decays into oppositely-charged muon or Ï lepton pairs are considered for final states requiring either the presence or absence of b-jets. No statistically significant excess over the expected background is observed and exclusion limits at the 95% confidence level are derived. The exclusion limits are for the production cross-section of a generic neutral Higgs boson, Ï, as a function of the Higgs boson mass and for h/A/H production in the MSSM as a function of the parameters mA and tan ÎČ in the mhmax scenario for mA in the range of 90GeV to 500 GeV. Copyright CERN
STAT3 Is Activated by JAK2 Independent of Key Oncogenic Driver Mutations in Non-Small Cell Lung Carcinoma
- Author
- A Jarnicki
- A Quintas-Cardama
- A Vultur
- AH Ramos
- AV Jones
- B He
- B Seruga
- BD Looyenga
- BJ Jenkins
- Brendan D. Looyenga
- Chris Kingsley
- Danielle Hutchings
- DJ Gough
- DJ Waugh
- DM Jackman
- E Lucia
- E Rumi
- EB Haura
- Glen J. Weiss
- H Yu
- H Yu
- I Astsaturov
- Irene Cherni
- J Amann
- J Bromberg
- JA Engelman
- JA Engelman
- Jeffrey P. MacKeigan
- JF Bromberg
- JP Pignon
- Juri G. Gelovani
- JV Alvarez
- K Leslie
- KF Chen
- KS McCarty Jr
- L Ding
- LA Gilbert
- M Gadina
- M Hedvat
- MB Meads
- PA Janne
- R Beroukhim
- R Chiarle
- R Kralovics
- R Rosell
- S Veeriah
- S Verstovsek
- SA Forbes
- SP D'Angelo
- SP Gao
- T Mukohara
- YK Yoon
- Z Yao
- Publication venue
- Public Library of Science
- Publication date
- 02/02/2012
- Field of study
Constitutive activation of STAT3 is a common feature in many solid tumors including non-small cell lung carcinoma (NSCLC). While activation of STAT3 is commonly achieved by somatic mutations to JAK2 in hematologic malignancies, similar mutations are not often found in solid tumors. Previous work has instead suggested that STAT3 activation in solid tumors is more commonly induced by hyperactive growth factor receptors or autocrine cytokine signaling. The interplay between STAT3 activation and other well-characterized oncogenic âdriverâ mutations in NSCLC has not been fully characterized, though constitutive STAT3 activation has been proposed to play an important role in resistance to various small-molecule therapies that target these oncogenes. In this study we demonstrate that STAT3 is constitutively activated in human NSCLC samples and in a variety of NSCLC lines independent of activating KRAS or tyrosine kinase mutations. We further show that genetic or pharmacologic inhibition of the gp130/JAK2 signaling pathway disrupts activation of STAT3. Interestingly, treatment of NSCLC cells with the JAK1/2 inhibitor ruxolitinib has no effect on cell proliferation and viability in two-dimensional culture, but inhibits growth in soft agar and xenograft assays. These data demonstrate that JAK2/STAT3 signaling operates independent of known driver mutations in NSCLC and plays critical roles in tumor cell behavior that may not be effectively inhibited by drugs that selectively target these driver mutations
PABPN1 gene therapy for oculopharyngeal muscular dystrophy
- Author
- A Abu-Baker
- A Banerjee
- A Bigot
- A Calado
- A Chartier
- A Chartier
- A Vignaud
- AC Nathwani
- AM Maguire
- B Benoit
- B Brais
- BS Carvalho
- C Kaeppel
- C Li
- C Trollet
- C Zincarelli
- D Bergeron
- DM McCarty
- DR Bisset
- E de Klerk
- F Borel
- F Mingozzi
- FM Tome
- GS Butler-Browne
- J Bennett
- JE Davies
- JE Davies
- JE Davies
- JE Davies
- JE Davies
- JF Lemay
- JP Bouchard
- JP Tavanez
- JP Tavanez
- JR Mendell
- K Mamchaoui
- KF Winklhofer
- L Muniz
- LH Apponi
- LH Apponi
- LM Stanek
- LP Corbeil-Girard
- M Conacci-Sorrell
- M Jenal
- M O'Reilly
- M Riaz
- M Tardieu
- ME Ritchie
- MW Becher
- N Barbezier
- N Kharma
- P Harish
- P Klein
- P Richard
- P Verheesen
- PM Palmer
- R Bengoechea
- R Zhang
- RE MacLaren
- S Millington-Ward
- S Perie
- SM Bresson
- SR Kumar
