Quark-gluon vertex in general kinematics

The original publication can be found at www.springerlink.com Submitted to Cornell University’s online archive www.arXiv.org in 2007 by Jon-Ivar Skullerud. Post-print sourced from www.arxiv.org.We compute the quark–gluon vertex in quenched lattice QCD in the Landau gauge, using an off-shell mean-field O(a)-improved fermion action. The Dirac-vector part of the vertex is computed for arbitrary kinematics. We find a substantial infrared enhancement of the interaction strength regardless of the kinematics.Ayse Kizilersu, Derek B. Leinweber, Jon-Ivar Skullerud and Anthony G. William

Mitella kiusiana Makino

原著和名: ツクシチャルメルサウ科名: ユキノシタ科 = Saxifragaceae採集地: 熊本県 阿蘇郡 波野村 (肥後 阿蘇郡 波野村)採集日: 1979/5/8採集者: 萩庭丈壽整理番号: JH000596国立科学博物館整理番号: TNS-VS-95059

Association results for chromosome 27.

<p><b>(A)</b> Chromosome 27 with association results colour-coded according to pair-wise LD (r²) with the top two SNPs (index 1 = chr27:745,156 bp and 2 = chr27:7,706,463 bp). <b>(B)</b> Minor allele frequency plot over chromosome 27. <b>(C)</b> Association results for the merged and imputed GWAS and sequence candidate SNPs dataset at the 0.7 Mb peak. The top SNP (chr27:735,281 bp) is located 418 bp upstream of <i>LACRT</i>, SNP position indicated by red arrow. Gene annotations are lifted over from the human genome. <b>(D)</b> Association results for the 7.7 Mb region. The top SNP (chr27:7,706,463 bp) is potentially located in an intron of <i>SLC38A4</i>.</p

Haplotypes in the chromosome 11 candidate region.

<p><b>(A)</b> Phylogenetic tree displaying haplotype relationship of 15 SNPs in the candidate region on chromosome 11. The 51 haplotypes can be formed into three groups based on the tree clusters (separated by dashed lines). <b>(B)</b> Case/control frequencies in the three haplotype groups in the ESS cohort. There is a lower proportion of cases in haplotype group 1 compared to group 3.</p

Top ten GWAS regions defined by linkage disequilibrium (cut-off p<0.1 and r²>0.2).

<p>Top ten GWAS regions defined by linkage disequilibrium (cut-off p<0.1 and r²>0.2).</p

Genome-wide association results.

<p><b>(A)</b> Quantile-quantile plot displaying a lambda of 1.00, indicating no residual inflation. Thin lines indicate 95% CI. SNPs with -log₁₀(p) values > 4 deviates from the expected distribution and are associated with CMT. <b>(B)</b> Manhattan plot displaying the results from the GWAS based on the Swedish ESS Illumina 170K genotypes. Genome-wide significance is reached for one SNP on chromosome 11 (73,290,522 bp) and nominal association is reached for seven SNPs on chromosomes 11 and 27.</p

SNPs associated with CMT.

<p>SNPs associated with CMT.</p

List of significantly associated haplotypes.

<p>List of significantly associated haplotypes.</p

Differentially expressed genes by the risk alleles at 29 Mb and 33 Mb play important role in T-cell immunity.

<p>A. The risk allele at the 29 Mb at homozygous state has a clear cis-regulation effect on the expression levels of <i>TRPC6</i>, <i>KIAA1377</i>, and <i>ANGPTL5</i>, three of the most proximal genes. <i>BIRC3</i>, which is also proximal to the 29 Mb risk locus, had a significant p-value, however the FDR value was slightly above the threshold of 0.05. The risk allele at 29 Mb was also associated with a regulatory effect on genes near the 33 Mb locus and a change in the expression of <i>PIK3R6</i> significantly. B. A large network of molecules that play a major role in activation of T-lymphocyte and other immune cells (IPA category: cell-to-cell signaling and interaction, hematological system development and function). This network includes 15 molecules of which expressions are significantly altered in individuals carrying at least one copy of the shared risk allele at the 33 Mb locus. The outcomes of such expression changes are significantly linked to decrease in T-cell activation.</p

Top 10 differentially expressed genes by the risk haplotype at each locus.

<p>Top 10 differentially expressed genes by the risk haplotype at each locus.</p