232 research outputs found
Specific Gene Expression Responses to Parasite Genotypes Reveal Redundancy of Innate Immunity in Vertebrates
- Author
- A Conesa
- A Karvonen
- A Mantovani
- A Mortazavi
- A Whitehead
- A Zaballos
- AE Crowden
- Anika Witten
- AV Koehler
- B Magnadóttir
- B Star
- BP Lazzaro
- C Trapnell
- CM Press
- D Haase
- David Haase
- Erich Bornberg-Bauer
- FC Jones
- G Gibson
- G Rauch
- G Rauch
- G Rauch
- H Kumar
- HW Chen
- J Jokela
- JC De Roode
- Jennifer K. Rieger
- JK Rieger
- KA Smith
- L Mikeš
- M Kalbe
- Martin Kalbe
- MF Flajnik
- Monika Stoll
- O Seppälä
- PGD Feulner
- R Medzhitov
- R Medzhitov
- RM Maizels
- RM Maizels
- S Nish
- Sebastian D. Fugmann
- SF Owen
- SK Whyte
- SK Whyte
- SM Barribeau
- ST Workenhe
- TBH Reusch
- Thorsten B. H. Reusch
- TL Lenz
- Y Benjamini
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 25/09/2014
- Field of study
Vertebrate innate immunity is the first line of defense against an invading pathogen and has long been assumed to be largely unspecific with respect to parasite/pathogen species. However, recent phenotypic evidence suggests that immunogenetic variation, i.e. allelic variability in genes associated with the immune system, results in host-parasite genotype-by-genotype interactions and thus specific innate immune responses. Immunogenetic variation is common in all vertebrate taxa and this reflects an effective immunological function in complex environments. However, the underlying variability in host gene expression patterns as response of innate immunity to within-species genetic diversity of macroparasites in vertebrates is unknown. We hypothesized that intra-specific variation among parasite genotypes must be reflected in host gene expression patterns. Here we used high-throughput RNA-sequencing to examine the effect of parasite genotypes on gene expression patterns of a vertebrate host, the three-spined stickleback (Gasterosteus aculeatus). By infecting naïve fish with distinct trematode genotypes of the species Diplostomum pseudospathaceum we show that gene activity of innate immunity in three-spined sticklebacks depended on the identity of an infecting macroparasite genotype. In addition to a suite of genes indicative for a general response against the trematode we also find parasite-strain specific gene expression, in particular in the complement system genes, despite similar infection rates of single clone treatments. The observed discrepancy between infection rates and gene expression indicates the presence of alternative pathways which execute similar functions. This suggests that the innate immune system can induce redundant responses specific to parasite genotypes
Towards a System Level Understanding of Non-Model Organisms Sampled from the Environment: A Network Biology Approach
- Author
- A Conesa
- A Koehler
- A Pistocchi
- AD Southam
- AI Saeed
- AM Diab
- Amer M. Diab
- AP Davis
- B Bierie
- B Fricke
- B Santiago-Josefat
- B Sun
- BP Lyons
- BP Lyons
- Brett P. Lyons
- Carolynn Mackenzie
- Christian von Mering
- CO Daub
- EG Bligh
- F Falciani
- FJ Warner
- Francesco Falciani
- G Dennis Jr
- G Stentiford
- G Van Aggelen
- GD Stentiford
- Grant D. Stentiford
- H Wu
- H Yoshiji
- HJ Small
- Huifeng Wu
- I Katsiadaki
- Ioanna Katsiadaki
- J Felsenstein
- JA Roling
- JM Herbert
- John B. Taggart
- John M. Herbert
- Joseph K. Abraham
- JR Jonsson
- K Abraham
- K Basso
- K Kiersch
- Katie L. Bartie
- Kevin J. Chipman
- M Fernandez
- M Raymond
- MA Fisher
- Mark R. Viant
- ME Baker
- MF Kirby
- Michael J. Leaver
- MJ Leaver
- MJ Leaver
- MJ Leaver
- N Bluthgen
- N Tijet
- Nil Turan
- Olga Hrydziuszko
- P Shannon
- P Thangavel
- PA Farazi
- PF Larsen
- PF Larsen
- PM Blumberg
- PW Moran
- R Yazawa
- S Golotvin
- S Gotz
- SB Wiseman
- SE Hook
- SG George
- Stephen G. George
- SW Feist
- T Andoh
- T Maass
- TD Williams
- TD Williams
