Traitement chirurgical de la maladie de Parkinson: Indications et limites

Thalamotomy, a surgical lesion of the ventrointermediate nucleus of the thalamus, is a well known surgical treatment of tremor in Parkinson's disease. Over the last years, new surgical therapies had been developed. These therapies, instead of making a lesion in the brain, consist in placing electrodes in specific areas of the brain and to inhibit neuronal function by electrical stimulation. Electrical stimulation of the subthalamic nucleus or of the pallidum are effective to treat motor symptoms of Parkinson's disease. The procedure can be done bilateraly, contrary to thalamotomy. A short overview of the indications and contra-indications of this kind of therapy is given.SCOPUS: re.jinfo:eu-repo/semantics/publishe

Repeated delirium from homatropine eye-drops. A case report

The administration of homatropine eye-drops precipitated several episodes of delirium in a 69-year-old woman. The unusual susceptibility of this patient to the central anticholinergic effect of homatropine is perhaps related to a preexisting mild cognitive defect possibly related to a cholinergic neurotransmitter deficit.SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Prognostic value of early cortical somatosensory evoked potentials after resuscitation from cardiac arrest

Short-latency somatosensory evoked potentials (SEPs) were evaluated in patients after cardiorespiratory arrest to study their pattern of recovery in the acute stage of anoxic-ischaemic coma. Fifty consecutive comatose patients were investigated within 8 h after cardiorespiratory resuscitation. In 30 patients no cortical SEPs were recorded and none of the patients recovered cognition. In 20 patients cortical SEPs were recorded and 5 recovered. The different susceptibility of frontal and parietal cortical structures to anoxia was reflected by the dissociated loss of parietal or frontal potentials in 6 patients. Post-mortem pathology in 15 patients confirmed extensive anoxic-ischaemic damage of cerebral and cerebellar cortex and thalamus in patients without cortical SEPs whereas the histological lesions were restricted to Sommer's sector and Purkinje cells in those with preserved SEPs. SEPs thus reflect the extent of brain damage after cardiorespiratory resuscitation.SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Thoughts about the IFCN recommendations for the recording of short latency somatosensory evoked potentials

SCOPUS: le.jinfo:eu-repo/semantics/publishe

Neck movement speed in cervical dystonia

Clinical scales of patients with cervical dystonia do not rate neck movement velocity. We prospectively measured range of neck movements and movement velocities in 35 consecutive patients with cervical dystonia (CD) and 29 normal controls. Reduction of peak velocities in patients with CD was the most robust abnormality and was correlated to TWSTRS. Coupled movements out of attempted movement plane were increased in the patient group. Movement range was moderately though significantly reduced. We conclude that slowing of voluntary neck movements is a frequent and hitherto unrecognized feature in CD. © 2009 Movement Disorder Society.SCOPUS: ar.jFLWINinfo:eu-repo/semantics/publishe

La maladie de Parkinson: Considérations cliniques et traitements

SCOPUS: cp.jinfo:eu-repo/semantics/publishe

Phrenic nerve conduction time measurement in pulmonary disorders

SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Qu'attendez-vous des potentiels évoqués en réanimation en 1991 ?

Short-latency somatosensory evoked potentials are a useful tool to explore the central nervous system at the levels of the spinal cord, the brainstem and thalamo-cortical structures. Although these potentials are very resistant to the actions of drugs they are very susceptible to anoxia and ischemia, two characteristics that are very useful for monitoring the patient in coma. Several studies deal with the prognostic significance of short-latency somatosensory potentials in coma of various etiologies. In anoxic-ischemic coma after cardiopulmonary arrest their place as a pronostic indicator seems clearly established :some studies indicate that the absence of the cortical potentials within 8 hours after coma onset excludes the recovery of cognitive function. This rule however does not apply to coma regardless of its etiology. The absence of cortical potentials in traumatic coma does not rule out recovery of cognitive functions. Thus clinical and physiopathological conditions have to be considered carefully if somatosensory evoked potentials are to be used for early definition of prognosis. © 1992 arnette S.A.SCOPUS: re.jinfo:eu-repo/semantics/publishe

ETUDE EVOLUTIVE DE LA CONDUCTION SOMESTHESIQUE CENTRALE DANS UN CAS D'AVITAMINOSE B12

We report clinical and neurophysiological data of a 58 year-old man with vitamin B12 deficiency and a 6 years follow-up. The initial clinical disorders did not permit a clear distinction between peripheral and central nervous system disease. Detailed analysis of the somatosensory central conduction time (measured from onset latencies of N11 and N20) showed clear evidence of slowed down conduction at spinal cervical level (reflected by the N11-P14 interval) whereas supraspinal conduction (as shown by the P14-N20 interval) was normal. The patient's condition improved under treatment, and the somatosensory central conduction time was progressively shortened. After 6 years of treatment, the neurological examination anh the somatosensory central conduction interval were normal. However, analysis of the spinal conduction still showed slowed down conduction.SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Evoked potentials in anesthesia: physiological considerations.

SCOPUS: ar.jinfo:eu-repo/semantics/publishe