Tree-width of hypergraphs and surface duality

In Graph Minors III, Robertson and Seymour write: "It seems that the tree-width of a planar graph and the tree-width of its geometric dual are approximately equal - indeed, we have convinced ourselves that they differ by at most one". They never gave a proof of this. In this paper, we prove a generalisation of this statement to embedding of hypergraphs on general surfaces, and we prove that our bound is tight

Tree-width of hypergraphs and surface duality

In Graph Minor III, Robertson and Seymour conjecture that the tree-width of a planar graph and that of its dual differ by at most one. We prove that given a hypergraph H on a surface of Euler genus k, the tree-width of H^* is at most the maximum of tw(H) + 1 + k and the maximum size of a hyperedge of H^*

Diversities and the Geometry of Hypergraphs

The embedding of finite metrics in $\ell_1$ has become a fundamental tool for both combinatorial optimization and large-scale data analysis. One important application is to network flow problems in which there is close relation between max-flow min-cut theorems and the minimal distortion embeddings of metrics into $\ell_1$. Here we show that this theory can be generalized considerably to encompass Steiner tree packing problems in both graphs and hypergraphs. Instead of the theory of $\ell_1$ metrics and minimal distortion embeddings, the parallel is the theory of diversities recently introduced by Bryant and Tupper, and the corresponding theory of $\ell_1$ diversities and embeddings which we develop here.Comment: 19 pages, no figures. This version: further small correction

Branchwidth is (1,g)-self-dual

A graph parameter is self-dual in some class of graphs embeddable in some surface if its value does not change in the dual graph by more than a constant factor. We prove that the branchwidth of connected hypergraphs without bridges and loops that are embeddable in some surface of Euler genus at most g is an (1,g)-self-dual parameter. This is the first proof that branchwidth is an additively self-dual width parameter.Comment: 10 page

A Fixed Parameter Tractable Approximation Scheme for the Optimal Cut Graph of a Surface

Given a graph $G$ cellularly embedded on a surface $\Sigma$ of genus $g$, a cut graph is a subgraph of $G$ such that cutting $\Sigma$ along $G$ yields a topological disk. We provide a fixed parameter tractable approximation scheme for the problem of computing the shortest cut graph, that is, for any $\varepsilon >0$, we show how to compute a $(1+ \varepsilon)$ approximation of the shortest cut graph in time $f(\varepsilon, g)n^3$. Our techniques first rely on the computation of a spanner for the problem using the technique of brick decompositions, to reduce the problem to the case of bounded tree-width. Then, to solve the bounded tree-width case, we introduce a variant of the surface-cut decomposition of Ru\'e, Sau and Thilikos, which may be of independent interest

European Journal of Combinatorics Index, Volume 27

BACKGROUND: Diabetes is an inflammatory condition associated with iron abnormalities and increased oxidative damage. We aimed to investigate how diabetes affects the interrelationships between these pathogenic mechanisms. METHODS: Glycaemic control, serum iron, proteins involved in iron homeostasis, global antioxidant capacity and levels of antioxidants and peroxidation products were measured in 39 type 1 and 67 type 2 diabetic patients and 100 control subjects. RESULTS: Although serum iron was lower in diabetes, serum ferritin was elevated in type 2 diabetes (p = 0.02). This increase was not related to inflammation (C-reactive protein) but inversely correlated with soluble transferrin receptors (r = - 0.38, p = 0.002). Haptoglobin was higher in both type 1 and type 2 diabetes (p &lt; 0.001) and haemopexin was higher in type 2 diabetes (p &lt; 0.001). The relation between C-reactive protein and haemopexin was lost in type 2 diabetes (r = 0.15, p = 0.27 vs r = 0.63, p &lt; 0.001 in type 1 diabetes and r = 0.36, p = 0.001 in controls). Haemopexin levels were independently determined by triacylglycerol (R(2) = 0.43) and the diabetic state (R(2) = 0.13). Regarding oxidative stress status, lower antioxidant concentrations were found for retinol and uric acid in type 1 diabetes, alpha-tocopherol and ascorbate in type 2 diabetes and protein thiols in both types. These decreases were partially explained by metabolic-, inflammatory- and iron alterations. An additional independent effect of the diabetic state on the oxidative stress status could be identified (R(2) = 0.5-0.14). CONCLUSIONS: Circulating proteins, body iron stores, inflammation, oxidative stress and their interrelationships are abnormal in patients with diabetes and differ between type 1 and type 2 diabetes</p