- TD Williams
- Tim D. Williams
- V Trevino
- W Huang da
- WE Johnson
- X Huang
- Y Benjamini
- Y Yamamoto
- Publication venue
- Public Library of Science
- Publication date
- 01/08/2011
- Field of study
The acquisition and analysis of datasets including multi-level omics and physiology from non-model species, sampled from field populations, is a formidable challenge, which so far has prevented the application of systems biology approaches. If successful, these could contribute enormously to improving our understanding of how populations of living organisms adapt to environmental stressors relating to, for example, pollution and climate. Here we describe the first application of a network inference approach integrating transcriptional, metabolic and phenotypic information representative of wild populations of the European flounder fish, sampled at seven estuarine locations in northern Europe with different degrees and profiles of chemical contaminants. We identified network modules, whose activity was predictive of environmental exposure and represented a link between molecular and morphometric indices. These sub-networks represented both known and candidate novel adverse outcome pathways representative of several aspects of human liver pathophysiology such as liver hyperplasia, fibrosis, and hepatocellular carcinoma. At the molecular level these pathways were linked to TNF alpha, TGF beta, PDGF, AGT and VEGF signalling. More generally, this pioneering study has important implications as it can be applied to model molecular mechanisms of compensatory adaptation to a wide range of scenarios in wild populations
Inflammasome-Mediated IL-1β Production in Humans with Cystic Fibrosis
- Author
- A Cantin
- A Churg
- A Di
- A Elizur
- A Piccini
- A Roy-Burman
- A Venkatakrishnan
- A Weber
- Aaron F. Hirschfeld
- AF Hirschfeld
- AJ Weber
- Anthony Tang
- Ashish Sharma
- BP O'Sullivan
- C Juliana
- C Kunsch
- C Sorio
- CA Dinarello
- CJ Blohmke
- CJ Blohmke
- David M. Ojcius
- DR Koehler
- DS Armstrong
- E Osika
- EA Miao
- EA Miao
- EE Smith
- EM Bruscia
- F Martinon
- FG Bauernfeind
- FL van de Veerdonk
- FR Greten
- FS Sutterwala
- H Levy
- J Cogswell
- J Hiscott
- JM Kahlenberg
- JR Ward
- L Agostini
- LE Sander
- M Averna
- M Lamkanfi
- M Meyer
- Mark A. Chilvers
- MG Netea
- MJ Hunter
- MW Konstan
- N Reiniger
- N Vij
- P Del Porto
- P Gasse
- PA Verhoef
- Pascal M. Lavoie
- R Hamutcu
- Roger Jen
- S Kotrange
- S LeibundGut-Landmann
- S Mariathasan
- S Mariathasan
- SE Rzemieniak
- SM Miggin
- Stuart E. Turvey
- T Bonfield
- T Fernandes-Alnemri
- TA Douglas
- TE Machen
- TN Hilliard
- TZ Khan
- V Hornung
- Y Xu
- Y Zheng
- Publication venue
- Public Library of Science
- Publication date
- 23/05/2012
- Field of study
Inflammation and infection are major determinants of disease severity and consequently, the quality of life and outcome for patients with cystic fibrosis (CF). Interleukin-1 beta (IL-1β) is a key inflammatory mediator. Secretion of biologically active IL-1β involves inflammasome-mediated processing. Little is known about the contribution of IL-1β and the inflammasomes in CF inflammatory disease. This study examines inflammasome-mediated IL-1β production in CF bronchial epithelial cell lines and human patients with CF.Bronchial epithelial cell lines were found to produce negligible amounts of basal or stimulated IL-1β compared to hematopoeitic cells and they did not significantly upregulate caspase-1 activity upon inflammasome stimulation. In contrast, peripheral blood mononuclear cells (PBMCs) from both CF and healthy control subjects produced large amounts of IL-1β and strongly upregulated caspase-1 activity upon inflammasome stimulation. PBMCs from CF patients and controls displayed similar levels of caspase-1 activation and IL-1β production when stimulated with inflammasome activators. This IL-1β production was