Keystroke dynamics as a biometric

Modern computer systems rely heavily on methods of authentication and identity verification to protect sensitive data. One of the most robust protective techniques involves adding a layer of biometric analysis to other security mechanisms, as a means of establishing the identity of an individual beyond reasonable doubt. In the search for a biometric technique which is both low-cost and transparent to the end user, researchers have considered analysing the typing patterns of keyboard users to determine their characteristic timing signatures.Previous research into keystroke analysis has either required fixed performance of known keyboard input or relied on artificial tests involving the improvisation of a block of text for analysis. I is proposed that this is insufficient to determine the nature of unconstrained typing in a live computing environment. In an attempt to assess the utility of typing analysis for improving intrusion detection on computer systems, we present the notion of ‘genuinely free text’ (GFT). Through the course of this thesis, we discuss the nature of GFT and attempt to address whether it is feasible to produce a lightweight software platform for monitoring GFT keystroke biometrics, while protecting the privacy of users.The thesis documents in depth the design, development and deployment of the multigraph-based BAKER software platform, a system for collecting statistical GFT data from live environments. This software platform has enabled the collection of an extensive set of keystroke biometric data for a group of participating computer users, the analysis of which we also present here. Several supervised learning techniques were used to demonstrate that the richness of keystroke information gathered from BAKER is indeed sufficient to recommend multigraph keystroke analysis, as a means of augmenting computer security. In addition, we present a discussion of the feasibility of applying data obtained from GFT profiles in circumventing traditional static and free text analysis biometrics

Faith Engagement at School

The ability of teachers to engage and develop the faith journey of students is seen as a central endeavour in faith-based schools. Much has been written in the literature regarding approaches that may be effective in facilitating this process. This study analysed qualitative data collected from 368 Year 5–12 students from one faith-based school in Australia. Students reported on the key areas they believe assist in developing their faith, in addition to aspects they believe could be improved in this regard. A significant difference was found between the attitudes of those who reported not having a faith background and those who did. This chapter builds onto findings reported in the previous chapter

The Relationship between School Climate and Faith Engagement

The study of school climate has gained increased attention over the past 25 years, with research indicating significant relationships between school climate and a number of social, emotional and academic outcomes for students. An aspect of school climate that has seen little attention, however, is the relationship between the development of faith and positive school climate. This study analyses data from 368 students in one faith-based school in Australia, utilising self-reported measures of school climate and faith engagement. A significant relationship is found between these variables, indicating the potential impact that school climate and faith engagement may have on each other. The study discusses the implications of these findings and makes suggestions in relation to the strengthening of these two areas within faith-based schools

Seismic stratigraphy of the Ontong Java Plateau

The Ontong Java Plateau, a large, deep-water carbonate plateau in the western equatorial Pacific, is an ideal location for studying responses of carbonate sedimentation to the effects of changing paleoceanographic conditions. These carbonate responses are often reflected in the physical properties of the sediment, which in turn control the appearance of seismic reflection profiles. Seismic stratigraphy analyses, correlating eight reflector horizons to each drill site, have been conducted in an attempt to map stratigraphic data. Accurate correlation of seismic stratigraphic data to drilling results requires conversion of traveltime to depth in meters. Synthetic seismogram models, using shipboard physical properties data, have been generated in an attempt to provide this correlation. Physical properties, including laboratory-measured and well-log data, were collected from sites drilled during Deep Sea Drilling Project Legs 30 and 89, and Ocean Drilling Program Leg 130, on the top and flank of the Ontong Java Plateau. Laboratory-measured density is corrected to in-situ conditions by accounting for porosity rebound resulting from removal of the sediment from its overburden. The correction of laboratory-measured compressional velocity to in situ appears to be largely a function of increases in elastic moduli (especially shear rigidity) with depth of burial, more than a function of changes in temperature, pressure, or density (porosity rebound). Well-log velocity and density data for the ooze intervals were found to be greatly affected by drilling disturbance; hence, they were disregarded and replaced by lab data for these intervals. Velocity and density data were used to produce synthetic seismograms. Correlation of seismic reflection data with synthetic data, and hence with depth below seafloor, at each drill site shows that a single velocity-depth function exists for sediments on the top and flank of the Ontong Java Plateau. A polynomial fit of this function provides an equation for domain conversion: Depth (mbsf) = 44.49 + 0.800(traveltime[ms]) + 3.308 × 10 4 (traveltime[ms]2 ) Traveltime (ms) = -35.18 + 1.118(depth[mbsf]) - 1.969 × KT* (depth[mbsf]2 ) Seismic reflection profiles down the flank of the plateau undergo three significant changes: (1) a drastic thinning of the sediment column with depth, (2) changes in the echo-character of the profile (development of seismic facies), and (3) loss of continuous, coherent reflections. Sediments on the plateau top were largely deposited by pelagic processes, with little significant postdepositional or syndepositional modification. Sediments on the flank of the plateau are also pelagic, but they have been modified by faulting, erosion, and mass movement. These processes result in disrupted and incoherent reflectors, development of seismic facies, and redistribution of sediment on the flank of the plateau. Seismic stratigraphic analyses have shown that the sediment section decreases in thickness by as much as 65% between water depths of 2000 m water depth (at the top of the plateau) and 4000 m (near the base of the plateau). Thinning is attributed to increasing carbonate dissolution with depth. If this assumption is correct, then changes in the relative thicknesses of seismostratigraphic units at each drill site are indicative of changes in the position of the lysocline and the dissolution gradient between the lysocline and the carbonate compensation depth. We think that a shallow lysocline in the early Miocene caused sediment thinning. A deepening of the lysocline in the late-early Miocene caused relative thickening at each site. Within the middle Miocene, a sharp rise in lysoclinal depth occurs, concurrent with a steepening of the dissolution gradient. These events result in sediment thinning at all four sites. The thicker sections in the late Miocene likely correspond to a deepening of the lysocline, and a subsequent rise in the lysocline again hinders accumulation of sediment in the very late Miocene and Pliocene

