81 research outputs found
Characterizations of k-commutative equalities for some outer generalized inverses
[EN] In this paper, we present necessary and sufficient conditions for the k-commutative equality , where X is an outer generalized inverse of the square matrix A. Also, we give new representations for core EP, DMP, and CMP inverses of square matrices as outer inverses with prescribed null space and range. In addition, we characterize the core EP inverse as the solution of a new system of matrix equations.D. E. Ferreyra F. E. Levis Partially supported by a Consejo Nacional de Investigaciones Científicas y Técnicas CONICET s Posdoctoral Research Fellowship, UNRC [grant number PPI 18/C472] and CONICET
[grant number PIP 112-201501-00433CO], respectively. N. Thome Partially supported by Secretaría de Estado de Investigación, Desarrollo e Innovación Ministerio de Economía, Industria y Competitividad of Spain [grant number DGI MTM2013-43678-P and Grant Red de Excelen- cia PMTM2017-90682-REDT]. D. E. Ferreyra and N. Thome Partially supported Universidad Nacional de La Pampa (UNLPam), Facultad de Ingeniería [grant Resol. No 155/14].Ferreyra, DE.; Levis, F.; Thome, N. (2018). Characterizations of k-commutative equalities for some outer generalized inverses. Linear and Multilinear Algebra. 1-16. https://doi.org/10.1080/03081087.2018.1500994S116Baksalary, O. M., & Trenkler, G. (2010). Core inverse of matrices. Linear and Multilinear Algebra, 58(6), 681-697. doi:10.1080/03081080902778222Manjunatha Prasad, K., & Mohana, K. S. (2013). Core–EP inverse. Linear and Multilinear Algebra, 62(6), 792-802. doi:10.1080/03081087.2013.791690Malik, S. B., & Thome, N. (2014). On a new generalized inverse for matrices of an arbitrary index. Applied Mathematics and Computation, 226, 575-580. doi:10.1016/j.amc.2013.10.060Mehdipour, M., & Salemi, A. (2017). On a new generalized inverse of matrices. Linear and Multilinear Algebra, 66(5), 1046-1053. doi:10.1080/03081087.2017.1336200Malik, S. B., Rueda, L., & Thome, N. (2016). The class ofm-EPandm-normal matrices. Linear and Multilinear Algebra, 64(11), 2119-2132. doi:10.1080/03081087.2016.1139037Wang, H. (2016). Core-EP decomposition and its applications. Linear Algebra and its Applications, 508, 289-300. doi:10.1016/j.laa.2016.08.008Wang H, Chen J. Weak group inverse. Available from: http://arxiv.org/abs/1704.08403v1Wei, Y. (1998). A characterization and representation of the generalized inverse A(2)T,S and its applications. Linear Algebra and its Applications, 280(2-3), 87-96. doi:10.1016/s0024-3795(98)00008-1Rakić, D. S., Dinčić, N. Č., & Djordjević, D. S. (2014). Core inverse and core partial order of Hilbert space operators. Applied Mathematics and Computation, 244, 283-302. doi:10.1016/j.amc.2014.06.112Stanimirović, P. S., Katsikis, V. N., & Ma, H. (2016). Representations and properties of theW-Weighted Drazin inverse. Linear and Multilinear Algebra, 65(6), 1080-1096. doi:10.1080/03081087.2016.1228810Ferreyra, D. E., Levis, F. E., & Thome, N. (2017). Revisiting the core EP inverse and its extension to rectangular matrices. Quaestiones Mathematicae, 41(2), 265-281. doi:10.2989/16073606.2017.1377779Deng, C. Y., & Du, H. K. (2009). REPRESENTATIONS OF THE MOORE-PENROSE INVERSE OF 2×2 BLOCK OPERATOR VALUED MATRICES. Journal of the Korean Mathematical Society, 46(6), 1139-1150. doi:10.4134/jkms.2009.46.6.1139Wang, H., & Liu, X. (2014). Characterizations of the core inverse and the core partial ordering. Linear and Multilinear Algebra, 63(9), 1829-1836. doi:10.1080/03081087.2014.97570
Delay in maturation of the submandibular gland in Chagas disease correlates with lower DNA synthesis
K+ channel openers restore verapamil-inhibited lung fluid resolution and transepithelial ion transport