dependent on NF-κB activity and could be enhanced by priming with LPS. Finally, chemical inhibition of CFTR activity in control PBMCs and THP-1 cells did not significantly alter IL-1β or IL-8 production in response to P. aeruginosa.Hematopoeitic cells appear to be the predominant source of inflammasome-induced pro-inflammatory IL-1β in CF. PBMCs derived from CF subjects display preserved inflammasome activation and IL-1β secretion in response to the major CF pathogen Pseudomonas aeruginosa. However, our data do not support the hypothesis that increased IL-1β production in CF subjects is due to an intrinsic increase in NF-κB activity through loss of CFTR function
Eicosanoid Release Is Increased by Membrane Destabilization and CFTR Inhibition in Calu-3 Cells
- Author
- A Bailleux
- A Debbaghi
- A Di
- A Habib
- A Perez
- AG Buckland
- Aleksander Edelman
- AO Hueber
- BC Bastian
- BJ Rosenstein
- BK Rubin
- BP Wallner
- BQ Shen
- C de Coupade
- D Wang
- Diane-Lore Vieu
- Dominik Hartl
- DR Koehler
- E Klapisz
- F Besancon
- F Brouillard
- F Brouillard
- F Brouillard
- F Ratjen
- Florence Borot
- Franck Brouillard
- Grazyna Faure
- H Chap
- H Grassme
- H Yu
- HL Hsieh
- J Ayala-Sanmartin
- J Lipecka
- J Liu
- Janine Fritsch
- Jesus Ayala-Sanmartin
- JF Chmiel
- Julien Colas
- KS Koumanov
- L Touqui
- LA Borthwick
- Lhousseine Touqui
- LK Hughes
- M Bajmoczi
- M Grell
- M La
- M Ollero
- Marc Chanson
- Mario Ollero
- Maryvonne Baudouin-Legros
- MP Kowalski
- MW Konstan
- N Bensalem
- N Cabrera-Poch
- N Vij
- OJ Jupp
- PM Haggie
- R Meli
- R Tirouvanziam
- RW Ganster
- S Medjane
- S Moon
- S Rao
- Sandra Moriceau
- SW Kim
- T Dudez
- T Harder
- T Ma
- T Rhen
- T Wu
- TE Machen
- TZ Khan
- YA Choi
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2009
- Field of study
The antiinflammatory protein annexin-1 (ANXA1) and the adaptor S100A10 (p11), inhibit cytosolic phospholipase A2 (cPLA2α) by direct interaction. Since the latter is responsible for the cleavage of arachidonic acid at membrane phospholipids, all three proteins modulate eicosanoid production. We have previously shown the association of ANXA1 expression with that of CFTR, the multifactorial protein mutated in cystic fibrosis. This could in part account for the abnormal inflammatory status characteristic of this disease. We postulated that CFTR participates in the regulation of eicosanoid release by direct interaction with a complex containing ANXA1, p11 and cPLA2α. We first analyzed by plasmon surface resonance the in vitro binding of CFTR to the three proteins. A significant interaction between p11 and the NBD1 domain of CFTR was found. We observed in Calu-3 cells a rapid and partial redistribution of all four proteins in detergent resistant membranes (DRM) induced by TNF-α. This was concomitant with increased IL-8 synthesis and cPLA2α activation, ultimately resulting in eicosanoid (PGE2 and LTB4) overproduction. DRM destabilizing agent methyl-β-cyclodextrin induced further cPLA2α activation and eicosanoid release, but inhibited IL-8 synthesis. We tested in parallel the effect of short exposure of cells to CFTR inhibitors Inh172 and Gly-101. Both inhibitors induced a rapid increase in eicosanoid production. Longer exposure to Inh172 did not increase further eicosanoid release, but inhibited TNF-α-induced relocalization to DRM. These results show that (i) CFTR may form a complex with cPLA2α and ANXA1 via interaction with p11, (ii) CFTR inhibition and DRM disruption induce eicosanoid synthesis, and (iii) suggest that the putative cPLA2/ANXA1/p11/CFTR complex may participate in the modulation of the TNF-α-induced production of eicosanoids, pointing to the importance of membrane composition and CFTR function in the regulation of inflammation mediator synthesis
Addressing the educational needs of children with Williams syndrome : a rather neglected area of research?