Critical Roles for LIGHT and Its Receptors in Generating T Cell-Mediated Immunity during Leishmania donovani Infection

LIGHT (TNFSF14) is a member of the TNF superfamily involved in inflammation and defence against infection. LIGHT signals via two cell-bound receptors; herpes virus entry mediator (HVEM) and lymphotoxin-beta receptor (LTβR). We found that LIGHT is critical for control of hepatic parasite growth in mice with visceral leishmaniasis (VL) caused by infection with the protozoan parasite Leishmania donovani. LIGHT-HVEM signalling is essential for early dendritic cell IL-12/IL-23p40 production, and the generation of IFNγ- and TNF-producing T cells that control hepatic infection. However, we also discovered that LIGHT-LTβR interactions suppress anti-parasitic immunity in the liver in the first 7 days of infection by mechanisms that restrict both CD4+ T cell function and TNF-dependent microbicidal mechanisms. Thus, we have identified distinct roles for LIGHT in infection, and show that manipulation of interactions between LIGHT and its receptors may be used for therapeutic advantage

Restoration of IFNγR Subunit Assembly, IFNγ Signaling and Parasite Clearance in Leishmania donovani Infected Macrophages: Role of Membrane Cholesterol

Despite the presence of significant levels of systemic Interferon gamma (IFNγ), the host protective cytokine, Kala-azar patients display high parasite load with downregulated IFNγ signaling in Leishmania donovani (LD) infected macrophages (LD-MØs); the cause of such aberrant phenomenon is unknown. Here we reveal for the first time the mechanistic basis of impaired IFNγ signaling in parasitized murine macrophages. Our study clearly shows that in LD-MØs IFNγ receptor (IFNγR) expression and their ligand-affinity remained unaltered. The intracellular parasites did not pose any generalized defect in LD-MØs as IL-10 mediated signal transducer and activator of transcription 3 (STAT3) phosphorylation remained unaltered with respect to normal. Previously, we showed that LD-MØs are more fluid than normal MØs due to quenching of membrane cholesterol. The decreased rigidity in LD-MØs was not due to parasite derived lipophosphoglycan (LPG) because purified LPG failed to alter fluidity in normal MØs. IFNγR subunit 1 (IFNγR1) and subunit 2 (IFNγR2) colocalize in raft upon IFNγ stimulation of normal MØs, but this was absent in LD-MØs. Oddly enough, such association of IFNγR1 and IFNγR2 could be restored upon liposomal delivery of cholesterol as evident from the fluorescence resonance energy transfer (FRET) experiment and co-immunoprecipitation studies. Furthermore, liposomal cholesterol treatment together with IFNγ allowed reassociation of signaling assembly (phospho-JAK1, JAK2 and STAT1) in LD-MØs, appropriate signaling, and subsequent parasite killing. This effect was cholesterol specific because cholesterol analogue 4-cholestene-3-one failed to restore the response. The presence of cholesterol binding motifs [(L/V)-X1–5-Y-X1–5-(R/K)] in the transmembrane domain of IFNγR1 was also noted. The interaction of peptides representing this motif of IFNγR1 was studied with cholesterol-liposome and analogue-liposome with difference of two orders of magnitude in respective affinity (KD: 4.27×10−9 M versus 2.69×10−7 M). These observations reinforce the importance of cholesterol in the regulation of function of IFNγR1 proteins. This study clearly demonstrates that during its intracellular life-cycle LD perturbs IFNγR1 and IFNγR2 assembly and subsequent ligand driven signaling by quenching MØ membrane cholesterol

The Relationship between Faith Engagement and School Climate at a Faith Based K-12 School

Over the past two decades the concept of school climate has gained prominence due to increased research linking it to a wide range of important school outcomes for students, such as academic achievement (Berkowitz, Moore, Astor, & Benbenishty, 2016), learning motivation (Marsh, Martin, & Cheng, 2008), school attendance (Sakız, 2017), school satisfaction (Zullig, Huebner, & Patton, 2011), depressive symptoms (Way, Reddy, & Rhodes, 2007) and aggressive behaviour (Petrie, 2014). There is however a dearth of exploration within Christian education, on the relationship between faith engagement/development and positive school climate. While one might assume this relationship exists, it was not identified as a correlate within studies on school climate