<p>Abstract</p> <p>Background</p> <p>Lung epithelial Na<sup>+ </sup>channels (ENaC) are regulated by cell Ca<sup>2+ </sup>signal, which may contribute to calcium antagonist-induced noncardiogenic lung edema. Although K<sup>+ </sup>channel modulators regulate ENaC activity in normal lungs, the therapeutical relevance and the underlying mechanisms have not been completely explored. We hypothesized that K<sup>+ </sup>channel openers may restore calcium channel blocker-inhibited alveolar fluid clearance (AFC) by up-regulating both apical and basolateral ion transport.</p> <p>Methods</p> <p>Verapamil-induced depression of heterologously expressed human αβγ ENaC in <it>Xenopus </it>oocytes, apical and basolateral ion transport in monolayers of human lung epithelial cells (H441), and <it>in vivo </it>alveolar fluid clearance were measured, respectively, using the two-electrode voltage clamp, Ussing chamber, and BSA protein assays. Ca<sup>2+ </sup>signal in H441 cells was analyzed using Fluo 4AM.</p> <p>Results</p> <p>The rate of <it>in vivo </it>AFC was reduced significantly (40.6 ± 6.3% of control, <it>P </it>< 0.05, n = 12) in mice intratracheally administrated verapamil. K<sub>Ca3.1 </sub>(1-EBIO) and K<sub>ATP </sub>(minoxidil) channel openers significantly recovered AFC. In addition to short-circuit current (Isc) in intact H441 monolayers, both apical and basolateral Isc levels were reduced by verapamil in permeabilized monolayers. Moreover, verapamil significantly altered Ca<sup>2+ </sup>signal evoked by ionomycin in H441 cells. Depletion of cytosolic Ca<sup>2+ </sup>in αβγ ENaC-expressing oocytes completely abolished verapamil-induced inhibition. Intriguingly, K<sub>V </sub>(pyrithione-Na), K <sub>Ca3.1 </sub>(1-EBIO), and K<sub>ATP </sub>(minoxidil) channel openers almost completely restored the verapamil-induced decrease in Isc levels by diversely up-regulating apical and basolateral Na<sup>+ </sup>and K<sup>+ </sup>transport pathways.</p> <p>Conclusions</p> <p>Our observations demonstrate that K<sup>+ </sup>channel openers are capable of rescuing reduced vectorial Na<sup>+ </sup>transport across lung epithelial cells with impaired Ca<sup>2+ </sup>signal.</p
Measurement of the Forward-Backward Asymmetry in the B -> K(*) mu+ mu- Decay and First Observation of the Bs -> phi mu+ mu- Decay
We reconstruct the rare decays , , and in a data sample
corresponding to collected in collisions at
by the CDF II detector at the Fermilab Tevatron
Collider. Using and decays we report the branching ratios. In addition, we report
the measurement of the differential branching ratio and the muon
forward-backward asymmetry in the and decay modes, and the
longitudinal polarization in the decay mode with respect to the squared
dimuon mass. These are consistent with the theoretical prediction from the
standard model, and most recent determinations from other experiments and of
comparable accuracy. We also report the first observation of the {\mathcal{B}}(B^0_s \to
\phi\mu^+\mu^-) = [1.44 \pm 0.33 \pm 0.46] \times 10^{-6}27 \pm 6B^0_s$ decay observed.Comment: 7 pages, 2 figures, 3 tables. Submitted to Phys. Rev. Let
Measurement of the Bottom-Strange Meson Mixing Phase in the Full CDF Data Set
We report a measurement of the bottom-strange meson mixing phase \beta_s
using the time evolution of B0_s -> J/\psi (->\mu+\mu-) \phi (-> K+ K-) decays
in which the quark-flavor content of the bottom-strange meson is identified at
production. This measurement uses the full data set of proton-antiproton
collisions at sqrt(s)= 1.96 TeV collected by the Collider Detector experiment
at the Fermilab Tevatron, corresponding to 9.6 fb-1 of integrated luminosity.
