4 research outputs found
The Strategies of the Spanish cotton textile companies before the Civil War: the road to longevity
- Author
- Astrachan M.C.
- Benaul J.M.
- Benaul J.M.
- Boyson R.
- Cabana F.
- Cabana F.
- Cabana F.
- Calvo A.
- Casson M.
- Chandler A.
- Chandler A.
- Chapman S.
- Chapman S.D.
- Chapman S.D.
- Colli A.
- Colli A.
- Colli A.
- Daunton M.
- Deu i Baigual E.
- Deu i Baigual E.
- Deu i Baigual E.
- Dorel-Ferré G.
- Dorel-Ferré G.
- F. Javier Fernández-Roca
- Farnie D.
- Fernández P.
- Fernández Pérez P.
- Fernández-Roca J.
- Fernández-Roca J.
- Fernández-Roca J.
- Ferrer L.
- Fitton R-S.
- Handler W.C.
- Harrison J.
- Higgins D.
- Higgins D.
- Law B.R.
- Lazonick W.
- Lazonick W.
- Licini S.
- Lloyd-Jones R.
- Maluquer de Motes J.
- Mass W.
- Nadal J.
- Nadal J.
- Nadal J.
- Nadal J.
- Palafox J.
- Parejo A.
- Prat M.
- Prat M.
- Prat M.
- Prat M.
- Prat M.
- Ribas Mirangels E.
- Romano R.
- Rose M.B.
- Rose M.B.
- Rose M.B.
- Rose M.B.
- Rosés J.
- Rosés J.
- Salini A.
- Scranton P.
- Sharma P.
- Soler R.
- Solà A.
- Sudrià C.
- Sudrià C.
- Sudrià C.
- Sudrià C.
- Tafunell X.
- Tafunell X.
- Tafunell X.
- Tafunell X.
- Toms J.S.
- Toms J.S.
- Toms J.S.
- Toms J.S.
- Toms J.S.
- Toms J.S.
- Torres Villanueva E.
- Westhead P.
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2012
- Field of study
This study, based on family business theories, offers an innovative vision of the Spanish cotton industry. It proves that Spanish cotton companies, just like their European counterparts, implemented a strategy that was consistent with their nature as family businesses and went beyond the economic-institutional frames within which they developed. The article identifies this strategy as `conservative, because its main objectives were longevity and family control and because it was based on a high percentage of own resources, low levels of indebtedness and organic growth, thus sacrificing profitability for the sake of security.Universidad Pablo de OlavidePostprin
EXPRESIÓN DE MOLÉCULAS MICA EN TEJIDO GÁSTRICO TUMORAL Y NO TUMORAL EN PACIENTES CON CÁNCER GÁSTRICO
- Author
- Publication venue
- 'Elsevier BV'
- Publication date
- Field of study
A Comparative Review on Current and Future Drug Targets Against Bacteria & Malaria
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- A.S. Sanket
- Abdel-Sattar E.
- Adam H.J.
- Alker A.P.
- Angeletti S.
- Angerhofer C.K.
- Ariey F.
- Artini M.
- Banerjee R.
- Basco L.K.
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- 'Bentham Science Publishers Ltd.'
- Publication date
- Field of study
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Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale. Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter; identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation; analyses timings and patterns of tumour evolution; describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity; and evaluates a range of more-specialized features of cancer genomes