- Author
- A Järvinen
- A Steele
- BA Dunning
- BP Klein-Tasman
- C Reilly
- C Reilly
- CA Morris
- CB Mervis
- CB Mervis
- Department for Education
- DJ Fidler
- DM Riby
- EM Dykens
- G Lindsay
- J Herwegen Van
- JH Brown
- Jo Van Herwegen
- K Burgoyne
- L Riglin
- MA Martens
- Maria Ashworth
- MH Fisher
- N Humphrey
- Olympia Palikara
- P Howlin
- P Howlin
- RM Hodapp
- S Lindsay
- SM Reis
- U Koehler
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 13/04/2018
- Field of study
Williams syndrome (WS) is a rare neurodevelopmental disorder associated with physical health problems, limitations in cognitive abilities and increased risk of mental health difficulties. This profile of complex needs may make it challenging to support children with WS in schools. Surprisingly, in the current international move for inclusion, limited research exists on the educational provision and academic achievements of children with WS, including the non-existing literature on their voices and the perspectives of key stakeholders. This letter calls for additional research on the risk and protective factors associated with the educational outcomes of these children, the perspectives of the children themselves and the development of the evidence-base about the effectiveness of education intervention programs
Maintenance of genome stability by Fanconi anemia proteins
- Author
- A Adamo
- A Aguilera
- A Benitez
- A Chen
- A Folias
- A Haitjema
- A Hira
- A Khanna
- A Lyakhovich
- A Madireddy
- A Smogorzewska
- A Smogorzewska
- A Sobeck
- A Zunino
- AD Auerbach
- AE Newell
- AF Alpi
- AJ Deans
- AJ Deans
- AL Medhurst
- AM Ali
- AR Meetei
- AS Balajee
- AT Wang
- AT Wang
- AZ Al-Minawi
- B Frett
- B Sengerova
- BP Alter
- C Clauson
- C Lachaud
- C MacKay
- CA Strathdee
- CS Tremblay
- D Adachi
- D Bluteau
- D Cortez
- D Gisselsson
- D Lopez-Martinez
- D Sridharan
- DI Kutler
- DR Koehler
- DR Semlow
- DT Long
- DW Brois
- E Ozeri-Galai
- E Renaud
- EA Foley
- EL Virts
- F Langevin
- F Vaz
- F Yuan
- FA Kruyt
- G Iliakis
- G Nalepa
- G Pagano
- G Pagano
- GL Moldovan
- GM Adair
- GP Voulgaridou
- H Chen
- H Dong
- H Wiseman
- I Garcia-Higuera
- ID Hickson
- J Bartkova
- J Du
- J Li
- J Lightfoot
- J Michl
- J Panneerselvam
- J Zhang
- J Zhang
- JA Lefferts
- JJ Ventura
- JP Duxin
- JR Lo Ten Foe
- JT Sczepanski
- JY Park
- K Hanada
- K Hanada
- K Kratz
- K Skourti-Stathaki
- K Skourti-Stathaki
- K Yoshikiyo
- KA Hoadley
- KA Rickman
- KL Chan
- KR Kumaresan
- L Booth
- L Faivre
- L Liang
- L Pikor
- L Qian
- L Zheng
- LA Fisher
- LC Wang
- LW McMahon
- LW McMahon
- LW McMahon
- M Bogliolo
- M Bogliolo
- M Budzowska
- M Castella
- M Ferrer
- M Futaki
- M Larin
- M Raschle
- M Szaumkessel
- M Tischkowitz
- M Valko
- MA Collart
- MC Kottemann
- MF Arlt
- ML Garcia-Rubio
- MR Hodskinson
- MR Saadatzadeh
- MS Cooke
- MW Lensch
- MZ Xie
- N Bennardo
- N Bhagwat
- N Gudimchuk
- N Matsushita
- NB Collins
- NE Mamrak
- NG Howlett
- NS Gavande
- O Bachs
- O Murina
- OI Olopade
- P Verma
- P Zhang
- R Ceccaldi
- RA Boisvert
- RA Schwab
- RD Shereda
- S Jaber
- S Kim
- S Negrini
- S Quentin
- S Ying
- SC Wang
- SJ Park
- SL Sawyer
- SL Thompson
- SM Howard
- T Liu
- TD Halazonetis
- TV Nguyen
- U Kumari
- V Naim
- W Du
- W Du
- W Zhou
- X Renaudin
- X Zhu
- Y Huang
- Y Kim
- Y Su
- YG Yang
- YK Shin
- Z Abdul-Sater
- Z Kais
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Measurement of the cross section for inclusive isolated-photon production in pp collisions at √s=13TeV using the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abreu R
- Abulaiti Y
- Acharya BS
- Adachi S
- Adamczyk L
- Adelman J
- Adersberger M
- Adye T
- Affolder AA
- Afik Y
- Agatonovic-Jovin T
