Path-Based Epidemic Spreading in Networks

Conventional epidemic models assume omnidirectional contact-based infection. This strongly associates the epidemic spreading process with node degrees. The role of the infection transmission medium is often neglected. In real-world networks, however, the infectious agent as the physical contagion medium usually flows from one node to another via specific directed routes ( path-based infection). Here, we use continuous-time Markov chain analysis to model the influence of the infectious agent and routing paths on the spreading behavior by taking into account the state transitions of each node individually, rather than the mean aggregated behavior of all nodes. By applying a mean field approximation, the analysis complexity of the path-based infection mechanics is reduced from exponential to polynomial. We show that the structure of the topology plays a secondary role in determining the size of the epidemic. Instead, it is the routing algorithm and traffic intensity that determine the survivability and the steady-state of the epidemic. We define an infection characterization matrix that encodes both the routing and the traffic information. Based on this, we derive the critical path-based epidemic threshold below which the epidemic will die off, as well as conditional bounds of this threshold which network operators may use to promote/suppress path-based spreading in their networks. Finally, besides artificially generated random and scale-free graphs, we also use real-world networks and traffic, as case studies, in order to compare the behaviors of contact- and path-based epidemics. Our results further corroborate the recent empirical observations that epidemics in communication networks are highly persistent

Interplay of network dynamics and ties heterogeneity on spreading dynamics

The structure of a network dramatically affects the spreading phenomena unfolding upon it. The contact distribution of the nodes has long been recognized as the key ingredient in influencing the outbreak events. However, limited knowledge is currently available on the role of the weight of the edges on the persistence of a pathogen. At the same time, recent works showed a strong influence of temporal network dynamics on disease spreading. In this work we provide an analytical understanding, corroborated by numerical simulations, about the conditions for infected stable state in weighted networks. In particular, we reveal the role of heterogeneity of edge weights and of the dynamic assignment of weights on the ties in the network in driving the spread of the epidemic. In this context we show that when weights are dynamically assigned to ties in the network an heterogeneous distribution is able to hamper the diffusion of the disease, contrary to what happens when weights are fixed in time.Comment: 10 pages, 10 figure

Simulation analysis on flight delay propagation under different network configurations

This paper investigates flight delay propagation in air transportation networks (ATNs) by considering both network structures and airport operation performance. An airport susceptible-infected-recovered (ASIR) model is established based on the mechanism of epidemic spreading, where the focus is on the impact of the infection rate in order to properly map and understand the probability of delay propagation. Different network configurations are abstracted under complex network theory, in which the ASIR model can be simulated upon. The simulation results show that the original airport traffic, airport connection and the level of airport turnaround services play important roles in influencing delay propagation in different airports. In addition, changes of network structure such as the emerging of secondary hubs can also influence the delay propagation

Epidemic processes in complex networks

In recent years the research community has accumulated overwhelming evidence for the emergence of complex and heterogeneous connectivity patterns in a wide range of biological and sociotechnical systems. The complex properties of real-world networks have a profound impact on the behavior of equilibrium and nonequilibrium phenomena occurring in various systems, and the study of epidemic spreading is central to our understanding of the unfolding of dynamical processes in complex networks. The theoretical analysis of epidemic spreading in heterogeneous networks requires the development of novel analytical frameworks, and it has produced results of conceptual and practical relevance. A coherent and comprehensive review of the vast research activity concerning epidemic processes is presented, detailing the successful theoretical approaches as well as making their limits and assumptions clear. Physicists, mathematicians, epidemiologists, computer, and social scientists share a common interest in studying epidemic spreading and rely on similar models for the description of the diffusion of pathogens, knowledge, and innovation. For this reason, while focusing on the main results and the paradigmatic models in infectious disease modeling, the major results concerning generalized social contagion processes are also presented. Finally, the research activity at the forefront in the study of epidemic spreading in coevolving, coupled, and time-varying networks is reported.Comment: 62 pages, 15 figures, final versio

Flight delay propagation analysis based on the mechanism of the susceptible-infected-susceptible model

This paper investigates flight delay propagation in the air transport networks. An integrate flight-based susceptible-infected-susceptible (FSIS) model is generated using the mechanism of epidemic spreading. Furthermore, the propagation probability in the FSIS model is analyzed through the regression model and later applied to China Easter Airline. The results show that propagation probability varies from different routes, which related to the flight frequency of airports, route distances, scheduled buffer times, and propagated delay times, and the FSIS model can efficiently reveal the process of flight delay propagation, and evaluate the number of delayed flights

Path-Based Epidemic Spreading in Networks

Conventional epidemic models assume omni-directional contact-based infection. This strongly associates the epidemic spreading process with node degrees. The role of the infection transmission medium is often neglected. In real-world networks, however, the infectious agent as the physical contagion medium usually flows from one node to another via specific directed routes (path-based infection). Here, we use continuous-time Markov chain analysis to model the influence of the infectious agent and routing paths on the spreading behavior by taking into account the state transitions of each node individually, rather than the mean aggregated behavior of all nodes. By applying a mean field approximation, the analysis complexity of the path-based infection mechanics is reduced from exponential to polynomial. We show that the structure of the topology plays a secondary role in determining the size of the epidemic. Instead, it is the routing algorithm and traffic intensity that determine the survivability and the steady-state of the epidemic. We define an infection characterization matrix that encodes both the routing and the traffic information. Based on this, we derive the critical path-based epidemic threshold below which the epidemic will die off, as well as conditional bounds of this threshold which network operators may use to promote/suppress path-based spreading in their networks. Finally, besides artificially generated random and scale-free graphs, we also use real-world networks and traffic, as case studies, in order to compare the behaviors of contact- and path-based epidemics. Our results further corroborate the recent empirical observations that epidemics in communication networks are highly persistent