Pion Exchange Effects in Elastic Backward Proton-Deuteron Scattering

The elastic backward proton-deuteron scattering is analyzed within a covariant approach based on the Bethe-Salpeter equation with realistic meson-exchange interaction. Contributions of the one-nucleon and one-pion exchange mechanisms to the cross section and polarization observables are investigated in explicit form. Results of numerical calculations for the cross section, tensor analyzing power and spin transfers are presented. The one-pion exchange contribution is essential for describing the spin averaged cross section, while in polarization observables it is found to be less important.Comment: 19 LaTeX pages, including 5 eps figure

Field induced phase transition in the few photon regime

Some features of the field induced phase transition accompanied by the vacuum creation of an electron-positron plasma (EPP) in strong time-dependent electric fields have been discussed in the work [1] in the domain of the tunneling mechanism ($\omega \ll m$, where $\omega$ is the characteristic frequency of the external field and $m$ is the electron mass). In the present contribution the features of the this process will be considered in the few photon domain where $\omega \sim m$. We observe a narrowing of the transient domain of the fast oscillations and, mainly, a considerable growth of the effectiveness of the EPP production. Under these circumstances, we see an increase of the effectiveness of the EPP creation in the particular case of a bifrequent excitation, where both mechanisms (tunneling and few photon) act simultaneously [2,3].Comment: 6 pages, 4 figures, contribution to the Proceedings of the XXIII International Baldin Seminar on "High Energy Physics Problems", Dubna, Russia, September 19-24, 201

Staphylococcal enterotoxin B (SEB) activates TCR- and CD28-mediated inflammatory signals in the absence of MHC class II molecules

The inflammatory activity of staphylococcal enterotoxin B (SEB) relies on its capacity to trigger polyclonal T‐cell activation by binding both T‐cell receptor (TCR) and costimulatory receptor CD28 on T cells and MHC class II and B7 molecules on antigen presenting cells (APC). Previous studies highlighted that SEB may bind TCR and CD28 molecules independently of MHC class II, yet the relative contribution of these interactions to the pro‐inflammatory function of SEB remained unclear. Here, we show that binding to MHC class II is dispensable for the inflammatory activity of SEB, whereas binding to TCR, CD28 and B7 molecules is pivotal, in both human primary T cells and Jurkat T cell lines. In particular, our finding is that binding of SEB to B7 molecules suffices to trigger both TCR‐ and CD28‐mediated inflammatory signalling. We also provide evidence that, by strengthening the interaction between CD28 and B7, SEB favours the recruitment of the TCR into the immunological synapse, thus inducing lethal inflammatory signallin