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahlen SP
- Ahmadov F
- Aielli G
- Akatsuka S
- Akerstedt H
- Akesson TPA
- Akilli E
- Akimov AV
- Alberghi GL
- Albert J
- Albicocco P
- Alconada Verzini MJ
- Alderweireldt SC
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexander G
- Alexopoulos T
- Alhroob M
- Ali B
- Aliev M
- Alimonti G
- Alison J
- Alkire SP
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alstaty MI
- Alvarez Piqueras D
- Alviggi MG
- Amadio BT
- Amelung C
- Amidei D
- Amor Dos Santos SP
- Amoroso S
- Amundsen G
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Anderson KJ
- Andreazza A
- Andrei V
- Angelidakis S
- Angelozzi I
- Angerami A
- Anisenkov AV
- Anjos N
- Annovi A
- Antel C
- Antonelli M
- Antonov A
- Antrim DJ
- Anulli F
- Aoki M
- Arabidze G
- Arai Y
- Araque JP
- Araujo Ferraz V
- Araya ST
- Arce ATH
- Ardell RE
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Arik M
- Armbruster AJ
- Armitage LJ
- Arnaez O
- Arnold H
- Arratia M
- Arslan O
- Artamonov A
- Artoni G
- Artz S
- Asai S
- Asbah N
- Ashkenazi A
- Asquith L
- Assamagan K
- Astalos R
- Astigarraga MEP
- Atkinson M
- Atlay NB
- Augsten K
- Avolio G
- Axen B
- Ayoub MK
- Azuelos G
- Baas AE
- Baca MJ
- Bachacou H
- Bachas K
- Backes M
- Bagnaia P
- Bahmani M
- Bahrasemani H
- Baines JT
- Bajic M
- Baker OK
- Bakker PJ
- Baldin EM
- Balek P
- Balli F
- Balunas WK
- Banas E
- Bandyopadhyay A
- Banerjee S
- Bannoura AAE
- Barajas CAC
- Barak L
- Barberio EL
- Barberis D
- Barbero M
- Barillari T
- Barisits M-S
- Barkeloo JT
- Barklow T
- Barlow N
- Barnes SL
- Barnett BM
- Barnett RM
- Barnovska-Blenessy Z
- Baroncelli A
- Barone G
- Barr AJ
- Barranco Navarro L
- Barreiro F
- Bartoldus R
- Barton AE
- Bartos P
- Basalaev A
- Bassalat A
- Bates RL
- Batista SJ
- Batley JR
- Battaglia M
- Bauce M
- Bauer F
- Bawa HS
- Beacham JB
- Beattie MD
- Beau T
- Beauchemin PH
- Bechtle P
- Beck HC
- Beck HP
- Becker K
- Becker M
- Becot C
- Beddall A
- Beddall AJ
- Bednyakov VA
- Bedognetti M
- Bee CP
- Beermann TA
- Begalli M
- Begel M
- Behr JK
- Bell AS
- Bella G
- Bella LA
- Bellagamba L
- Bellerive A
- Bellomo M
- Belotskiy K
- Beltramello O
- Belyaev NL
- Benary O
- Benchekroun D
- Bender M
- Benekos N
- Benhammou Y
- Benitez J
- Benjamin DP
- Benoit M
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Berger N
- Bergsten LJ
- Beringer J
- Berlendis S
- Berlingen JM
- Bernard NR
- Bernardi G
- Bernius C
- Bernlochner FU
- Berry T
- Berta P
- Bertella C
- Bertoli G
- Bertram IA
- Bertsche C
- Besjes GJ
- Bessner M
- Besson N
- Bethani A
- Bethke S
- Betti A
- Bevan AJ
- Beyer J
- Bianchi RM
- Biebel O
- Biedermann D
- Bielski R
- Bierwagen K
- Biesuz NV
- Biglietti M
- Billoud TRV
- Bilokon H
- Bindi M
- Bingul A
- Bini C
- Biondi S
- Bisanz T
- Bittrich C
- Bjergaard DM
- Black JE
- Black KM
- Blair RE
- Blazek T
- Bloch I
- Blocker C
- Blue A
- Blumenschein U
- Blunier D
- Bobbink GJ
- Bobrovnikov VS
- Bocchetta SS
- Bocci A
- Bock C
- Boehler M
- Boeriu OEV
- Boerner D
- Bogavac D
- Bogdanchikov AG
- Bohm C
- Boisvert V
- Bokan P
- Bold T
- Boldyrev AS
- Bolz AE
- Bomben M
- Bona M
- Boonekamp M
- Borisov A
- Borissov G
- Bortfeldt J
- Bortoletto D
- Bortolotto V
- Boscherini D
- Bosman M
- Boudreau J
- Bouhova-Thacker EV
- Boumediene D
- Bourdarios C
- Boutle SK
- Boveia A
- Boyd J
- Boyko IR
- Bozson AJ
- Bracinik J
- Brandt A
- Brandt G
- Brandt O
- Braren F
- Bratzler U
- Brau B
- Brau JE
- Bravo AG
- Brendlinger K
- Brennan AJ
- Brenner L
- Brenner R
- Bressler S
- Bret MC
- Briglin DL
- Bristow TM
- Britton D
- Britzger D
- Brochu FM
- Brock I
- Brock R
- Brooijmans G
- Brooks T
- Brooks WK
- Brosamer J
- Brost E
