On the behavior of threshold models over finite networks

We study a model for cascade effects over finite networks based on a deterministic binary linear threshold model. Our starting point is a networked coordination game where each agent's payoff is the sum of the payoffs coming from pairwise interaction with each of the neighbors. We first establish that the best response dynamics in this networked game is equivalent to the linear threshold dynamics with heterogeneous thresholds over the agents. While the previous literature has studied such linear threshold models under the assumption that each agent may change actions at most once, a study of best response dynamics in such networked games necessitates an analysis that allows for multiple switches in actions. In this paper, we develop such an analysis. We establish that agent behavior cycles among different actions in the limit, we characterize the length of such limit cycles, and reveal bounds on the time steps required to reach them. We finally propose a measure of network resilience that captures the nature of the involved dynamics. We prove bounds and investigate the resilience of different network structures under this measure.Irwin Mark Jacobs and Joan Klein Jacobs Presidential FellowshipSiebel ScholarshipUnited States. Air Force Office of Scientific Research (Grant FA9550-09-1-0420)United States. Army Research Office (Grant W911NF-09-1-0556

Outage Capacity and Optimal Transmission for Dying Channels

In wireless networks, communication links may be subject to random fatal impacts: for example, sensor networks under sudden power losses or cognitive radio networks with unpredictable primary user spectrum occupancy. Under such circumstances, it is critical to quantify how fast and reliably the information can be collected over attacked links. For a single point-to-point channel subject to a random attack, named as a \emph{dying channel}, we model it as a block-fading (BF) channel with a finite and random delay constraint. First, we define the outage capacity as the performance measure, followed by studying the optimal coding length $K$ such that the outage probability is minimized when uniform power allocation is assumed. For a given rate target and a coding length $K$, we then minimize the outage probability over the power allocation vector \mv{P}_{K}, and show that this optimization problem can be cast into a convex optimization problem under some conditions. The optimal solutions for several special cases are discussed. Furthermore, we extend the single point-to-point dying channel result to the parallel multi-channel case where each sub-channel is a dying channel, and investigate the corresponding asymptotic behavior of the overall outage probability with two different attack models: the independent-attack case and the $m$-dependent-attack case. It can be shown that the overall outage probability diminishes to zero for both cases as the number of sub-channels increases if the \emph{rate per unit cost} is less than a certain threshold. The outage exponents are also studied to reveal how fast the outage probability improves over the number of sub-channels.Comment: 31 pages, 9 figures, submitted to IEEE Transactions on Information Theor

Griffiths phases and localization in hierarchical modular networks

We study variants of hierarchical modular network models suggested by Kaiser and Hilgetag [Frontiers in Neuroinformatics, 4 (2010) 8] to model functional brain connectivity, using extensive simulations and quenched mean-field theory (QMF), focusing on structures with a connection probability that decays exponentially with the level index. Such networks can be embedded in two-dimensional Euclidean space. We explore the dynamic behavior of the contact process (CP) and threshold models on networks of this kind, including hierarchical trees. While in the small-world networks originally proposed to model brain connectivity, the topological heterogeneities are not strong enough to induce deviations from mean-field behavior, we show that a Griffiths phase can emerge under reduced connection probabilities, approaching the percolation threshold. In this case the topological dimension of the networks is finite, and extended regions of bursty, power-law dynamics are observed. Localization in the steady state is also shown via QMF. We investigate the effects of link asymmetry and coupling disorder, and show that localization can occur even in small-world networks with high connectivity in case of link disorder.Comment: 18 pages, 20 figures, accepted version in Scientific Report

Thresholds for epidemic spreading in networks

We study the threshold of epidemic models in quenched networks with degree distribution given by a power-law. For the susceptible-infected-susceptible (SIS) model the activity threshold lambda_c vanishes in the large size limit on any network whose maximum degree k_max diverges with the system size, at odds with heterogeneous mean-field (HMF) theory. The vanishing of the threshold has not to do with the scale-free nature of the connectivity pattern and is instead originated by the largest hub in the system being active for any spreading rate lambda>1/sqrt{k_max} and playing the role of a self-sustained source that spreads the infection to the rest of the system. The susceptible-infected-removed (SIR) model displays instead agreement with HMF theory and a finite threshold for scale-rich networks. We conjecture that on quenched scale-rich networks the threshold of generic epidemic models is vanishing or finite depending on the presence or absence of a steady state.Comment: 5 pages, 4 figure

Collective versus hub activation of epidemic phases on networks

We consider a general criterion to discern the nature of the threshold in epidemic models on scale-free (SF) networks. Comparing the epidemic lifespan of the nodes with largest degrees with the infection time between them, we propose a general dual scenario, in which the epidemic transition is either ruled by a hub activation process, leading to a null threshold in the thermodynamic limit, or given by a collective activation process, corresponding to a standard phase transition with a finite threshold. We validate the proposed criterion applying it to different epidemic models, with waning immunity or heterogeneous infection rates in both synthetic and real SF networks. In particular, a waning immunity, irrespective of its strength, leads to collective activation with finite threshold in scale-free networks with large exponent, at odds with canonical theoretical approaches.Comment: Revised version accepted for publication in PR

Disease Spreading in Structured Scale-Free Networks

We study the spreading of a disease on top of structured scale-free networks recently introduced. By means of numerical simulations we analyze the SIS and the SIR models. Our results show that when the connectivity fluctuations of the network are unbounded whether the epidemic threshold exists strongly depends on the initial density of infected individuals and the type of epidemiological model considered. Analytical arguments are provided in order to account for the observed behavior. We conclude that the peculiar topological features of this network and the absence of small-world properties determine the dynamics of epidemic spreading.Comment: 7 pages, 6 figures. EPJ styl

Epidemic processes in complex networks

In recent years the research community has accumulated overwhelming evidence for the emergence of complex and heterogeneous connectivity patterns in a wide range of biological and sociotechnical systems. The complex properties of real-world networks have a profound impact on the behavior of equilibrium and nonequilibrium phenomena occurring in various systems, and the study of epidemic spreading is central to our understanding of the unfolding of dynamical processes in complex networks. The theoretical analysis of epidemic spreading in heterogeneous networks requires the development of novel analytical frameworks, and it has produced results of conceptual and practical relevance. A coherent and comprehensive review of the vast research activity concerning epidemic processes is presented, detailing the successful theoretical approaches as well as making their limits and assumptions clear. Physicists, mathematicians, epidemiologists, computer, and social scientists share a common interest in studying epidemic spreading and rely on similar models for the description of the diffusion of pathogens, knowledge, and innovation. For this reason, while focusing on the main results and the paradigmatic models in infectious disease modeling, the major results concerning generalized social contagion processes are also presented. Finally, the research activity at the forefront in the study of epidemic spreading in coevolving, coupled, and time-varying networks is reported.Comment: 62 pages, 15 figures, final versio

Epidemic spreading in evolving networks

A model for epidemic spreading on rewiring networks is introduced and analyzed for the case of scale free steady state networks. It is found that contrary to what one would have naively expected, the rewiring process typically tends to suppress epidemic spreading. In particular it is found that as in static networks, rewiring networks with degree distribution exponent $\gamma >3$ exhibit a threshold in the infection rate below which epidemics die out in the steady state. However the threshold is higher in the rewiring case. For $2<\gamma \leq 3$ no such threshold exists, but for small infection rate the steady state density of infected nodes (prevalence) is smaller for rewiring networks.Comment: 7 pages, 7 figure