Threshold for the Outbreak of Cascading Failures in Degree-degree Uncorrelated Networks

In complex networks, the failure of one or very few nodes may cause cascading failures. When this dynamical process stops in steady state, the size of the giant component formed by remaining un-failed nodes can be used to measure the severity of cascading failures, which is critically important for estimating the robustness of networks. In this paper, we provide a cascade of overload failure model with local load sharing mechanism, and then explore the threshold of node capacity when the large-scale cascading failures happen and un-failed nodes in steady state cannot connect to each other to form a large connected sub-network. We get the theoretical derivation of this threshold in degree-degree uncorrelated networks, and validate the effectiveness of this method in simulation. This threshold provide us a guidance to improve the network robustness under the premise of limited capacity resource when creating a network and assigning load. Therefore, this threshold is useful and important to analyze the robustness of networks.Comment: 11 pages, 4 figure

Impact of Community Structure on Cascades

The threshold model is widely used to study the propagation of opinions and technologies in social networks. In this model, individuals adopt the new behavior based on how many neighbors have already chosen it. Specifically, we consider the permanent adoption model where individuals that have adopted the new behavior cannot change their state. We study cascades under the threshold model on sparse random graphs with community structure to see whether the existence of communities affects the number of individuals who finally adopt the new behavior. When seeding a small number of agents with the new behavior, the community structure has little effect on the final proportion of people that adopt it, i.e., the contagion threshold is the same as if there were just one community. On the other hand, seeding a fraction of the population with the new behavior has a significant impact on the cascade with the optimal seeding strategy depending on how strongly the communities are connected. In particular, when the communities are strongly connected, seeding in one community outperforms the symmetric seeding strategy that seeds equally in all communities. We also investigate the problem of optimum seeding given a budget constraint, and propose a gradient-based heuristic seeding strategy. Our algorithm, numerically, dispels commonly held beliefs in the literature that suggest the best seeding strategy is to seed over the nodes with the highest number of neighbors.Comment: Version to be published to EC 201

Analysis of complex contagions in random multiplex networks

We study the diffusion of influence in random multiplex networks where links can be of $r$ different types, and for a given content (e.g., rumor, product, political view), each link type is associated with a content dependent parameter $c_i$ in $[0,\infty]$ that measures the relative bias type-$i$ links have in spreading this content. In this setting, we propose a linear threshold model of contagion where nodes switch state if their "perceived" proportion of active neighbors exceeds a threshold \tau. Namely, a node connected to $m_i$ active neighbors and $k_i-m_i$ inactive neighbors via type-$i$ links will turn active if $\sum{c_i m_i}/\sum{c_i k_i}$ exceeds its threshold \tau. Under this model, we obtain the condition, probability and expected size of global spreading events. Our results extend the existing work on complex contagions in several directions by i) providing solutions for coupled random networks whose vertices are neither identical nor disjoint, (ii) highlighting the effect of content on the dynamics of complex contagions, and (iii) showing that content-dependent propagation over a multiplex network leads to a subtle relation between the giant vulnerable component of the graph and the global cascade condition that is not seen in the existing models in the literature.Comment: Revised 06/08/12. 11 Pages, 3 figure

Modeling self-sustained activity cascades in socio-technical networks

The ability to understand and eventually predict the emergence of information and activation cascades in social networks is core to complex socio-technical systems research. However, the complexity of social interactions makes this a challenging enterprise. Previous works on cascade models assume that the emergence of this collective phenomenon is related to the activity observed in the local neighborhood of individuals, but do not consider what determines the willingness to spread information in a time-varying process. Here we present a mechanistic model that accounts for the temporal evolution of the individual state in a simplified setup. We model the activity of the individuals as a complex network of interacting integrate-and-fire oscillators. The model reproduces the statistical characteristics of the cascades in real systems, and provides a framework to study time-evolution of cascades in a state-dependent activity scenario.Comment: 5 pages, 3 figure

Virus Propagation in Multiple Profile Networks

Suppose we have a virus or one competing idea/product that propagates over a multiple profile (e.g., social) network. Can we predict what proportion of the network will actually get "infected" (e.g., spread the idea or buy the competing product), when the nodes of the network appear to have different sensitivity based on their profile? For example, if there are two profiles $\mathcal{A}$ and $\mathcal{B}$ in a network and the nodes of profile $\mathcal{A}$ and profile $\mathcal{B}$ are susceptible to a highly spreading virus with probabilities $\beta_{\mathcal{A}}$ and $\beta_{\mathcal{B}}$ respectively, what percentage of both profiles will actually get infected from the virus at the end? To reverse the question, what are the necessary conditions so that a predefined percentage of the network is infected? We assume that nodes of different profiles can infect one another and we prove that under realistic conditions, apart from the weak profile (great sensitivity), the stronger profile (low sensitivity) will get infected as well. First, we focus on cliques with the goal to provide exact theoretical results as well as to get some intuition as to how a virus affects such a multiple profile network. Then, we move to the theoretical analysis of arbitrary networks. We provide bounds on certain properties of the network based on the probabilities of infection of each node in it when it reaches the steady state. Finally, we provide extensive experimental results that verify our theoretical results and at the same time provide more insight on the problem