- Broughton JH
- Bruncko D
- Bruni A
- Bruni G
- Bruni LS
- Bruno S
- Brunt BH
- Bruschi M
- Bruscino N
- Bryant P
- Bryngemark L
- Buanes T
- Buat Q
- Buchholz P
- Buckley AG
- Budagov IA
- Buehrer F
- Bugge MK
- Bulekov O
- Bullock D
- Burch TJ
- Burdin S
- Burgard CD
- Burger AM
- Burghgrave B
- Burka K
- Burke S
- Burmeister I
- Burr JTP
- Buscher D
- Buscher V
- Bussey P
- Butler JM
- Buttar CM
- Butterworth JM
- Butti P
- Buttinger W
- Buzatu A
- Buzykaev AR
- Bylund OB
- Cabrera Urban S
- Caforio D
- Cai H
- Cairo VM
- Cakir IT
- Cakir O
- Calace N
- Calafiura P
- Calandri A
- Calderini G
- Calfayan P
- Callea G
- Caloba LP
- Calvente Lopez S
- Calvet D
- Calvet S
- Calvet TP
- Camarda S
- Camarri P
- Cameron D
- Caminal Armadans R
- Camincher C
- Campana S
- Campanelli M
- Camplani A
- Campoverde A
- Canale V
- Cantero J
- Cao T
- Caprini I
- Caprini M
- Capua M
- Carbone RM
- Cardarelli R
- Cardillo F
- Carli I
- Carli T
- Carlino G
- Carlson BT
- Carminati L
- Carney RMD
- Caron S
- Carquin E
- Carra S
- Carrillo-Montoya GD
- Casadei D
- Casado MP
- Casha AF
- Casolino M
- Casper DW
- Castelijn R
- Castillo Gimenez V
- Castillo IS
- Castillo LRF
- Castro NF
- Catinaccio A
- Catmore JR
- Cattai A
- Caudron J
- Cavaliere V
- Cavallaro E
- Cavalli D
- Cavalli-Sforza M
- Cavasinni V
- Celebi E
- Ceradini F
- Cerda Alberich L
- Cerqueira AS
- Cerri A
- Cerrito L
- Cerutti F
- Cervelli A
- Cetin SA
- Chafaq A
- Chakraborty D
- Chan SK
- Chan WS
- Chan YL
- Chang P
- Chapman JD
- Charlton DG
- Chau CC
- Che S
- Cheatham S
- Chegwidden A
- Chekanov S
- Chekulaev SV
- Chelkov GA
- Chelstowska MA
- Chen C
- Chen C
- Chen H
- Chen J
- Chen S
- Chen S
- Chen X
- Chen Y
- Cheng HC
- Cheng HJ
- Cheplakov A
- Cheremushkina E
- Cheu E
- Cheung K
- Chevalier L
- Chiarella V
- Chiarelli G
- Chiodini G
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- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2017
- Field of study
Inclusive isolated-photon production in pp collisions at a centre-of-mass energy of 13TeVis studied with the ATLAS detector at the LHC using a data set with an integrated luminosity of 3.2fb−1. The cross section is measured as a function of the photon transverse energy above 125GeVin different regions of photon pseudorapidity. Next-to-leading-order perturbative QCD and Monte Carlo event-generator predictions are compared to the cross-section measurements and provide an adequate description of the data
Measurement of the inelastic proton-proton cross section at √s=13 TeV with the ATLAS detector at the LHC
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdallah J
- Abdinov O
- Abeloos B
- Aben R
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
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- Abulaiti Y
- Acharya BS
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- Akerstedt H
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- Kostyukhin VV
- Kotwal A
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- Kourkoumelis C
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- Loew KM
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- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2015
- Field of study
This Letter presents a measurement of the inelastic proton-proton cross section using 60 μb −1 of pp collisions at a center-of-mass energy √s of 13 TeV with the ATLAS detector at the LHC. Inelastic interactions are selected using rings of plastic scintillators in the forward region (2.0710 −6 , where M X is the larger invariant mass of the two hadronic systems separated by the largest rapidity gap in the event. In this ξ range the scintillators are highly efficient. For diffractive events this corresponds to cases where at least one proton dissociates to a system with M X >13 GeV . The measured cross section is compared with a range of theoretical predictions. When extrapolated to the full phase space, a cross section of 78.1±2.9 mb is measured, consistent with the inelastic cross section increasing with center-of-mass energy