- T Gidaro
- X Fan
- X Li
- YB Beaulieu
- YP Bao
- Z Wang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2017
- Field of study
International audienceOculopharyngeal muscular dystrophy (OPMD) is an autosomal dominant, late-onset muscle disorder characterized by ptosis, swallowing difficulties, proximal limb weakness and nuclear aggregates in skeletal muscles. OPMD is caused by a trinucleotide repeat expansion in the PABPN1 gene that results in an N-terminal expanded polyalanine tract in polyA-binding protein nuclear 1 (PABPN1). Here we show that the treatment of a mouse model of OPMD with an adeno-associated virus-based gene therapy combining complete knockdown of endogenous PABPN1 and its replacement by a wild-type PABPN1 substantially reduces the amount of insoluble aggregates, decreases muscle fibrosis, reverts muscle strength to the level of healthy muscles and normalizes the muscle transcriptome. The efficacy of the combined treatment is further confirmed in cells derived from OPMD patients. These results pave the way towards a gene replacement approach for OPMD treatment
Profile of men's health in Malaysia: problems and challenges
- Author
- Addis ME
- Baker P
- Banks I
- Cheah PY
- Chirk Jenn Ng
- Chute CG
- Courtenay W
- Courtenay WH
- Dawie Usop AK
- Galdas P
- Galdas PM
- Garraway WM
- Homma Y
- Homma Y
- Kesteloot H
- Khoo EM
- Kraemer S
- Lee SWH
- Li MK
- Lim G
- Low WY
- Macdonald JJ
- Mafauzy M
- Mafauzy M
- Mariappan P
- Mathers CD
- McCarty EJ
- Mohamad WB
- Moreira E
- Nawawi HM
- Nazri SM
- Ng CJ
- Ng CJ
- Ng CJ
- Ng CJ
- Nicolosi A
- Nicolosi A
- Othman NH
- Pu YS
- Quek KF
- Rampal L
- Robertson LM
- Schofield T
- Selvin E
- Seng Fah Tong
- Smith JA
- Tan HM
- Teh GC
- Tikkanen M
- Trueman P
- Volp U
- Wah Yun Low
- White A
- Zainal SB
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
- Author
- Abdel-Aziz AK
- Abdelfatah S
- Abdellatif M
- Abdoli A
- Abel S
- Abeliovich H
- Abildgaard MH
- Abudu YP
- Acevedo-Arozena A
- Adamopoulos IE
- Adeli K
- Adolph TE
- Adornetto A
- Aflaki E
- Agam G
- Agarwal A
- Aggarwal BB
- Agnello M
- Agostinis P
- Agrewala JN
- Agrotis A
- Aguilar PV
- Ahmad ST
- Ahmed ZM
- Ahumada-Castro U
- Aits S
- Aizawa S
- Akkoc Y
- Akoumianaki T
- Akpinar HA
- Al-Abd AM
- Al-Akra L
- Al-Gharaibeh A
- Alaoui-Jamali MA
- Alberti S
- Alcocer-GĂłmez E
- Alessandri C
- Ali M
- Alim Al-Bari MA
- Aliwaini S
- Alizadeh J
- Almacellas E
- Almasan A
- Alonso A
- Alonso GD
- Altan-Bonnet N
- Altieri DC
- Alves da Costa C
- Alves S
- Alzaharna MM
- Amadio M
- Amantini C
- Amaral C
- Ambrosio S
- Amer AO
- Ammanathan V
- An Z
- Andersen SU
- Andrabi SA
- Andrade-Silva M
- Andres AM
- Angelini S
- Ann D
- Anozie UC
- Ansari MY
- Antas P
- Antebi A
- AntĂłn Z
- Anwar T
- Apetoh L
- Apostolova N
- Araki T
- Araki Y
- Arasaki K
- Araya J
- AraĂșjo WL
- Arden C
- Arguelles S
- Arias E
- Arikkath J
- Arimoto H
- Ariosa AR
- Armstrong-James D
- Arnauné-Pelloquin L
- Aroca A
- Arroyo DS
- Arsov I
- Artero R
- Arévalo M-A
- Asaro DML
- Aschner M
- Ashrafizadeh M
- Ashur-Fabian O
- Atanasov AG
- Au AK
- Auberger P
- Auner HW
- Aurelian L
- Autelli R
- Avagliano L
- Aveic S
- Aveleira CA
- Avin-Wittenberg T
- Aydin Y
- Ayton S
- Ayyadevara S
- Azzopardi M
- Baba M
- Backer JM
- Backues SK
- Bae D-H
- Bae O-N
- Bae SH
- Baehrecke EH
- Baek A
- Baek S-H
- Baek SH
- Bagetta G
- Bagniewska-Zadworna A
- Bai H
- Bai J
- Bai X
- Bai Y
- Bairagi N
- Baksi S
- Balazadeh S
- Balbi T
- Baldari CT
- Balduini W
- Ballabio A
- Ballester M
- Balzan R
- Bandopadhyay R
- Banerjee S
- Banerjee S
- Bao Y
- Baptista MS
- Baracca A
- Barbati C
- Bargiela A
- BarilĂ D
- Barlow PG
- Barmada SJ
- Barreiro E
- Barreto GE
- Bartek J
- Bartel B
- Bartolome A
- Barve GR
- Basagoudanavar SH
- Bassham DC
- Bast RC
- Basu A
- Batoko H
- Batten I
- Baulieu EE