We report confidence regions in the two-dimensional space of \beta_s and the
B0_s decay-width difference \Delta\Gamma_s, and measure \beta_s in [-\pi/2,
-1.51] U [-0.06, 0.30] U [1.26, \pi/2] at the 68% confidence level, in
agreement with the standard model expectation. Assuming the standard model
value of \beta_s, we also determine \Delta\Gamma_s = 0.068 +- 0.026 (stat) +-
0.009 (syst) ps-1 and the mean B0_s lifetime, \tau_s = 1.528 +- 0.019 (stat) +-
0.009 (syst) ps, which are consistent and competitive with determinations by
other experiments.Comment: 8 pages, 2 figures, Phys. Rev. Lett 109, 171802 (2012
Measurements of the properties of Lambda_c(2595), Lambda_c(2625), Sigma_c(2455), and Sigma_c(2520) baryons
We report measurements of the resonance properties of Lambda_c(2595)+ and
Lambda_c(2625)+ baryons in their decays to Lambda_c+ pi+ pi- as well as
Sigma_c(2455)++,0 and Sigma_c(2520)++,0 baryons in their decays to Lambda_c+
pi+/- final states. These measurements are performed using data corresponding
to 5.2/fb of integrated luminosity from ppbar collisions at sqrt(s) = 1.96 TeV,
collected with the CDF II detector at the Fermilab Tevatron. Exploiting the
largest available charmed baryon sample, we measure masses and decay widths
with uncertainties comparable to the world averages for Sigma_c states, and
significantly smaller uncertainties than the world averages for excited
Lambda_c+ states.Comment: added one reference and one table, changed order of figures, 17
pages, 15 figure
Search for a New Heavy Gauge Boson Wprime with Electron + missing ET Event Signature in ppbar collisions at sqrt(s)=1.96 TeV
We present a search for a new heavy charged vector boson decaying
to an electron-neutrino pair in collisions at a center-of-mass
energy of 1.96\unit{TeV}. The data were collected with the CDF II detector
and correspond to an integrated luminosity of 5.3\unit{fb}^{-1}. No
significant excess above the standard model expectation is observed and we set
upper limits on . Assuming standard
model couplings to fermions and the neutrino from the boson decay to
be light, we exclude a boson with mass less than
1.12\unit{TeV/}c^2 at the 95\unit{%} confidence level.Comment: 7 pages, 2 figures Submitted to PR
RhoH Regulates Subcellular Localization of ZAP-70 and Lck in T Cell Receptor Signaling
RhoH is an hematopoietic-specific, GTPase-deficient Rho GTPase that plays a role in T development. We investigated the mechanisms of RhoH function in TCR signaling. We found that the association between Lck and CD3ζ was impaired in RhoH-deficient T cells, due to defective translocation of both Lck and ZAP-70 to the immunological synapse. RhoH with Lck and ZAP-70 localizes in the detergent-soluble membrane fraction where the complex is associated with CD3ζ phosphorylation. To determine if impaired translocation of ZAP-70 was a major determinant of defective T cell development, Rhoh-/- bone marrow cells were transduced with a chimeric myristoylation-tagged ZAP-70. Myr-ZAP-70 transduced cells partially reversed the in vivo defects of RhoH-associated thymic development and TCR signaling. Together, our results suggest that RhoH regulates TCR signaling via recruitment of ZAP-70 and Lck to CD3ζ in the immunological synapse. Thus, we define a new function for a RhoH GTPase as an adaptor molecule in TCR signaling pathway
Mechanisms of T cell organotropism
F.M.M.-B. is supported by the British Heart Foundation, the Medical Research Council of the UK and the Gates Foundation
Search for heavy bottom-like quarks decaying to an electron or muon and jets in collisions at TeV
We report the most sensitive direct search for pair production of
fourth-generation bottom-like chiral quarks () each decaying promptly to
. We search for an excess of events with an electron or muon, at least five
jets (one indentified as due to a or quark) and an imbalance of
transverse momentum using data from collisions collected by the CDF
II detector at Fermilab with an integrated luminosity of 4.8 fb. We
observe events consistent with background expectation and calculate upper
limits on the pair production cross section ( fb for 375 GeV/) and exclude \gevcc at 95%
confidence level.Comment: For submission to PR
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