Search for High-Mass Resonances Decaying to τν in pp Collisions at √s=13 TeV with the ATLAS Detector
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- Yildiz H Duran
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- Yusuff I
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- Zalieckas J
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- Zemaityte G
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- Zeng Q
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- Zhemchugov A
- Zhou B
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- Zhou M
- Zhou M
- Zhou N
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- Zhu CG
- Zhu H
- Zhu H
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Zinser M
- Ziolkowski M
- Zivkovic L
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zu Theenhausen H Meyer
- zur Nedden M
- Zwalinski L
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2018
- Field of study
A search for high-mass resonances decaying to τν using proton-proton collisions at √s=13 TeV produced by the Large Hadron Collider is presented. Only τ-lepton decays with hadrons in the final state are considered. The data were recorded with the ATLAS detector and correspond to an integrated luminosity of 36.1 fb−1. No statistically significant excess above the standard model expectation is observed; model-independent upper limits are set on the visible τν production cross section. Heavy W′ bosons with masses less than 3.7 TeV in the sequential standard model and masses less than 2.2–3.8 TeV depending on the coupling in the nonuniversal G(221) model are excluded at the 95% credibility level
Search for the direct production of charginos and neutralinos in final states with tau leptons in √s=13 TeV collisions with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abreu R
- Abulaiti Y
- Acharya BS
- Adachi S
- Adamczyk L
- Adelman J
- Adersberger M
- Adye T
- Affolder AA
- Afik Y
- Agatonovic-Jovin T
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahlen SP
- Ahmadov F
- Aielli G
- Akatsuka S
- Akerstedt H
- Akesson TPA
- Akilli E
- Akimov AV
- Albergh GL
- Albert J
- Albicocco P
- Alconada Verzini MJ
- Alderweireldt SC
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexander G
- Alexopoulos T
- Alhroob M
- Ali B
- Aliev M
- Alimonti G
- Alison J
- Alkire SP
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alstaty MI
- Alvarez Piqueras D
- Alviggi MG
- Amadio BT
- Amelung C
- Amidei D
- Amoroso S
- Amundsen G
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Anderson KJ
- Andreazza A
- Andrei V
- Angelidakis S
- Angelozzi I
- Angerami A
- Anisenkov AV
- Anjos N
- Annovi A
- Antel C
- Antonelli M
- Antonov A
- Antrim DJ
- Anulli F
- Aoki M
- Arabidze G
- Arai Y
- Araque JP
- Arce ATH
- Ardell RE
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Arik M
- Armadans R Caminal
- Armbruster AJ
- Armitage LJ
- Arnaez O
- Arnold H
- Arratia M
- Arslan O
- Artamonov A
- Artoni G
- Artz S
- Asai S
- Asbah N
- Ashkenazi A
- Asquith L
- Assamagan K
- Astalos R
- Astigarraga ME Pozo
- Atkinson M
- Atlay NB
- Augsten K
- Avolio G
- Axen B
- Ayoub MK
- Azuelos G
- Baas AE
- Baca MJ
- Bachacou H
- Bachas K
- Backes M
- Bagnaia P
- Bahmani M
- Bahrasemani H
- Baines JT
- Bajic M
- Baker OK
- Baldin EM
- Balek P
- Balli F
- Balunas WK
- Banas E
- Bandyopadhyay A
- Banerjee Sw
- Bannoura AAE
