Pulsed-field gel electrophoresis group and host origin of <i>Salmonella</i> Typhimurium isolates.

<p>Pulsed-field gel electrophoresis group and host origin of <i>Salmonella</i> Typhimurium isolates.</p

Distribution of <i>S.</i> Typhimurium DT40, DT56(v) and DT160 incidents, 1993–2012 in garden birds and humans.

<p>(a) Garden bird data expressed as total number of incidents by region (b) Human data expressed as number of incidents per 100,000 people by region, according to 2001 census data.</p

Number of garden bird incidents versus human incidents (per 100,000 people) with <i>S</i>. Typhimurium infection, 1993–2012.

<p>(a) Garden bird data expressed as total number of incidents by government office region (b) Human data expressed as number of incidents per 100,000 people by government office region, according to 2001 census data.</p

Number of garden bird incidents versus human incidents with <i>S</i>. Typhimurium infection by year, 1993–2012.

<p>Correlations for the individual phage types: DT56v r₁₈ = 0.80, P<0.001; DT160 r₁₈ = 0.59, P = 0.003 and DT40 r₁₈ = 0.39, P = 0.046.</p

Number of garden bird and human incidents caused by <i>S</i>. Typhimurium phage types.

<p>(a) Number of garden bird incidents with <i>S</i>. Typhimurium DT40 (red), DT56(v) (blue) and DT160 (green), (b) Number of human incidents with <i>S</i>. Typhimurium DT40, DT56(v) and DT160 infection by year; 1993–2012.</p

Pulsed-field gel electrophoresis on human and passerine-derived <i>Salmonella</i> Typhimurium DT40 and DT56(v) isolates.

<p>Dendrogram showing the percent similarity between representative patterns from <i>Salmonella</i> Typhimurium DT40 and DT56(v) isolates digested with <i>Xba</i>I restriction enzyme. PFGE band profiles are shown against kb scale. Phage type (DT40 and DT56v) and PFGE groupings (1–12) data are given along with the number of study isolates in each group. PFGE profiles of contemporary <i>S</i>. Typhimurium DT104 isolates from a human and a pig are included for comparison.</p

Seasonal incidence of opportunistic reports in all garden bird mortality 2001–2006.

<p>Hatched bars – winter (Dec-Feb), open bars – spring (March–May), stippled bars – summer (June–Aug) and black bars - autumn (Sept-Nov). Note break in axis indicating an unprecedented level of reporting in autumn 2006.</p

Regional change in greenfinch occurrence in gardens in response to trichomonosis.

<p>Mean reporting rate from GBW of greenfinch, chaffinch and dunnock in spring 2005/06 (filled bars) and 2007 (open bars) in areas of Low, Intermediate and High incidence of trichomonosis incidence (see <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0012215#pone-0012215-g003" target="_blank">Fig. 3</a>). Bars represent 95% confidence limits.</p

Distribution of finch trichomonosis incidents in 2006.

<p>Gardens reporting at least one incident of finch trichomonosis (large red dots) and all other sites (small yellow dots) contributing to the systematic survey. The shading indicates relative incidence of trichomonosis recorded by the opportunistic survey (incidents per thousand households for each county interpolated from county centroids). The heavy lines delineate areas of High, Intermediate and Low incidence, based on the opportunistic survey data.</p

Seasonal variation in greenfinch occurrence in gardens.

<p>(a) Reporting rate for greenfinch in all GBW gardens for the years 1996–2005 (grey lines), 2006 (red) and 2007 (blue). (b) Fitted seasonal pattern of mean peak greenfinch count in 828 GBW gardens with complete counts in 2005. (c) Difference in mean peak count throughout the year between 2005 and 2006 for greenfinch, dashed lines represent 95% confidence limits.</p