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- Waguri S
- Waite KA
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- Wang Q
- Wang QA
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- Wang W
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- Wehman AM
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- WeiergrÀber OH
- Weihl CC
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- Weiskirchen R
- Wells A
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- Weykopf B
- Wheatley SP
- Whitton JL
- Whitworth AJ
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- Wildenberg ME
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- Wilkinson S
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- Williams RSB
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- Winsor NJ
- Witkin SS
- Wodrich H
- Woehlbier U
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- Wu A-G
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- Yakhine-Diop SMS
- Yamaguchi M
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- Yen PM
- Yi C
- Yi-Ren Hong C-WH
- Yin X-M
- Yin Y
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- Ying M
- Ying Z
- Yip CK
- Yiu SPT
- Yoo YH
- Yoshida K
- Yoshii SR
- Yoshimori T
- Yousefi B
- Yu B
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- Zanella I
- Zang QS
- Zanivan S
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- Zaragoza P
- Zarbalis KS
- Zarebkohan A
- Zarrouk A
- Zeitlin SO
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- Zhang H-L
- Zhang J
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- Zhang J-P
- Zhang KYB
- Zhang L
- Zhang L
- Zhang L
- Zhang L
- Zhang LW
- Zhang M
- Zhang P
- Zhang S
- Zhang W
- Zhang X
- Zhang X
- Zhang X
- Zhang X
- Zhang X
- Zhang X-W
- Zhang XD
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Y-D
- Zhang Y-Y
- Zhang Z
- Zhang Z
- Zhang Z
- Zhang Z
- Zhang Z
- Zhang Z
- Zhao H
- Zhao L
- Zhao S
- Zhao T
- Zhao X-F
- Zhao Y
- Zhao Y
- Zhao Y
- Zhao Y
- Zheng G
- Zheng K
- Zheng L
- Zheng S
- Zheng X-L
- Zheng Y
- Zheng Z-G
- Zhivotovsky B
- Zhong Q
- Zhou A
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- Zhou K
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- Zhou X-J
- Zhou Y
- Zhou Y
- Zhou Y
- Zhou Z
- Zhou Z-Y
- Zhu B
- Zhu C
- Zhu G-Q
- Zhu H
- Zhu H
- Zhu H
- Zhu W-G
- Zhu Y
- Zhu Y
- Zhuang H
- Zhuang X
- Zientara-Rytter K
- Zimmermann CM
- Ziviani E
- Zoladek T
- Zong W-X
- Zorov DB
- Zorzano A
- Zou W
- Zou Z
- Zou Z
- Zuryn S
- Zwerschke W
- Ălvarez ĂMC
- Ăvalos Y
- Ănal G
- ĂstĂŒn S
- ÄoliÄ M
- ÄokiÄ J
- Ćœerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
A novel multiplexed immunoassay identifies CEA, IL-8 and prolactin as prospective markers for Dukesâ stages A-D colorectal cancers
- Author
- A Georgakis
- A Khan
- A Litvka
- AA Ellington
- Abidali Mohamedali
- Alamgir Khan
- C Coghlin
- C Rubie
- CE Dukes
- CS Ang
- D Japink
- D Juncker
- D Li
- David Cantor
- DJ Waugh
- E Assarsson
- E Nice
- Edmond J Breen
- Edouard Nice
- Emma Rennel
- FM Speetjens
- G Galizia
- Gabriella Edfeldt
- H Terada
- H Tilg
- Harish R Cheruku
- HJ Hansen
- HJ Nielsen
- I Kirman
- I Ortea
- J Dimberg
- J Liu
- JE Peterson
- JM Bhatavdekar
- JM Bhatavdekar
- JW Choi
- K Rupertus
- KA Heichman
- KF Chin
- KF Chin
- L Malaguarnera
- LM Ahlemann
- M Crittenden
- M Kang
- M Kawamura
- M Loberg
- M Peeters
- M TaghipourFardArdekani
- M Yamada
- Mark S Baker
- Md Golam Muktadir
- MF McCarty
- MJ Duffy
- N Ben-Jonathan
- NK Neradugomma
- P Lea
- P Marianelli
- PF Bray
- PK Baier
- PK Turner
- Q Sun
- RC Langan
- RI Fernando
- S Bunger
- S Malicki
- S Neagu
- Sadia Mahboob
- SB Thorsen
- Seong Beom Ahn
- Shoba Ranganathan
- Simon Fredriksson
- Sock-Hwee Tan
- T Kantola
- U Ward
- Y Ning
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
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