- Barajas CA Chavez
- Barak L
- Barberio EL
- Barberis D
- Barbero M
- Barillari T
- Barisits M-S
- Barkeloo JT
- Barklow T
- Barlow N
- Barnes SL
- Barnett BM
- Barnett RM
- Barnovska-Blenessy Z
- Baroncelli A
- Barone G
- Barr AJ
- Barranco Navarro L
- Barreiro F
- Bartoldus R
- Barton AE
- Bartos P
- Basalaev A
- Bassalat A
- Bates RL
- Batista SJ
- Batley JR
- Battaglia M
- Bauce M
- Bauer F
- Bawa HS
- Beacham JB
- Beattie MD
- Beau T
- Beauchemin PH
- Bechtle P
- Beck HC
- Beck HP
- Becker K
- Becker M
- Becot C
- Beddall A
- Beddall AJ
- Bednyakov VA
- Bedognetti M
- Bee CP
- Beermann TA
- Begalli M
- Begel M
- Behr JK
- Bell AS
- Bella G
- Bella L Aperio
- Bellagamba L
- Bellerive A
- Bellomo M
- Belotskiy K
- Beltramello O
- Belyaev NL
- Benary O
- Benchekroun D
- Bender M
- Bendtz K
- Benekos N
- Benhammou Y
- Benitez J
- Benjamin DP
- Benoit M
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Berger N
- Beringer J
- Berlendis S
- Berlingen J Montejo
- Bernard NR
- Bernardi G
- Bernius C
- Bernlochner FU
- Berry T
- Berta P
- Bertella C
- Bertoli G
- Bertolucci F
- Bertram IA
- Bertsche C
- Bertsche D
- Besjes GJ
- Bessner M
- Besson N
- Bethani A
- Bethke S
- Bevan AJ
- Beyer J
- Bianchi RM
- Biebel O
- Biedermann D
- Bielski R
- Bierwagen K
- Biesuz NV
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- Vetterli MC
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- Villar DI Narrias
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- Yao W-M
- Yap YC
- Yasu Y
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- Yeletskikh I
- Yigitbasi E
- Yildirim E
- Yildiz H Duran
- Yorita K
- Yoshihara K
- Young C
- Young CJS
- Yu J
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- Yuen SPY
- Yusuff I
- Zabinski B
- Zacharis G
- Zaidan R
- Zaitsev AM
- Zakharchuk N
- Zalieckas J
- Zaman A
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- Zanzi D
- Zeitnitz C
- Zemaityte G
- Zemla A
- Zeng JC
- Zeng Q
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- Zhemchugov A
- Zhou B
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- Zhuang X
- Zhukov K
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann C
- Zimmermann S
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- Zinser M
- Ziolkowski M
- Zivkovic L
- Zobernig G
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- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2017
- Field of study
A search for the direct production of charginos and neutralinos in final states with at least two hadronically decaying tau leptons is presented. The analysis uses a dataset of pp collisions corresponding to an integrated luminosity of 36.1 fb−1, recorded with the ATLAS detector at the Large Hadron Collider at a centre-of-mass energy of 13TeV.Nosignificant deviation from the expected Standard Model background is observed. Limits are derived in scenarios of ˜χ+1 ˜χ−1 pair production and of ˜χ±1 ˜χ02 and ˜χ+1 ˜χ−1 production in simplified models where the neutralinos and charginos decay solely via intermediate left-handed staus and tau sneutrinos, and the mass of the ˜ τL state is set to be halfway between the masses of the ˜χ±1 and the ˜χ01. Chargino masses up to 630 GeV are excluded at 95% confidence level in the scenario of direct production of ˜χ+1 ˜χ−1 for a massless ˜χ01. Common ˜χ±1 and ˜χ02 masses up to 760 GeV are excluded in the case of production of ˜χ±1 ˜χ02 and ˜χ+1 ˜χ−1 assuming a massless ˜χ01. Exclusion limits for additional benchmark scenarios with large and small mass-splitting between the ˜χ±1 and the ˜χ01 are also studied by varying the ˜ τL mass between the masses of the ˜χ±1 and the ˜χ01
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